X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (138) 138
gastroenterology and hepatology (105) 105
index medicus (103) 103
gastroenterology & hepatology (85) 85
barrett's esophagus (55) 55
barrett esophagus - pathology (49) 49
article (43) 43
gastroesophageal reflux (42) 42
abridged index medicus (40) 40
esophagus (40) 40
male (37) 37
adenocarcinoma (34) 34
female (33) 33
barrett esophagus - metabolism (30) 30
cancer (30) 30
metaplasia (29) 29
oncology (28) 28
esophagus - pathology (27) 27
barrett esophagus - genetics (25) 25
animals (24) 24
esophageal adenocarcinoma (24) 24
middle aged (24) 24
dysplasia (23) 23
esophageal neoplasms - pathology (23) 23
expression (23) 23
gastroesophageal-reflux disease (23) 23
biopsy (21) 21
cell line (21) 21
aged (20) 20
apoptosis (20) 20
esophageal cancer (20) 20
research (20) 20
digestive system diseases (19) 19
analysis (18) 18
epithelial cells - metabolism (18) 18
esophageal neoplasms - genetics (18) 18
gastroenterology (18) 18
adenocarcinoma - pathology (17) 17
development and progression (17) 17
gastroesophageal reflux - complications (17) 17
physiology (17) 17
proton pump inhibitors (17) 17
barretts-esophagus (16) 16
cytokines (16) 16
esophageal neoplasms - metabolism (16) 16
inflammation (16) 16
mutation (16) 16
risk factors (16) 16
adenocarcinoma - genetics (15) 15
epithelial cells - drug effects (14) 14
epithelial cells - pathology (14) 14
genetic aspects (14) 14
phosphorylation (14) 14
adult (13) 13
cells (13) 13
medicine & public health (13) 13
pathogenesis (13) 13
risk (13) 13
epithelial-cells (12) 12
esophagitis (12) 12
esophagus - metabolism (12) 12
gastroesophageal reflux disease (12) 12
gene expression (12) 12
gene-expression (12) 12
immunohistochemistry (12) 12
signal transduction (12) 12
activation (11) 11
adenocarcinoma - metabolism (11) 11
apoptosis - drug effects (11) 11
carcinogenesis (11) 11
care and treatment (11) 11
cell biology (11) 11
diagnosis (11) 11
disease progression (11) 11
endoscopy (11) 11
epithelium (11) 11
esophagoscopy (11) 11
gastroesophageal reflux - metabolism (11) 11
gastroesophageal reflux - pathology (11) 11
hepatology (11) 11
high-grade dysplasia (11) 11
progression (11) 11
proteins (11) 11
rodents (11) 11
barrett esophagus - diagnosis (10) 10
barrett’s esophagus (10) 10
biochemistry & molecular biology (10) 10
epithelial cells (10) 10
health aspects (10) 10
mice (10) 10
multidisciplinary sciences (10) 10
physiological aspects (10) 10
proton pump inhibitors - therapeutic use (10) 10
rats (10) 10
stem cells (10) 10
telomerase (10) 10
time factors (10) 10
barrett esophagus - etiology (9) 9
biochemistry, general (9) 9
cell transformation, neoplastic - genetics (9) 9
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal of Gastroenterology, ISSN 0944-1174, 7/2017, Volume 52, Issue 7, pp. 767 - 776
Reflux esophagitis damages the squamous epithelium that normally lines the esophagus, and promotes replacement of the damaged squamous lining by the intestinal... 
Squamous cells | Barrett’s esophagus | NF-κB | Medicine & Public Health | Cdx2 | Colorectal Surgery | Hepatology | Gastroenterology | Cytokine | Abdominal Surgery | Surgical Oncology | cytokine | CDX2 | squamous cells
Journal Article
JOURNAL OF GASTROENTEROLOGY, ISSN 0944-1174, 07/2017, Volume 52, Issue 7, pp. 767 - 776
Reflux esophagitis damages the squamous epithelium that normally lines the esophagus, and promotes replacement of the damaged squamous lining by the intestinal... 
Squamous cells | HYPOXIA-INDUCIBLE FACTOR | Cdx2 | GASTROESOPHAGEAL JUNCTION | Cytokine | FACTOR-KAPPA-B | SQUAMOUS-CELLS | NF-kappa B | CDX2 EXPRESSION | INTESTINAL DIFFERENTIATION | Barrett's esophagus | PROTEIN EXPRESSION | NITRIC-OXIDE | GASTROENTEROLOGY & HEPATOLOGY | GENE COPY NUMBER | HOMEOBOX GENE | Hepatocyte Nuclear Factor 3-beta - genetics | Humans | Hedgehog Proteins - metabolism | NF-kappa B - metabolism | Barrett Esophagus - etiology | CDX2 Transcription Factor - genetics | Cellular Reprogramming | SOXB1 Transcription Factors - metabolism | Epithelial Cells - physiology | Barrett Esophagus - pathology | Esophagitis, Peptic - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | SOXB1 Transcription Factors - genetics | Cell Transdifferentiation | Proto-Oncogene Proteins c-akt - metabolism | Barrett Esophagus - genetics | Esophagitis, Peptic - complications | Hepatocyte Nuclear Factor 3-beta - metabolism | SOX9 Transcription Factor - metabolism | Barrett Esophagus - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Cytokines - metabolism | Signal Transduction | Esophagitis, Peptic - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | CDX2 Transcription Factor - metabolism | Esophagitis, Peptic - genetics | Stem Cells - physiology | SOX9 Transcription Factor - genetics | Gastroesophageal reflux | Esophagitis | B cells | T cells | Analysis | Adenocarcinoma | Stomach | NF-κB protein | Transcription factors | Cytokines | Leukocyte migration | Epithelial cells | Pathogenesis | Lymphocytes T | Inflammation | Epithelium | Esophagus | Metaplasia | Lymphocytes B | Intestine | Rodents | Stem cells | Bone marrow | Hypoxia | Cell migration
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50037
Background: Patients who have esophageal eosinophilia without gastroesophageal reflux disease (GERD) nevertheless can respond to proton pump inhibitors (PPIs),... 
