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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 33, pp. 13915 - 13920
Journal Article
Plos One, ISSN 1932-6203, 2011, Volume 6, Issue 9, pp. e24308 - e24308
To date there are 9 known diseases caused by an expanded polyglutamine repeat, with the most prevalent being Huntington's disease. Huntington's disease is a... 
NEURODEGENERATIVE DISEASE | RNA INTERFERENCE | POLYGLUTAMINE | REPEAT LENGTH | MULTIDISCIPLINARY SCIENCES | MACHADO-JOSEPH-DISEASE | MUTANT HUNTINGTIN | MUSCULAR-DYSTROPHY | NORMAL HUNTINGTIN FUNCTION | BINDING PROTEIN | ALLELE-SELECTIVE INHIBITION | Ataxin-1 | Ataxin-3 | Humans | Molecular Targeted Therapy | RNA, Messenger - metabolism | Ataxins | Peptides - metabolism | Myoclonic Epilepsies, Progressive - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Fibroblasts - metabolism | Spinocerebellar Ataxias - genetics | Cell Line | Oligonucleotides, Antisense - pharmacology | RNA, Messenger - genetics | Mutant Proteins - genetics | Repressor Proteins - genetics | Mutant Proteins - metabolism | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - therapy | Nuclear Proteins - metabolism | Fibroblasts - pathology | Nerve Tissue Proteins - genetics | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Huntingtin Protein | Fibroblasts - drug effects | Trinucleotide Repeat Expansion - genetics | RNA | Nervous system diseases | Glutamine | Huntingtons disease | Transcription | Huntingtin | Dentatorubral-pallidoluysian atrophy | Oligonucleotides | Genomes | Muscular dystrophy | Proteins | Atrophy | Ataxin | Neurodegeneration | Rodents | Spinocerebellar ataxia | Fibroblasts | Ataxia | Phosphorothioate | Mathematical models | Expansion | Trinucleotide repeat diseases | Lymphoblasts | Polyglutamine | Neurodegenerative diseases | Trinucleotide repeats | Antisense oligonucleotides | Huntington's disease | Hereditary diseases | Machado-Joseph disease | Acids | MicroRNAs | trinucleotide repeat diseases | phosphorothioate | Index Medicus
Journal Article
Plos One, ISSN 1932-6203, 2015, Volume 10, Issue 3, pp. e0121556 - e0121556
Myotonic Dystrophy type 1 (DM1) is a multisystemic disease caused by toxic RNA from a DMPK gene carrying an expanded (CTG*CAG)n repeat. Promising strategies... 
DISRUPTION | RNA TOXICITY | REPEAT | ABNORMALITIES | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MUSCLE-FIBERS | DISEASE | MUSCULAR-DYSTROPHY | ANIMAL-MODELS | MDX MICE | Myotonic Dystrophy - metabolism | Calcium - metabolism | Humans | Mice, Inbred C57BL | Middle Aged | Cell Membrane Permeability | Male | Evans Blue - pharmacokinetics | Animals | Dystrophin - genetics | Myotonic Dystrophy - drug therapy | Aged, 80 and over | Adult | Female | Aged | Cell Membrane - metabolism | Mice | Child | Dystrophin - metabolism | Drugs | Drug delivery systems | RNA | Myotonic dystrophy | Utrophin | Muscles | Cell membranes | Dystrophin | Vehicles | Protein binding | Brain | Therapy | Animal models | Membranes | Calcium | Disease | Creatine kinase | Pathogenesis | Membrane structure | Oligonucleotides | Biology | Drug delivery | Kinases | Creatine | Cell surface | Muscular dystrophy | Proteins | DMPK protein | Rodents | Animal tissues | Life sciences | Toxic diseases | Enzymes | Polyglutamine | Calcium (intracellular) | RNA-protein interactions | Splicing | Cardiac muscle | Fitness equipment | Trinucleotide repeats | Antisense oligonucleotides | Permeability | Cell membranes (biology) | Ribonucleic acid--RNA | Patients | Skeletal muscle | Membrane proteins | Biopsy | Dystrophy | Aberration | Protein structure | Integrity | Index Medicus | Ribonucleic acid
Journal Article
Nucleic Acid Therapeutics, ISSN 2159-3337, 06/2017, Volume 27, Issue 3, pp. 144 - 158
Clinical efficacy of antisense oligonucleotides (AONs) for the treatment of neuromuscular disorders depends on efficient cellular uptake and proper... 
Gene silencing | Antisense | Delivery | RNA | Splicing | MEDICINE, RESEARCH & EXPERIMENTAL | delivery | SYSTEMIC DELIVERY | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | gene silencing | MUSCLE-CELLS | CELLULAR UPTAKE | MDX MICE | PHOSPHOROTHIOATE | RNA TOXICITY | MOUSE MODEL | GENE-EXPRESSION | DUCHENNE MUSCULAR-DYSTROPHY | splicing | MYOTONIC-DYSTROPHY | antisense | Cell Line | Oligonucleotides, Antisense - drug effects | Cell Proliferation | Oligonucleotides, Antisense - pharmacology | Humans | Oligonucleotides, Antisense - chemistry | Mice, Transgenic | RNA - drug effects | Clathrin - metabolism | Hydrazones - pharmacology | RNA - genetics | Chloroquine - pharmacology | Microscopy, Confocal | Endocytosis | Animals | Analysis of Variance | Muscle Fibers, Skeletal - chemistry | Oligonucleotides, Antisense - chemical synthesis | Endosomes - chemistry | Cell Differentiation | Cell Nucleus - chemistry | Mice | Muscular Dystrophy, Duchenne - therapy | RNA - metabolism | Cell culture | Clathrin | Myotubes | Chloroquine | Oligonucleotides | Disorders | Routing | Antisense oligonucleotides | Gene expression | Ribonucleic acid--RNA | Low level | Biological activity | Myoblasts | Cellular biology | Microscopy | Membrane trafficking | Phosphorothioate | Intracellular | Polymers | Position (location) | Localization | Neuromuscular system | In vitro methods and tests
Journal Article
The Journal of Bone and Joint Surgery, ISSN 0021-9355, 05/2019, Volume 101, Issue 9, pp. 787 - 796
BACKGROUND:There is no consensus as to whether displaced extra-articular distal radial fractures should be treated operatively or nonoperatively. We compared... 
SURGERY | OLDER | NONOPERATIVE TREATMENT | OUTCOMES | ORTHOPEDICS | AGE
Journal Article
Journal Article
Journal Article
Journal Article
British Journal of Management, ISSN 1045-3172, 12/2012, Volume 23, Issue 4, pp. 561 - 574
Journal Article
12.