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Epilepsia, ISSN 0013-9580, 11/2007, Volume 48, Issue 8, pp. 11 - 13
Journal Article
Epilepsia, ISSN 0013-9580, 11/2007, Volume 48, Issue s8, pp. 11 - 13
Journal Article
Epilepsia, ISSN 0013-9580, 11/2007, Volume 48, Issue s8, p. 11
Journal Article
Neurobiology of Learning and Memory, ISSN 1074-7427, 03/2018, Volume 149, pp. 144 - 153
The anterior bed nucleus of stria terminalis (BNST) is involved in reinstatement of extinguished fear, and neuropeptide Y2 receptors influence local synaptic... 
Fear extinction | Neuropeptides | Remote fear memory | Extended amygdala | Anxiety disorder | ANXIETY | POTENTIATED STARTLE | CONDITIONED FEAR | NEUROSCIENCES | BED NUCLEUS | PSYCHOLOGY | PSYCHOLOGY, MULTIDISCIPLINARY | CORTICOTROPIN-RELEASING-FACTOR | STRIA TERMINALIS | BEHAVIORAL SCIENCES | MICE | STRESS | EXPRESSION | AMYGDALA
Journal Article
Neuropharmacology, ISSN 0028-3908, 10/2012, Volume 63, Issue 5, pp. 806 - 817
Injection of the seaweed toxin kainic acid (KA) in rats induces a severe status epilepticus initiating complex neuropathological changes in limbic brain areas... 
Epileptogenesis | Temporal lobe epilepsy | Subiculum | Entorhinal cortex | Animal epilepsy model | EEG | KAINATE-INDUCED EPILEPSY | TEMPORAL-LOBE EPILEPSY | NEUROSCIENCES | NEURONAL DAMAGE | NEUROPEPTIDE-Y | LAYER-III | PILOCARPINE MODEL | PHARMACOLOGY & PHARMACY | RAT HIPPOCAMPUS | SPONTANEOUS MOTOR SEIZURES | SPONTANEOUS RECURRENT SEIZURES | TRANSGENIC MICE | Microglia - metabolism | Neurons - pathology | Entorhinal Cortex - metabolism | Astrocytes - pathology | Status Epilepticus - drug therapy | Male | Hippocampus - drug effects | Kainic Acid | Time Factors | Nerve Degeneration - prevention & control | Microglia - pathology | Neurogenesis - drug effects | Neurons - metabolism | Neurons - drug effects | Drug Resistance | Epilepsy, Temporal Lobe - prevention & control | Disease Models, Animal | Severity of Illness Index | Astrocytes - drug effects | Microglia - drug effects | Status Epilepticus - pathology | Anticonvulsants - therapeutic use | Rats | Epilepsy, Temporal Lobe - etiology | Hippocampus - pathology | Entorhinal Cortex - drug effects | Organ Specificity | Rats, Sprague-Dawley | Marine Toxins | Hippocampus - metabolism | Animals | Status Epilepticus - physiopathology | Diazepam - therapeutic use | Entorhinal Cortex - pathology | Status Epilepticus - metabolism | Astrocytes - metabolism | RNA | Neurons | Analysis | Epilepsy | Amino acids | Glutamate decarboxylase | Seizures (Medicine) | Glutamate | Intermediate filament proteins | Status epilepticus | Diazepam | Brain | Astrocytes | mRNA | Seaweeds | Microglia | Cortex (entorhinal) | Dentate gyrus | Toxins | Glutamic acid transporter | Kainic acid | Hippocampus | Seizures | KA, kainic acid | GFAP, glial fibrillary acidic protein | GAD65 and GAD67, glutamate decarboxylase 65 and 67 | TLE, temporal lobe epilepsy | VGLUT1, vesicular glutamate transporter 1 | DG, dentate gyrus | SE, status epilepticus | NeuN, neuron-specific nuclear protein | Sub, subiculum | FJ-C, Fluoro Jade C
Journal Article
Cell Metabolism, ISSN 1550-4131, 02/2013, Volume 17, Issue 2, pp. 236 - 248
Neuropepetide Y (NPY) is best known for its powerful stimulation of food intake and its effects on reducing energy expenditure. However, the pathways involved... 
