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Acta neuropathologica, ISSN 0001-6322, 08/2019, pp. 1 - 12
Low-level somatic mutations have been shown to be the major genetic etiology of intractable epilepsy. The extents thereof, however, have yet to be... 
TOR protein | TSC2 protein | Genetic counseling | Etiology | Epilepsy | Mutation | TSC1 protein | Paraffin
Journal Article
CANCERS, ISSN 2072-6694, 01/2019, Volume 11, Issue 1, p. 41
Epilepsy at presentation is an independent favorable prognostic factor in glioblastoma (GBM). In this study, we analyze the oncologic signaling pathways that... 
epilepsy | SURVIVAL | hypoxia | STAT5b | HIF-1 alpha | PHOSPHORYLATION | GLIOMA | IDH1 | CELL INVASION | GSEA | VALPROIC ACID | tissue microarrays | glioblastoma | ONCOLOGY | mesenchymal transformation | GENE-EXPRESSION | translational research | NF-KAPPA-B | HIF-1α
Journal Article
Oncotarget, ISSN 1949-2553, 06/2018, Volume 9, Issue 46, pp. 28103 - 28115
Glioneuronal tumours, including gangliogliomas and dysembryoplastic neuroepithelial tumours, represent the most common low-grade epilepsy-associated brain... 
Low-grade epilepsy-associated brain tumours | Ganglioglioma | Epilepsy | Glioneuronal tumour | Dysembryoplastic neuroepithelial tumour
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 9, p. e44789
Increasing evidence supports the involvement of microRNAs (miRNA) in the regulation of inflammation in human neurological disorders. In the present study we... 
INTERLEUKIN-1 | CORTICAL DYSPLASIA | ACTIVATION | MALFORMATIONS | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTOR | DISEASE | EPILEPSY | NF-KAPPA-B | EXPRESSION | RAT MODEL | Brain Diseases - metabolism | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1beta - pharmacology | Humans | Astrocytes - pathology | Brain Diseases - genetics | Epilepsy - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Brain Diseases - immunology | Ganglioglioma - pathology | Inflammation Mediators - metabolism | Epilepsy - genetics | Female | Toll-Like Receptors - metabolism | Malformations of Cortical Development - immunology | Malformations of Cortical Development - pathology | Interleukin-6 - metabolism | Astrocytes - drug effects | Malformations of Cortical Development, Group I | Immunity, Innate - drug effects | Malformations of Cortical Development - metabolism | Malformations of Cortical Development - genetics | Receptors, Interleukin-1 - metabolism | Gene Expression Regulation - drug effects | Pregnancy | Ganglioglioma - genetics | Brain Diseases - pathology | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Cell Line, Tumor | MicroRNAs - genetics | Ganglioglioma - metabolism | Epilepsy - pathology | Astrocytes - metabolism | Astrocytes - secretion | Medical research | Nervous system diseases | Immune response | MicroRNA | Cytokines | Epilepsy | Medicine, Experimental | Inflammation | Research | Enzyme-linked immunosorbent assay | Neurosciences | Downstream effects | Disorders | Inflammatory response | Nervous system | Kinases | Interleukin 6 | Transfection | Modulation | Lesions | Immune system | IRAK protein | Encephalitis | Immunomodulation | Astrocytes | MiRNA | Cultures | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Neurological diseases | Pathology | Signaling | Brain research | MicroRNAs | Cyclooxygenase-2 | RNA | Ribonucleic acid
Journal Article
Data in Brief, ISSN 2352-3409, 08/2018, Volume 19, pp. 6 - 12
In this article, we report data on vessel wall thickness parameters derived from different arterial segments of the circle of Willis and its primary branches... 
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6
Tuberous Sclerosis Complex (TSC) is a genetic hamartoma syndrome frequently associated with severe intractable epilepsy. In some TSC patients epilepsy surgery... 
