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Gut, ISSN 0017-5749, 04/2011, Volume 60, Issue 4, pp. 463 - 472
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 2, pp. 355 - 365.e4
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2011, Volume 108, Issue 16, pp. 6603 - 6608
Journal Article
Cancer Letters, ISSN 0304-3835, 2012, Volume 324, Issue 1, pp. 98 - 108
Abstract We recently demonstrated that p38α is required to maintain colorectal cancer (CRC) metabolism, as its inhibition leads to FoxO3A activation,... 
Hematology, Oncology and Palliative Medicine | Animal models | p38 | Cell death | Colorectal cancer | ERK | P38 | CARCINOMA-CELLS | ACTIVATED PROTEIN-KINASE | HUMAN BREAST | CELL-PROLIFERATION | ERK1/2 PATHWAY | MAPK SIGNALING PATHWAYS | COLON-CANCER | DEPENDENT REGULATION | ONCOLOGY | AMPK-FOXO3A AXIS | P38-ALPHA | Phosphorylation | Diphenylamine - pharmacology | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Apoptosis - drug effects | Humans | Middle Aged | Caspase 8 - metabolism | Apoptosis - genetics | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Kinase 1 - genetics | Diphenylamine - analogs & derivatives | Cell Death - genetics | Mitogen-Activated Protein Kinase 14 - metabolism | Colorectal Neoplasms - drug therapy | Aged, 80 and over | Female | Benzamides - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Death - drug effects | Colorectal Neoplasms - metabolism | MAP Kinase Kinase 1 - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | MAP Kinase Kinase 1 - metabolism | Imidazoles - pharmacology | HT29 Cells | Xenograft Model Antitumor Assays | Animals | Mice, Nude | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Aged | Mice | Pyridines - pharmacology | Colorectal Neoplasms - pathology | Growth | Analysis | Medical genetics | Physiological aspects | Models | Apoptosis | Cell culture | Genotype & phenotype | Cell growth | Rodents | Clinical trials | Kinases | Cancer therapies | Tumors | Cancer | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2013, Volume 144, Issue 7, pp. 1497 - 1507.e13
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e104449 - e104449
Background & Aims: Liver regeneration (LR) is a valuable model for studying mechanisms modulating hepatocyte proliferation. Nuclear receptors (NRs) are key... 
CIRCADIAN CLOCK | PARTIAL-HEPATECTOMY | GLUCOCORTICOID-RECEPTOR | PPAR-DELTA | MULTIDISCIPLINARY SCIENCES | X-RECEPTOR | HEPATIC REGENERATION | CELL-PROLIFERATION | HUMAN HEPATOCELLULAR-CARCINOMA | ADAPTIVE RESPONSE | VARIABLE IMPORTANCE | Cell Line | Cell Proliferation | Liver Neoplasms - genetics | Humans | Liver - metabolism | Gene Expression Regulation | Transcriptome | Male | Gene Expression Profiling | Receptors, Cytoplasmic and Nuclear - genetics | Hepatocytes - metabolism | Liver Regeneration - genetics | Animals | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - metabolism | Mice | Carcinoma, Hepatocellular - metabolism | Cluster Analysis | Receptors, Cytoplasmic and Nuclear - metabolism | Cluster analysis | Transcription factors | Glucocorticoids | Liver | Hepatocellular carcinoma | Thyroid gland | Hormones | Hepatoma | Discriminators | Kinases | Carcinogenesis | Modulators | Proteins | Liver cancer | Receptors | Carcinogens | Rodents | Hepatology | Cell cycle | Tumor necrosis factor-TNF | Bioindicators | Growth factors | Thyroid | Circadian rhythms | Liver diseases | Cytokines | Liver X receptors | Cell division | Pharmacology | Interdisciplinary aspects | Circadian rhythm | Clustering | Gene expression | Nuclear receptors | Fatty acids | Disease control | Nutrition research | Regeneration | Biomarkers | Ligands | Peroxisome proliferator-activated receptors | Apoptosis | Bile | Index Medicus
Journal Article
Journal Article
Cell Cycle, ISSN 1538-4101, 09/2011, Volume 10, Issue 17, pp. 2937 - 2945
Journal Article