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Journal of Diabetes and Metabolic Disorders, ISSN 2251-6581, 12/2018, Volume 17, Issue 2, pp. 381 - 391
Energy imbalance resulting from high calorie food intake and insufficient metabolic activity leads to increased body mass index (BMI) and sets the stage for... 
Life-style | Cardiovascular | Atherosclerosis | Inflammation | Diabetes | Epigenetic | NASH | Body mass index | Complications and side effects | Epigenetic inheritance | Research | Cardiovascular diseases | Health aspects | Risk factors | Epigenetics | Oxidative stress | Metabolism | Pathogenesis | Liver
Journal Article
Journal of Cardiovascular Pharmacology, ISSN 0160-2446, 07/2018, Volume 72, Issue 1, pp. 3 - 10
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A9805 - A9805
To circumvent the residual cardiovascular disease burden in patients receiving statin therapy, HDL-raising was suggested as a plausible approach. However, lack... 
Journal Article
Atherosclerosis, ISSN 0021-9150, 01/2011, Volume 214, Issue 1, pp. 86 - 93
Fenofibrate, a PPAR-α agonist and rosiglitazone, a PPAR-γ agonist, reduce triglycerides and fatty acids in humans and in animal disease models. The efficacy of... 
Fenofibrate | HDL-C | Animal model | PPAR | LDL-C | Atherosclerosis | LXR | Rosiglitazone | Hamster | LIPID-METABOLISM | LDL RECEPTOR | DIETARY-CHOLESTEROL | CHOLESTERYL ESTER TRANSFER | ACTIVATED RECEPTOR-ALPHA | DEFICIENT MICE | LIPOPROTEIN METABOLISM | TRANSFER PROTEIN EXPRESSION | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | GOLDEN-SYRIAN-HAMSTERS
Journal Article
Atherosclerosis, ISSN 0021-9150, 2010, Volume 214, Issue 1, pp. 86 - 93
Abstract Background Fenofibrate, a PPAR-α agonist and rosiglitazone, a PPAR-γ agonist, reduce triglycerides and fatty acids in humans and in animal disease... 
Cardiovascular
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 04/2009, Volume 607, Issue 1-3, p. 258
Fenofibrate and rosiglitazone are prescribed to treat hypertriglyceridemia and diabetes, respectively. Since fenofibrate improves lipid profile in diabetic... 
Glycerol | Lipids | Triglycerides | Glucose | Fatty acids | Dextrose | Glycerin | Synthesis | Messenger RNA | Insulin resistance | Diabetes | Fibric acids | Protein binding
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2009, Volume 607, Issue 1, pp. 258 - 263
Fenofibrate and rosiglitazone are prescribed to treat hypertriglyceridemia and diabetes, respectively. Since fenofibrate improves lipid profile in diabetic... 
Fenofibrate | SREBP1c (sterol response element binding protein 1c) | PPAR (peroxisome proliferator activated receptor) | PEPCK (phosphoenol pyruvate carboxykinase) | Rosiglitazone | Liver x receptor | DGAT (diacyl glycerol acyl transferase) | KKAy | Diabetes | 11β-HSD1 (11beta hydroxysteroid dehydrogenase)
Journal Article
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 12/2010, Volume 345, Issue 1, pp. 197 - 206
Journal Article
Atherosclerosis (Supplements) (Component), ISSN 1567-5688, 06/2018, Volume 32, pp. 129 - 130
Journal Article
Atherosclerosis, ISSN 0021-9150, 2014, Volume 236, Issue 1, pp. 91 - 100
Abstract Despite the benefit of statins in reducing cardiovascular risk, a sizable proportion of patients still remain at risk. Since HDL reduces CVD risk... 