ACTIVATION | EPITHELIAL-CELLS | GASTROESOPHAGEAL-REFLUX DISEASE | ATPASE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ADULTS | ACIDIFICATION | CONSENSUS RECOMMENDATIONS | ASSOCIATION | CHILDREN | Eosinophilic Esophagitis - metabolism | Eosinophils - metabolism | Eosinophils - drug effects | Humans | Interleukin-4 - metabolism | Omeprazole - administration & dosage | DNA-Binding Proteins - drug effects | Eosinophilic Esophagitis - pathology | STAT6 Transcription Factor - genetics | RNA Polymerase II - metabolism | STAT6 Transcription Factor - metabolism | Promoter Regions, Genetic - drug effects | Chemokine CCL26 | Gene Expression Regulation - drug effects | Gastric Acid - secretion | Proton Pump Inhibitors - administration & dosage | Interleukin-4 - administration & dosage | Chemokines, CC - genetics | Chemokines, CC - biosynthesis | Eosinophilic Esophagitis - drug therapy | Cell Culture Techniques | Eosinophils - cytology | Chemokines, CC - metabolism | Gastroesophageal reflux | Omeprazole | Care and treatment | Messenger RNA | Cytokines | Esophagitis | Enzyme-linked immunosorbent assay | Lansoprazole | Phosphorylation | Disease | Veterans | Acidification | Lymphocytes T | DNA-directed RNA polymerase | Western blotting | Eotaxin | Interleukin 4 | Dysphagia | Telomerase | Binding | Translocation | Squamous cells | Internal medicine | Leukocytes (eosinophilic) | Cultures | Inflammation | RNA polymerase | Gene expression | Ribonucleic acid--RNA | Patients | Eosinophilia | Nuclear transport | Esophagus | Asthma | Medicine | Polymerase | Molecular modelling | Acids | Biopsy | Production methods | Ulcers | Stat6 protein | RNA polymerase II | mRNA stability | Cancer | RNA | Ribonucleic acid
Journal Article
Gut, ISSN 0017-5749, 06/2013, Volume 62, Issue 6, pp. 824 - 832
Objective Eosinophilic oesophagitis (EoE) and gastro-oesophageal reflux disease (GORD) can have similar clinical and histological features. Proton pump... 
INTERLEUKIN-4 | ACTIVATION | EPITHELIAL-CELLS | GASTROESOPHAGEAL-REFLUX DISEASE | FOOD ALLERGY | GENE-EXPRESSION | ADULTS | MECHANISMS | BARRETTS-ESOPHAGUS | GASTROENTEROLOGY & HEPATOLOGY | CONSENSUS RECOMMENDATIONS | Epithelial Cells - metabolism | Th2 Cells - physiology | Interleukin-13 - antagonists & inhibitors | Epithelial Cells - drug effects | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Chemokine CCL26 | Interleukin-4 - pharmacology | Adult | Female | Gastroesophageal Reflux - drug therapy | Real-Time Polymerase Chain Reaction | Eosinophilic Esophagitis - metabolism | Chemokines, CC - drug effects | Gastroesophageal Reflux - metabolism | Omeprazole - therapeutic use | Interleukin-4 - antagonists & inhibitors | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Gene Expression Regulation | Interleukin-13 - pharmacology | Proton Pump Inhibitors - therapeutic use | Esophagus - pathology | Chemokines, CC - genetics | Eosinophilic Esophagitis - drug therapy | Chemokines, CC - metabolism | Omeprazole | Gastroesophageal reflux | Patient outcomes | Physiological aspects | Esophagitis | Genetic aspects | Dosage and administration | Research | Drug therapy | Chemokines | Cell culture | Transcription | Veterans | Gene regulation | Lymphocytes T | Eotaxin | Interleukin 4 | Dysphagia | Endoscopy | Telomerase | Squamous cells | Food allergies | Cytokines | Review boards | Leukocytes (eosinophilic) | Interleukin 13 | Gene expression | Eosinophilia | Esophagus | Studies | Molecular modelling | Acids | Cell lines | Clinical medicine | proton pump inhibitors | Th2 cytokines | eosinophilic oesophagitis | GORD | eotaxin-3
Journal Article
Journal Article
American Heart Journal, ISSN 0002-8703, 2015, Volume 169, Issue 5, pp. 631 - 638.e7
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 8/2018, Volume 63, Issue 8, pp. 1985 - 1987
Journal Article
The American Journal of Gastroenterology, ISSN 0002-9270, 06/2007, Volume 102, Issue 6, pp. 1301 - 1306
Journal Article