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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2017, Volume 114, Issue 44, p. E9328
B cell receptor signaling and downstream NF-kB activity are crucial for the maturation and functionality of all major B cell subsets, yet the molecular players... 
Cell proliferation | Neonates | NF-κB protein | Immunoglobulins | Chromatin | Animal models | Maturation | Transcription | BRCA1 protein | Differentiation (biology) | Activation | Ontogeny | Breast cancer | Cell differentiation | Immunity | Signaling | Cell activation | Lymphocytes B | Heavy chains | Bone marrow | Immunity (humoral) | Mice | Genetic modification
Journal Article
Cell, ISSN 0092-8674, 06/2013, Volume 153, Issue 6, pp. 1266 - 1280
Journal Article
Nature, ISSN 0028-0836, 07/2016, Volume 535, Issue 7612, pp. 382 - 387
Cells deficient in the Brca1 and Brca2 genes have reduced capacity to repair DNA double-strand breaks by homologous recombination and consequently are... 
BREAST-CANCER | RECOMBINATION | LYSINE-4 METHYLTRANSFERASE COMPLEX | MUTANT-CELLS | 53BP1 | MULTIDISCIPLINARY SCIENCES | RESISTANCE | HOMOLOGY-DIRECTED REPAIR | CONDITIONAL MOUSE MODEL | END RESECTION | DNA-REPAIR | Embryonic Stem Cells - metabolism | DNA Replication - drug effects | Homologous Recombination | DNA Repair - genetics | DNA Breaks, Double-Stranded | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | Neoplasms - genetics | Gene Deletion | Nuclear Proteins - deficiency | DNA Repair Enzymes - metabolism | DNA Damage - genetics | Genes, BRCA2 | DNA - biosynthesis | Female | DNA Repair Enzymes - antagonists & inhibitors | Genes, BRCA1 | Nuclear Proteins - genetics | DNA Helicases - genetics | DNA Damage - drug effects | DNA Repair - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Cisplatin - pharmacology | DNA - metabolism | Carrier Proteins - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Poly(ADP-ribose) Polymerases - genetics | DNA Replication - physiology | Embryonic Stem Cells - drug effects | Cell Line, Tumor | Mice | Poly (ADP-Ribose) Polymerase-1 | Neoplasms - pathology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Chemotherapy | Replication fork | B cells | DNA damage | Cancer | Proteins | Lymphocytes | Stem cells | Breast cancer | Drug resistance | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Journal Article
Blood, ISSN 0006-4971, 04/2012, Volume 119, Issue 17, pp. 4034 - 4046
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2017, Volume 114, Issue 44, pp. E9328 - E9337
Journal Article
Genes and Development, ISSN 0890-9369, 01/2016, Volume 30, Issue 2, pp. 149 - 163
Journal Article
Current stem cell research & therapy, 06/2011, Volume 6, Issue 2, p. 142
In mammals, hematopoiesis is the continuous formation of all blood cell types from a limited pool of hematopoietic stem cells (HSCs) residing in specialized... 
Transcription Factors - physiology | Animals | Hematopoietic Stem Cells - cytology | Humans | Gene Expression Regulation | Hematopoietic Stem Cells - physiology | Hematopoiesis - physiology | Cell Differentiation | MicroRNAs - physiology
Journal Article
Biology of Sport, ISSN 0860-021X, 2015, Volume 32, Issue 1, pp. 3 - 9
To investigate the association between multiple single-nucleotide polymorphisms (SNPs), aerobic performance and elite endurance athlete status in Russians. By... 
GWA | Sgenotype | Phism | Polymor | Endurance VO | Athletes | GLY482SER | CAPACITY | SPORT SCIENCES | GWAS | SPORT | RECEPTOR | polymorphism | athletes | VOmax | MICRORNAS | endurance | AEROBIC PERFORMANCE | genotype | SKELETAL-MUSCLE | POLISH | VO2max | Original
Journal Article