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Human Gene Therapy, ISSN 1043-0342, 10/2010, Volume 21, Issue 10, pp. 1273 - 1285
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 03/2014, Volume 370, Issue 10, pp. 901 - 910
Journal Article
NATURE MEDICINE, ISSN 1078-8956, 07/2019, Volume 25, Issue 7, pp. 1131 - 1131
Huntington's disease (HD) is a dominantly inherited neurodegenerative disorder caused by a CAG trinucleotide expansion in the huntingtin gene (HTT), which... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | ZINC-FINGER PROTEINS | SUPPRESSION | PERFORMANCE EVALUATION | CELL BIOLOGY | INACTIVATION | DNA | MOUSE MODEL | HD GENE | DYNAMICS | EXPRESSION | BRAIN | Care and treatment | Rats as laboratory animals | Huntington's chorea | Models | Zinc finger proteins | Health aspects | Risk factors
Journal Article
Diabetes, ISSN 0012-1797, 02/2010, Volume 59, Issue 2, pp. 509 - 518
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2019, Volume 25, Issue 7, pp. 1131 - 1142
Huntington’s disease (HD) is a dominantly inherited neurodegenerative disorder caused by a CAG trinucleotide expansion in the huntingtin gene (HTT), which... 
Huntingtons disease | Brain | Transcription factors | Animal models | Polyglutamine | Huntingtin | Neurodegenerative diseases | Neurons | Medical treatment | Trinucleotide repeats | Gene expression | Huntington's disease | Zinc | Proteins | Gene silencing | Molecular modelling | Alleles | Fibroblasts | Zinc finger proteins | Trinucleotide repeat diseases
Journal Article
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2009, Volume 37, Issue 2, pp. 384 - 393
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e96137
Following spinal cord injury (SCI) there are drastic changes that occur in the spinal microvasculature, including ischemia, hemorrhage, endothelial cell death... 
FUNCTIONAL RECOVERY | EXPERIMENTAL DIABETIC-NEUROPATHY | CHRONIC PAIN | MULTIDISCIPLINARY SCIENCES | CONTUSION INJURY | RECEPTOR EXPRESSION | INTRASPINAL INJECTION | BLOOD-FLOW | TRANSCRIPTION FACTOR | ENDOTHELIAL GROWTH-FACTOR | NEUROLOGICAL FUNCTION | Vascular Endothelial Growth Factor A - biosynthesis | Zinc Fingers | Adenoviridae | Hyperalgesia - metabolism | Rats, Wistar | Spinal Cord Injuries - metabolism | Neovascularization, Physiologic - genetics | Spinal Cord Injuries - complications | Humans | Rats | Vascular Endothelial Growth Factor A - genetics | Hyperalgesia - pathology | Spinal Cord Injuries - pathology | Animals | Hyperalgesia - therapy | HEK293 Cells | Hyperalgesia - etiology | Female | Spinal Cord Injuries - therapy | Genetic Therapy - methods | Spinal cord injuries | Ischemia | Gene therapy | Vascular endothelial growth factor | Analysis | Genes | Bioengineering | Neuroprotection | Neurosciences | Spinal cord | Compression | Recovery of function | Diabetic neuropathy | Spinal cord injury | Hemorrhage | Blood | Proteins | Angiogenesis | Windows (intervals) | Pain | Health care networks | Rodents | RecA protein | Paralysis | Injuries | Pain perception | Preservation | Blood vessels | Gene expression | Vectors (Biology) | Zinc | Endothelial cells | Immunosuppression | Brain research | Cell death | Isoforms | Microvasculature
Journal Article
Genes and Development, ISSN 0890-9369, 04/1994, Volume 8, Issue 7, pp. 796 - 810
Journal Article