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The Journal of Pathology, ISSN 0022-3417, 04/2018, Volume 244, Issue 4, pp. 485 - 498
Pulmonary vascular remodeling is the main pathological hallmark of pulmonary hypertension disease. We undertook a comprehensive and multilevel approach to... 
pulmonary vascular remodeling | fate mapping | LUNG | RECRUITMENT | PATHOLOGY | MESENCHYMAL TRANSITION | HYPOXIA | ADULT-MOUSE | ONCOLOGY | MARROW-DERIVED CELLS | ARTERIAL-HYPERTENSION | SMOOTH-MUSCLE-CELLS | THYMIDINE-H-3 | EXPRESSION | Vascular Remodeling | Cadherins - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Antigens, CD - genetics | Muscle, Smooth, Vascular - physiopathology | Respiratory Hypersensitivity - pathology | Familial Primary Pulmonary Hypertension - metabolism | Familial Primary Pulmonary Hypertension - physiopathology | Actins - genetics | Antigens, CD - metabolism | Hypoxia - metabolism | Pulmonary Artery - metabolism | Respiratory Hypersensitivity - physiopathology | Cadherins - genetics | Myocytes, Smooth Muscle - metabolism | Genes, Reporter | Disease Models, Animal | Familial Primary Pulmonary Hypertension - pathology | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Respiratory Hypersensitivity - metabolism | Proteoglycans - metabolism | Mice, Transgenic | Antigens - metabolism | Pulmonary Artery - physiopathology | Cell Lineage | Respiratory Hypersensitivity - genetics | Hypoxia - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Hypoxia - pathology | Receptor, Platelet-Derived Growth Factor alpha - genetics | Fluorescent Antibody Technique | Hypoxia - physiopathology | Luminescent Proteins - genetics | Lung - blood supply | Chronic Disease | Proteoglycans - genetics | Pulmonary Artery - pathology | Antigens - genetics | Luminescent Proteins - metabolism | Cell proliferation | Cell culture | Allergens | Flow cytometry | Animal models | Pathogenesis | Smooth muscle | Labelling | Remodeling | Neuronal-glial interactions | Arteries | Vascular diseases | Cell fate | Actin | Rodents | Localization | Thickening | Hypertension | Pulmonary arteries | Lung diseases | Markers | Transgenic mice | Muscles | Blood vessels | Electron microscopy | Pulmonary artery | Endothelial cells | Pulmonary hypertension | Hypoxia | Chondroitin sulfate | Sulfate | Immunofluorescence | Veins & arteries | Original Paper | Original Papers
Journal Article
Pulmonary Circulation, ISSN 2045-8932, 7/2019, Volume 9, Issue 3, p. 204589401983221
The diagnosis of idiopathic pulmonary arterial hypertension (iPAH) is complex and, besides invasive hemodynamic evaluation, includes several diagnostic steps... 
CARDIAC & CARDIOVASCULAR SYSTEMS | DLCO | lung histology | RESPIRATORY SYSTEM | high-resolution lung CT | idiopathic pulmonary arterial hypertension | Emphysema | Lung diseases | Pulmonary hypertension
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 03/2020, Volume 145, Issue 3, pp. 818 - 833.e11
Idiopathic pulmonary fibrosis (IPF) is a disease with high 5-year mortality and few therapeutic options. Prostaglandin (PG) E2 exhibits antifibrotic properties... 
PGE2 | fibrocytes | idiopathic pulmonary fibrosis | 15-PGDH | APOPTOSIS | TGF-BETA | CYCLOOXYGENASE-2 | LUNG INJURY | LIPID MEDIATORS | IMMUNOLOGY | DEHYDROGENASE | FIBROBLASTS | PGE | EPITHELIAL-CELLS | ALLERGY | INFLAMMATION | PROSTAGLANDIN E-2
Journal Article
EUROPEAN RESPIRATORY JOURNAL, ISSN 0903-1936, 09/2019, Volume 54, Issue 3
The interleukin (IL)-1 family of cytokines is strongly associated with systemic sclerosis (SSc) and pulmonary involvement, but the molecular mechanisms are... 
INTERSTITIAL LUNG-DISEASE | BRONCHOALVEOLAR LAVAGE FLUID | MATRIX | TUMOR-NECROSIS-FACTOR | ACTIVATION | RESPIRATORY SYSTEM | MACROPHAGES | SERUM-LEVELS | INTERLEUKIN-1 RECEPTOR | ASSOCIATION | FIBROBLASTS
Journal Article
European Journal of Cancer, ISSN 0959-8049, 09/2018, Volume 101, pp. 165 - 180
Journal Article
Oncotarget, ISSN 1949-2553, 04/2019, Volume 10, Issue 26, pp. 2546 - 2560
The lung cancer stem cell (LuCSC) model comprises an attractive framework to explore acquired drug resistance in non-small cell lung cancer (NSCLC) treatment.... 
Journal Article
Journal of thoracic oncology, ISSN 1556-0864, 01/2017, Volume 12, Issue 1, pp. S1141 - S1142
Journal Article
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