ENERGY-BALANCE | BODY-WEIGHT | MESSENGER-RNA | ADAPTIVE THERMOGENESIS | FOOD-INTAKE | ENDOCRINOLOGY & METABOLISM | BROWN ADIPOSE-TISSUE | NORMAL RATS | NEUROPEPTIDE-Y INFUSION | DIET-INDUCED OBESITY | VENTROMEDIAL HYPOTHALAMUS | CELL BIOLOGY | Thermogenesis - genetics | Tyrosine 3-Monooxygenase - metabolism | Temperature | Receptors, Neuropeptide Y - metabolism | Arcuate Nucleus of Hypothalamus - cytology | Male | Neurons - cytology | Neuropeptide Y - genetics | Paraventricular Hypothalamic Nucleus - enzymology | RNA, Messenger - metabolism | Sympathetic Nervous System - metabolism | Neuropeptide Y - metabolism | Paraventricular Hypothalamic Nucleus - cytology | Mitochondrial Proteins - metabolism | Diet, High-Fat | Models, Animal | Energy Metabolism - genetics | Pro-Opiomelanocortin - genetics | Arcuate Nucleus of Hypothalamus - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Feeding Behavior | Signal Transduction - genetics | Agouti-Related Protein - metabolism | Pro-Opiomelanocortin - metabolism | Tyrosine 3-Monooxygenase - genetics | Animals | Ion Channels - metabolism | Oxygen Consumption - genetics | Neurons - enzymology | Glutamate Decarboxylase - metabolism | Adipose Tissue, Brown - metabolism | Mice | Uncoupling Protein 1 | Neuropeptide Y | Tyrosine | Neurosciences | Neurons | Eating disorders | Phenols | Universities and colleges | Hydroxylases
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 02/2010, Volume 107, Issue 7, pp. 3180 - 3185
Refractory temporal lobe epilepsy (TLE) is associated with a dysfunction of inhibitory signaling mediated by GABA A receptors. In particular, the use-dependent... 
Xenopus oocytes | GABA | receptor | Pilocarpine rat | Biological Sciences | GABAA receptor | pilocarpine rat
Journal Article
Journal of Molecular Neuroscience, ISSN 0895-8696, 1/2012, Volume 46, Issue 1, pp. 18 - 32
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 05/2014, Volume 111, Issue 19, pp. 7138 - 7143
The neuropeptide galanin (GAL) is widely distributed in the central and peripheral nervous systems. It is a modulator of various physiological and pathological... 
ECCRINE SWEAT GLAND | MULTIDISCIPLINARY SCIENCES | BRAIN GALANIN SYSTEM | KNOCKOUT MICE | ELEVATED PLUS-MAZE | Gcat variant 2 | galanin receptor | NEUROPEPTIDE-Y | serotonin | DEPRESSION-LIKE BEHAVIOR | ANXIOLYTIC-LIKE | OVEREXPRESSING MICE | STRESS | TRANSGENIC MICE
Journal Article
2007, Progress in Brain Research, ISBN 9780444530158, Volume 163, 13
Book Chapter
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 7, pp. 3180 - 3185
Refractory temporal lobe epilepsy (TLE) is associated with a dysfunction of inhibitory signaling mediated by ${\text{GABA}}_A $ receptors. In particular, the... 
Receptors | Neuroscience | Nervous system diseases | Neurons | Epilepsy | Rats | Temporal lobe epilepsy | Hippocampus | Oocytes | Seizures | GABA | Development and progression | Models | Seizures (Medicine) | Diagnosis | Hippocampus (Brain) | Properties
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2009, Volume 108, Issue 3, pp. 707 - 718
Journal Article
Cell Metabolism, ISSN 1550-4131, 02/2013, Volume 17, Issue 2, pp. 236 - 248
Neuropepetide Y (NPY) is best known for its powerful stimulation of food intake and its effects on reducing energy expenditure. However, the pathways involved... 
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2010, Volume 107, Issue 7, p. 3180
  Refractory temporal lobe epilepsy (TLE) is associated with a dysfunction of inhibitory signaling mediated by GABA(A) receptors. In particular, the... 
Proteins | Brain | Convulsions & seizures | Molecular structure | Neurons | Epilepsy | Rodents
Journal Article