Journal Article
Brain, ISSN 0006-8950, 2011, Volume 134, Issue Part 4, pp. 1015 - 1032
Recent evidence in experimental models of seizures and in temporal lobe epilepsy support an important role of high-mobility group box 1 and toll-like receptor... 
epilepsy | focal cortical dysplasia | tuberous sclerosis complex | inflammation | glioneuronal tumours | MAMMALIAN TARGET | INNATE | ASTROCYTES | NEUROSCIENCES | CLINICAL NEUROLOGY | GLIONEURONAL TUMORS | ADAPTIVE IMMUNITY | CHROMATIN PROTEIN | MOBILITY GROUP BOX-1 | NEURONAL EXCITABILITY | EXPRESSION | GLYCATION END-PRODUCTS | Neurons - pathology | Cell Count | Humans | Astrocytes - pathology | Cerebral Cortex - pathology | Epilepsy - metabolism | RNA, Messenger - metabolism | Cerebral Cortex - metabolism | HMGB1 Protein - genetics | HMGB1 Protein - metabolism | Statistics, Nonparametric | Toll-Like Receptors - physiology | Adult | Epilepsy - genetics | Neurons - metabolism | Toll-Like Receptors - metabolism | HMGB1 Protein - physiology | Malformations of Cortical Development - pathology | Child | Receptor for Advanced Glycation End Products | RNA, Messenger - genetics | Malformations of Cortical Development - metabolism | Malformations of Cortical Development - genetics | Reverse Transcriptase Polymerase Chain Reaction | Malformations of Cortical Development - complications | Blotting, Western | Epilepsy - complications | Analysis of Variance | Toll-Like Receptors - genetics | Adolescent | Signal Transduction - physiology | Receptors, Immunologic - genetics | Epilepsy - pathology | Receptors, Immunologic - metabolism | Astrocytes - metabolism
Journal Article
Oncotarget, ISSN 1949-2553, 11/2017, Volume 8, Issue 56, pp. 95516 - 95529
Subependymal giant cell astrocytomas (SEGAs) are rare, low-grade glioneuronal brain tumors that occur almost exclusively in patients with tuberous sclerosis... 
Journal Article
Brain Pathology, ISSN 1015-6305, 01/2014, Volume 24, Issue 1, pp. 1 - 17
Journal Article
Epilepsia, ISSN 0013-9580, 2010, Volume 51, Issue 9, pp. 1763 - 1773
P>Purpose: Induction of inflammatory pathways has been reported in epileptic patients with focal malformations of cortical development. In the present study we... 
Interleukin-1 | MCP1 | Dendritic cells | Lymphocytes | Epilepsy | Complement | Microglia | MAMMALIAN TARGET | RAPAMYCIN | ACTIVATION | TEMPORAL-LOBE EPILEPSY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | MTOR | CLINICAL NEUROLOGY | MALFORMATIONS | TUBEROUS SCLEROSIS COMPLEX | INTRACTABLE EPILEPSY | Immunohistochemistry | Inflammation - pathology | Interleukin-1 - genetics | Neurons - pathology | Chemokine CCL2 - immunology | Dendritic Cells - immunology | Humans | Receptors, Metabotropic Glutamate - immunology | Cerebral Cortex - pathology | Immunity, Innate - genetics | Dendritic Cells - pathology | Complement Activation - immunology | Microglia - immunology | Receptors, Metabotropic Glutamate - genetics | Immunity, Innate - physiology | Epilepsy - immunology | Interleukin-1 - immunology | Neurons - physiology | Microglia - pathology | Epilepsy - genetics | Adaptive Immunity - immunology | Malformations of Cortical Development - immunology | Malformations of Cortical Development - pathology | Adaptive Immunity - physiology | Neurons - immunology | Chemokine CCL2 - genetics | Inflammation - immunology | Malformations of Cortical Development - genetics | Complement Activation - genetics | Immunity, Innate - immunology | Cerebral Cortex - immunology | Inflammation - genetics | Epilepsy - pathology | Neurosciences | Dysplasia | Interleukins | Analysis | Cytochemistry | T cells | TOR protein | Immune response | CD8 antigen | Cortex | CD3 antigen | Inflammation | Lymphocytes T | Immunity | Cell activation | Complement activation | Interleukin 1 | Histocompatibility antigen HLA | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PLoS ONE, ISSN 1932-6203, 2016, Volume 11, Issue 3, p. e0151465
BACKGROUND: Central nervous system (CNS) primitive neuroectodermal tumors (PNETs) are malignant primary brain tumors that occur in young infants. Using current... 
