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MOLECULAR PHARMACOLOGY, ISSN 0026-895X, 05/2019, Volume 95, Issue 5, pp. 468 - 474
Heat shock proteins (Hsp) are a class of stress-inducible proteins that mainly act as molecular protein chaperones. This chaperone activity is diverse,... 
MIGRATION | HSP90 INHIBITOR | COMPLEX | ACTIVATION | PHOSPHORYLATION | COUPLED RECEPTOR | KINASE | GROWTH | PHARMACOLOGY & PHARMACY | CELL-CYCLE ARREST | EXPRESSION | Neoplasms - metabolism | HSP90 Heat-Shock Proteins - metabolism | Ligands | Signal Transduction - physiology | Humans
Journal Article
Vitamins and hormones, ISSN 0083-6729, 2019, Volume 111, p. 91
Heat shock protein 90 (Hsp90) is one of the central signal transduction regulators of the cell. Via client interactions with hundreds of proteins, including... 
Journal Article
The journal of pain : official journal of the American Pain Society, ISSN 1526-5900, 06/2019
Numerous studies have demonstrated a physiological interaction between the mu opioid receptor (MOR) and delta opioid receptor (DOR) systems. A few studies have... 
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 07/2014, Volume 39, Issue 8, p. 1902
  The G protein-coupled serotonin 2A receptor (5-HT2AR) is a prominent target for atypical antipsychotic drugs, such as clozapine. Although clozapine is known... 
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 5771 - 13
Here, we report the chemical synthesis of two DPDPE analogues 7a (NOVA1) and 7b (NOVA2). This entailed the solid-phase synthesis of two enkephalin precursor... 
THERAPEUTICS | DESIGN | PUBLICATION | SPECIFICITY | SIDE-CHAIN | MULTIDISCIPLINARY SCIENCES | 1,2,3-TRIAZOLE | OPIOID PEPTIDE AGONISTS | RECEPTOR | BINDING | CYCLIZATION | Solid phase methods | Pain perception | Opioid receptors | Enkephalins
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1696 - 12
Acetylation of microtubules (MT) confers mechanical stability necessary for numerous functions including cell cycle and intracellular transport. Although alpha... 
EPITHELIAL-MESENCHYMAL TRANSITION | MIGRATION | PATHWAYS | TGF-BETA | MULTIDISCIPLINARY SCIENCES | TUBULIN ACETYLATION | KINASE | DYNAMICS | TRANSDUCTION | KAPPA-B | ACETYLTRANSFERASE | Acetyltransferases - metabolism | Humans | Cercopithecus aethiops | Male | Gene Knockdown Techniques | Microtubules - metabolism | Microtubules - drug effects | Dioxoles - pharmacology | Metabolic Networks and Pathways - physiology | Benzamides - pharmacology | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | MAP Kinase Kinase Kinases - genetics | Cell Proliferation - physiology | Microtubule Proteins - metabolism | Zearalenone - analogs & derivatives | MAP Kinase Kinase Kinases - metabolism | Mice, Inbred ICR | Acetylation - drug effects | Animals | Signal Transduction - drug effects | Cell Cycle - physiology | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | HeLa Cells | Metabolic Networks and Pathways - drug effects | Cell Cycle - drug effects | COS Cells | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | RNA, Small Interfering - metabolism | Zearalenone - pharmacology | Cell culture | Brain | Alanine | TAK1 protein | AKT protein | Catalytic activity | Metabolism | Signaling | Microtubules | Cell cycle | Acetyltransferase | Acetylation | Catalysis
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 07/2018, Volume 61, Issue 14, pp. 6075 - 6086
A major limitation in the study of the mu-delta opioid receptor heterodimer (MDOR) is that few selective pharmacological tools exist and no heteromer-selective... 
HETEROMERS | CHEMISTRY, MEDICINAL | PHARMACOPHORES | HETEROBIVALENT LIGANDS | TOLERANCE | DRUG DISCOVERY | CELL-SURFACE | TARGETS | NANOCARRIER SYSTEMS | DEPENDENCE | AGONIST
Journal Article
Circulation Research, ISSN 0009-7330, 04/2010, Volume 106, Issue 8, pp. 1434 - 1443
RATIONALE:Activation of p38 mitogen-activated protein kinase (MAPK) has a significant impact on cardiac gene expression, contractility, extracellular matrix... 
Heart failure | P38 MAPK | COX-2 | MK2 | Hypertrophy | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | hypertrophy | CARDIOMYOPATHY | ISOLATED RAT HEARTS | MESSENGER-RNA STABILITY | p38 MAPK | TUMOR-NECROSIS-FACTOR | PROTEIN-KINASE-2 | P38 | PATHWAY | IN-VIVO | Heart Failure | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Protein-Serine-Threonine Kinases - deficiency | Heart Failure - enzymology | Heart Failure - physiopathology | Cardiomegaly - pathology | Male | Intracellular Signaling Peptides and Proteins - metabolism | MAP Kinase Kinase 3 - genetics | RNA, Messenger - metabolism | Intracellular Signaling Peptides and Proteins - deficiency | Myocytes, Cardiac - enzymology | Ventricular Remodeling | Transfection | Time Factors | Cyclooxygenase 2 - genetics | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Myocardial Contraction | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Enzyme Stability | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Cardiomegaly - physiopathology | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Heart Failure - genetics | Mice, Transgenic | Cardiomegaly - enzymology | Heart Failure - pathology | Rats, Sprague-Dawley | MAP Kinase Kinase 3 - metabolism | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Cyclooxygenase 2 - metabolism | Fibrosis | Mice | Enzyme Activation | Cardiomegaly - genetics
Journal Article