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Journal of Neuroscience, ISSN 0270-6474, 2013, Volume 33, Issue 40, pp. 15879 - 15893
Inflammation has been implicated in the progression of neurological disease, yet precisely how inflammation affects neuronal function remains unclear. Tumor... 
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2014, Volume 111, Issue 18, pp. 6810 - 6815
Dystroglycan (DG), a cell adhesion molecule well known to be essential for skeletal muscle integrity and formation of neuromuscular synapses, is also present... 
Brain | MicroRNA | Neurons | Protein synthesis | Small interfering RNA | Muscular dystrophies | Ligands | Physiological regulation | Dystroglycans | Synapses | Excitation-inhibition balance | AMPA receptors | Retardation | Dystrophin | Muscular dystrophy | ALPHA-DYSTROGLYCAN | RECEPTOR CLUSTERS | DYSTROPHIN-GLYCOPROTEIN COMPLEX | INHIBITORY SYNAPSES | MULTIDISCIPLINARY SCIENCES | dystrophin | DEFICIENT MDX MICE | HIPPOCAMPAL-NEURONS | muscular dystrophy | BRAIN DYSTROPHIN | DUCHENNE MUSCULAR-DYSTROPHY | GABA(A) RECEPTORS | excitation-inhibition balance | NEUROMUSCULAR-JUNCTION | retardation | Muscular Dystrophy, Animal - genetics | RNA, Small Interfering - genetics | Humans | Dystroglycans - metabolism | Homeostasis | N-Acetylglucosaminyltransferases - genetics | Male | Agrin - metabolism | Neuronal Plasticity - physiology | Synapses - metabolism | RNA Interference | Mice, Inbred mdx | Muscular Dystrophy, Animal - metabolism | Female | GABAergic Neurons - metabolism | Dystroglycans - antagonists & inhibitors | Rats | Glycosylation | Rats, Sprague-Dawley | N-Acetylglucosaminyltransferases - metabolism | Hippocampus - metabolism | Animals | N-Acetylglucosaminyltransferases - antagonists & inhibitors | Dystroglycans - genetics | Mice | Receptors, GABA-A - metabolism | Mutation | Neuroplasticity | GABA | Physiological aspects | Research | Neurological research | excitation–inhibition balance | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2007, Volume 104, Issue 24, pp. 10211 - 10216
Noninvasive monitoring of stem cells, using high-resolution molecular imaging, will be instrumental to improve clinical neural transplantation strategies. We... 
Brain | Imaging | Stem cells | Neural stem cells | Transplantation | Lesions | Cells | Tissue grafting | Animal migration behavior | Mesenchymal stem cells | Superparamagnetic iron oxide | Stem cell biology | MIGRATION | RAT | stem cell biology | MULTIDISCIPLINARY SCIENCES | SPINAL-CORD | CONTRAST AGENT | STROKE | superparamagnetic iron oxide | IN-VIVO | NEURONS | MAGNETIC-RESONANCE TRACKING | BRAIN | PARKINSONS-DISEASE | Iron - administration & dosage | Cell Proliferation | Humans | Oxides - chemistry | Injections, Intraventricular | Male | Neurons - cytology | Transplantation, Heterologous | Magnetics | Feasibility Studies | Nanoparticles | Stem Cell Transplantation | Stroke - pathology | Ferrosoferric Oxide | Monitoring, Physiologic | Neurons - transplantation | Dextrans | Neurons - physiology | Cell Differentiation | Magnetite Nanoparticles | Oxides - administration & dosage | Oxides - metabolism | Stroke - therapy | Animals, Newborn | Cell Survival | Iron - chemistry | Cells, Cultured | Rats | Brain Injuries - therapy | Mice, SCID | Iron - metabolism | Rats, Sprague-Dawley | Radiography | Cell Lineage | Magnetic Resonance Imaging | Neurons - diagnostic imaging | Patch-Clamp Techniques | Stroke - etiology | Animals | Biomarkers | Mice, Inbred NOD | Stem Cells - physiology | Mice | Brain Injuries - pathology | Cell Movement | Biological Sciences
Journal Article
Science, ISSN 0036-8075, 3/2002, Volume 295, Issue 5563, pp. 2282 - 2285
Activity-dependent modulation of synaptic efficacy in the brain contributes to neural circuit development and experience-dependent plasticity. Although glia... 