SURVIVAL | PROTEASE INHIBITOR-9 | APOPTOSIS | NK CELLS | MULTIDISCIPLINARY SCIENCES | IMMUNE ESCAPE | DELTA-T-CELLS | RADIOTHERAPY | IDENTIFICATION | MEDULLOBLASTOMA | CHILDREN | Granzymes - antagonists & inhibitors | Tumor Escape | CD8-Positive T-Lymphocytes - pathology | Antigens, CD1d - immunology | Humans | Brain Neoplasms - pathology | Neoplasm Proteins - immunology | Child, Preschool | Male | CD4-Positive T-Lymphocytes - pathology | CD4-Positive T-Lymphocytes - immunology | Brain Neoplasms - immunology | Neuroectodermal Tumors, Primitive - immunology | Female | Granzymes - immunology | Child | Histocompatibility Antigens Class I - immunology | Gene Expression Regulation, Neoplastic - immunology | Receptors, Antigen, T-Cell, gamma-delta - immunology | Serine Proteinase Inhibitors - immunology | Neuroectodermal Tumors, Primitive - therapy | Serpins - immunology | Adolescent | Brain Neoplasms - therapy | CD8-Positive T-Lymphocytes - immunology | Neuroectodermal Tumors, Primitive - pathology | Physiological aspects | Genetic aspects | Immune response | Research | Immunohistochemistry | Brain | Pediatrics | Toxicity | CD8 antigen | Brain tumors | Brain cancer | Central nervous system | Serpins | Cytotoxicity | Nervous system | Infants | Lymphocytes T | Cell surface | Cell adhesion & migration | Ethics | Immunotherapy | Tumorigenesis | Children | Immune system | Antigens | Nervous system diseases | Immunoglobulins | Cell survival | Review boards | Tumor cells | Patients | CD4 antigen | Studies | CD1d antigen | Pathology | Inhibitors | Major histocompatibility complex | Tumors | Cancer | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 2016, Volume 11, Issue 6, p. e0157396
Tuberous Sclerosis Complex (TSC) is a genetic hamartoma syndrome frequently associated with severe intractable epilepsy. In some TSC patients epilepsy surgery... 
DYSPLASIA TYPE-II | MALFORMATIONS | HIPPOCAMPAL SCLEROSIS | MULTIDISCIPLINARY SCIENCES | MRI FEATURES | ILAE CLASSIFICATION | EXPRESSION | INTEROBSERVER | LESIONS | CONSENSUS CLASSIFICATION | CHILDREN | Neurons - pathology | TOR Serine-Threonine Kinases - metabolism | Tuberous Sclerosis - complications | Humans | Middle Aged | Tuberous Sclerosis - surgery | Cerebral Cortex - pathology | Epilepsy - surgery | Child, Preschool | Epilepsy - metabolism | Male | Mechanistic Target of Rapamycin Complex 1 | Myelin Sheath - metabolism | Gliosis - pathology | Tuberous Sclerosis - pathology | Young Adult | Multiprotein Complexes - metabolism | Adult | Female | Child | Cerebral Cortex - surgery | Myelin Sheath - pathology | Gliosis - complications | Epilepsy - complications | TOR Serine-Threonine Kinases - analysis | Multiprotein Complexes - analysis | Tuberous Sclerosis - metabolism | Adolescent | Epilepsy - pathology | Complications and side effects | Hospital patients | Care and treatment | Analysis | Tuberous sclerosis | Genetic aspects | Histology, Pathological | TOR protein | Brain | Pediatrics | Epilepsy | Medical schools | Angiogenesis | Surgery | Oligodendrocytes | Giant cells | Lesions | Localization | Age | Automation | Neurons | Myelin | Leukocytes (eosinophilic) | Cortex | Exploration | Histology | Rapamycin | Substantia alba | Patients | Pathology | Neurology | Gliosis | Hospitals | Magnetic resonance imaging | Correlation analysis | Calcification | Mutation | Neoplasia
Journal Article