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Science, ISSN 0036-8075, 11/2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases | Animal models | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 09/2010, Volume 13, Issue 9, pp. 1113 - 1119
Prolonged exposure to drugs of abuse, such as cannabinoids and opioids, leads to pharmacological tolerance and receptor desensitization in the nervous system.... 
PRECIPITATED WITHDRAWAL | NEUROPATHIC PAIN | ACID AMIDE HYDROLASE | IN-VIVO | ANANDAMIDE | DELTA-TETRAHYDROCANNABINOL | CANNABINOID RECEPTOR ADAPTATION | MICE | RAT-BRAIN | NEUROSCIENCES | KNOCKOUT MOUSE | Cannabinoid Receptor Modulators - metabolism | Analgesics - pharmacology | Monoacylglycerol Lipases - antagonists & inhibitors | Neuronal Plasticity - drug effects | Male | Benzodioxoles - administration & dosage | Pain - metabolism | Brain - physiology | Monoacylglycerol Lipases - genetics | Enzyme Inhibitors - administration & dosage | Monoacylglycerol Lipases - metabolism | Neuronal Plasticity - physiology | Piperidines - pharmacology | Pain - drug therapy | Female | Models, Animal | Endocannabinoids | Amidohydrolases - metabolism | Synapses - drug effects | Piperidines - administration & dosage | Synapses - physiology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Analgesics - administration & dosage | Amidohydrolases - antagonists & inhibitors | Mice, Transgenic | Mice, Knockout | Brain - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Animals | Cannabinoid Receptor Modulators - antagonists & inhibitors | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Benzodioxoles - pharmacology | Complications and side effects | Cannabinoids | Drug tolerance | Physiological aspects | Neuropharmacology | Research | Properties | Drug receptors | Index Medicus
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2012, Volume 165, Issue 8, pp. 2485 - 2496
BACKGROUND AND PURPOSE Inflammatory pain presents a problem of clinical relevance and often elicits allodynia, a condition in which non‐noxious stimuli are... 
inflammatory pain | allodynia | tetrahydrocannabinol | endocannabinoids | fatty acid amide hydrolase | anandamide | Allodynia | Fatty acid amide hydrolase | Inflammatory pain | Endocannabinoids | Anandamide | URB597 | ALPHA | MODEL | NEUROPATHIC PAIN | CB2 | CB1 | CANNABINOID RECEPTOR | 9-tetrahydrocannabinol | PHARMACOLOGY & PHARMACY | SENSORY NERVES | MODULATION | ANALGESIA | Hyperalgesia - chemically induced | Inflammation - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | Male | Lipopolysaccharides | Brain - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Piperidines - pharmacology | Inflammation - drug therapy | Female | Amidohydrolases - deficiency | Pyridines - therapeutic use | Hyperalgesia - metabolism | Amidohydrolases - genetics | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Amidohydrolases - antagonists & inhibitors | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Enzyme Inhibitors - therapeutic use | Mice, Knockout | Brain - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Hyperalgesia - drug therapy | Piperidines - therapeutic use | Peripheral Nervous System - metabolism | Mice | Pyridines - pharmacology | Peripheral Nervous System - drug effects | Index Medicus | Research Papers | Δ9-tetrahydrocannabinol | Themed Section
Journal Article
Journal Article
Drug and Alcohol Dependence, ISSN 0376-8716, 10/2018, Volume 191, pp. 14 - 24
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 12/2017, Volume 174, Issue 23, pp. 4523 - 4539
Journal Article
Journal of Neuroimmune Pharmacology, ISSN 1557-1890, 6/2015, Volume 10, Issue 2, pp. 364 - 370
Journal Article