Brain | Neuroscience | Neurons | Astrocytes | Neuroglia | Antibodies | Cultured cells | Reports | Physical trauma | Behavioral neuroscience | Synapses | AMPA RECEPTOR ENDOCYTOSIS | NEURONS | IN-VITRO | TRANSMISSION | MULTIDISCIPLINARY SCIENCES | PLASTICITY
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2005, Volume 25, Issue 12, pp. 3219 - 3228
The proinflammatory cytokine tumor necrosis factor-alpha (TNF alpha) causes a rapid exocytosis of AMPA receptors in hippocampal pyramidal cells and is... 
GABA | Synaptic | Glutamate | Receptor | Trafficking | Cytokine | cytokine | NERVOUS-SYSTEM | BRAIN-INJURY | SYNAPTIC-TRANSMISSION | GLUR2 EXPRESSION | NEUROSCIENCES | synaptic | CULTURED HIPPOCAMPAL-NEURONS | CELL-DEATH | SIGNAL-TRANSDUCTION | receptor | RAT HIPPOCAMPUS | TNF-ALPHA | trafficking | DEPENDENT MECHANISM | glutamate | Excitatory Postsynaptic Potentials - radiation effects | Synaptic Transmission - physiology | Gene Expression - drug effects | Receptors, Tumor Necrosis Factor, Type I - immunology | Protein Transport - drug effects | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Excitatory Postsynaptic Potentials - physiology | Neural Inhibition - physiology | Neural Inhibition - radiation effects | Drug Interactions | Neurons - physiology | Synaptic Transmission - drug effects | Neurons - drug effects | Electric Stimulation - methods | Receptors, AMPA - metabolism | Animals, Newborn | Blotting, Western - methods | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Hippocampus - cytology | Antibodies - pharmacology | Immunohistochemistry - methods | Tumor Necrosis Factor-alpha - pharmacology | Animals | Receptors, GABA - metabolism | Biotinylation - methods | In Vitro Techniques | Dose-Response Relationship, Radiation | Neural Inhibition - drug effects | Cellular | Molecular
Journal Article
Nature Neuroscience, ISSN 1097-6256, 01/2003, Volume 6, Issue 1, pp. 15 - 16
Long-term potentiation (LTP) is thought to be critically involved not only in learning and memory, but also during the activity-dependent developmental phases... 
ACTIVATION | CALMODULIN-KINASE-II | ADENYLATE-CYCLASE | AREA CA1 | NEUROSCIENCES | BRAIN | Neural Pathways - enzymology | Synaptic Transmission - physiology | Peptide Fragments - pharmacology | Excitatory Postsynaptic Potentials - drug effects | Dose-Response Relationship, Drug | Excitatory Postsynaptic Potentials - physiology | Long-Term Potentiation - drug effects | Carrier Proteins - pharmacology | Long-Term Potentiation - physiology | Hippocampus - enzymology | Synaptic Transmission - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 | Hippocampus - growth & development | Animals, Newborn | Cyclic AMP-Dependent Protein Kinases - metabolism | Calcium-Calmodulin-Dependent Protein Kinases - genetics | Colforsin - pharmacology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Neural Pathways - growth & development | Enzyme Inhibitors - pharmacology | Intracellular Signaling Peptides and Proteins | Rats | Calcium-Calmodulin-Dependent Protein Kinases - antagonists & inhibitors | Mutation - genetics | Animals | Signal Transduction - drug effects | Neurons - enzymology | Signal Transduction - physiology | Calcium-Calmodulin-Dependent Protein Kinases - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Physiological aspects | Neural transmission | Research | Hippocampus (Brain) | Protein kinases
Journal Article
Current Opinion in Neurobiology, ISSN 0959-4388, 08/2019, Volume 57, pp. 179 - 185
The glial regulation of synaptic function provides important modulation of the synaptic and behavioral changes induced by drugs of abuse. In some cases, this... 
PLACE PREFERENCE | COCAINE-SEEKING | DOPAMINE | RECEPTOR STIMULATION INCREASES | TNF-ALPHA | AMPA RECEPTOR | UP-REGULATION | NEUROSCIENCES | GABAERGIC NEURONS | VENTRAL TEGMENTAL AREA | NUCLEUS-ACCUMBENS | Illegal drugs | Drug abuse | Models | Glutamate | Laws, regulations and rules
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 02/2010, Volume 107, Issue 8, pp. 3817 - 3822
Neurotrophin binding to the p75 neurotrophin receptor (p75 NTR ) activates neuronal apoptosis following adult central nervous system injury, but the underlying... 
Biological Sciences
Journal Article