Cardiovascular | apoE deficient | Preβ-HDL | Atherosclerosis | Inflammation | BET inhibitor | apoA-I-inducer | RVX-208 | ApoE deficient | ApoA-I-inducer | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | PRIMARY-PREVENTION TRIAL | HIGH-DENSITY-LIPOPROTEIN | HEART-DISEASE | SCAVENGER RECEPTOR | REVERSE CHOLESTEROL TRANSPORT | CARDIOVASCULAR-DISEASE | Pre beta-HDL | PERIPHERAL VASCULAR DISEASE | CORONARY-ARTERY-DISEASE | APOLIPOPROTEIN-A-I | Hyperlipidemias - genetics | Endothelial Cells | Hyperlipidemias - blood | Apolipoproteins E - deficiency | Humans | Aortic Diseases - blood | RNA, Messenger - analysis | Male | Gene Expression Profiling | Aorta - metabolism | Inflammation - blood | Atherosclerosis - etiology | Liver - drug effects | U937 Cells | Aortic Diseases - prevention & control | Betaine-Homocysteine S-Methyltransferase - antagonists & inhibitors | Cholesterol, LDL - blood | Hyperlipidemias - diet therapy | Drug Evaluation, Preclinical | Hyperlipidemias - complications | Aortic Diseases - pathology | Cytokines - blood | Cell Line | Atherosclerosis - pathology | Aorta - drug effects | Hyperlipidemias - drug therapy | Liver - metabolism | Mice, Inbred C57BL | Diet, Western - adverse effects | Mice, Knockout | Aorta - pathology | Atherosclerosis - blood | Animals | Quinazolines - therapeutic use | Diet, Fat-Restricted | Cholesterol, HDL - blood | Inflammation - prevention & control | Mice | Quinazolines - pharmacology | Aortic Diseases - etiology | Atherosclerosis - prevention & control | Apolipoprotein A-I - blood | Haptoglobin | Apolipoproteins | Low density lipoproteins | Statins | Protein binding | Triglycerides | Interferon | Cardiovascular agents | Index Medicus
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 01/2013, Volume 54, Issue 1, pp. 134 - 151
ETC-1002 (8-hydroxy-2,2,14,14-tetramethylpentadecanedioic acid) is a novel investigational drug being developed for the treatment of dyslipidemia and other... 
Fatty acid oxidation | LDL-cholesterol | Cardiovascular disease | Cholesterol synthesis | Metabolic syndrome | Fatty acid synthesis | RAT-LIVER | cardiovascular disease | cholesterol synthesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACETYL-COA CARBOXYLASE | RECEPTOR-ALPHA | metabolic syndrome | fatty acid synthesis | FATTY-ACID OXIDATION | INSULIN-RESISTANCE | DENSITY-LIPOPROTEIN | fatty acid oxidation | HEXADECANEDIOIC ACID | PEROXISOME PROLIFERATORS | HEPATOCYTE NUCLEAR FACTOR-4-ALPHA | ALPHA,OMEGA-DICARBOXYLIC ACIDS | Carbohydrate Metabolism - drug effects | AMP-Activated Protein Kinases - metabolism | ATP Citrate (pro-S)-Lyase - antagonists & inhibitors | Liver - enzymology | Fatty Acids - therapeutic use | Obesity - drug therapy | Calcium - metabolism | Diet - adverse effects | Humans | Male | Glucose - biosynthesis | Dicarboxylic Acids - pharmacology | Sterols - biosynthesis | Obesity - blood | Dicarboxylic Acids - therapeutic use | Dyslipidemias - blood | Liver - drug effects | Enzyme Inhibitors - chemistry | Female | Obesity - etiology | ATP Citrate (pro-S)-Lyase - metabolism | Molecular Targeted Therapy - methods | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | Cricetinae | Dyslipidemias - drug therapy | Fatty Acids - chemistry | Liver - metabolism | Enzyme Inhibitors - pharmacology | Rats | Biomarkers - blood | Enzyme Activation - drug effects | Enzyme Inhibitors - therapeutic use | Hep G2 Cells | Obesity - metabolism | Dicarboxylic Acids - chemistry | Animals | Signal Transduction - drug effects | Lipid Metabolism - drug effects | Dyslipidemias - metabolism | Glucagon - metabolism | Liver - cytology | Mice | Energy Metabolism - drug effects | Fatty Acids - biosynthesis | Fatty Acids - pharmacology | Index Medicus
Journal Article