1979, Goma serekuto, Volume S001, 286
Book
1999, ISBN 9784472301612, 156
Book
Earth, Planets and Space, ISSN 1343-8832, 12/2016, Volume 68, Issue 1
Journal Article
Earth, Planets and Space, ISSN 1343-8832, 12/2016, Volume 68, Issue 1
Journal Article
Earth, Planets and Space, ISSN 1343-8832, 12/2017, Volume 69, Issue 1
Journal Article
World Neurosurgery, ISSN 1878-8750, 10/2019, Volume 130, pp. e26 - e46
The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging societies....
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | SURGERY | CARE | CLINICAL NEUROLOGY | IMPACT | CLINICAL-VARIABLES | HOSPITAL VOLUME | GENDER | COMA
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | SURGERY | CARE | CLINICAL NEUROLOGY | IMPACT | CLINICAL-VARIABLES | HOSPITAL VOLUME | GENDER | COMA
Journal Article
Clinical Nutrition, ISSN 0261-5614, 02/2018, Volume 37, Issue 1, pp. 408 - 409
Journal Article
Molecular Brain, ISSN 1756-6606, 2012, Volume 5, Issue 1, pp. 35 - 35
Background: Parkinson's disease (PD) is a neurodegenerative disease characterized by selective degeneration of dopaminergic neurons in the substantia nigra...
Oxidative stress | Mitochondria | Parkinson's disease | Induced pluripotent stem cells | α-synuclein | Parkin | PROTEIN | DISORDERS | MECHANISMS | DEFICIENT MICE | DOPAMINERGIC-NEURONS | alpha-synuclein | NEUROSCIENCES | FIBROBLASTS | PLURIPOTENT STEM-CELLS | RECESSIVE JUVENILE PARKINSONISM | DISEASE | LEWY BODIES | Neurons - pathology | Humans | Middle Aged | Male | Membrane Potential, Mitochondrial - drug effects | Mitochondria - ultrastructure | Brain - metabolism | Neurons - ultrastructure | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Adult | Female | Neurons - metabolism | Parkinson Disease - metabolism | Lewy Bodies - metabolism | Neurons - drug effects | Lewy Bodies - pathology | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Parkinson Disease - pathology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - ultrastructure | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Brain - drug effects | Mitochondrial Turnover - drug effects | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Brain - pathology | Aged | Mice | Oxidative Stress - drug effects | Postmortem Changes | alpha-Synuclein - metabolism | Parkinson’s disease
Oxidative stress | Mitochondria | Parkinson's disease | Induced pluripotent stem cells | α-synuclein | Parkin | PROTEIN | DISORDERS | MECHANISMS | DEFICIENT MICE | DOPAMINERGIC-NEURONS | alpha-synuclein | NEUROSCIENCES | FIBROBLASTS | PLURIPOTENT STEM-CELLS | RECESSIVE JUVENILE PARKINSONISM | DISEASE | LEWY BODIES | Neurons - pathology | Humans | Middle Aged | Male | Membrane Potential, Mitochondrial - drug effects | Mitochondria - ultrastructure | Brain - metabolism | Neurons - ultrastructure | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Adult | Female | Neurons - metabolism | Parkinson Disease - metabolism | Lewy Bodies - metabolism | Neurons - drug effects | Lewy Bodies - pathology | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Parkinson Disease - pathology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - ultrastructure | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Brain - drug effects | Mitochondrial Turnover - drug effects | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Brain - pathology | Aged | Mice | Oxidative Stress - drug effects | Postmortem Changes | alpha-Synuclein - metabolism | Parkinson’s disease
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2012, Volume 18, Issue 3, pp. 378 - 381
Through an integrated molecular-and histopathology-based screening system, we performed a screening for fusions of anaplastic lymphoma kinase (ALK) and c-ros...
MEDICINE, RESEARCH & EXPERIMENTAL | ONCOGENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | EML4-ALK FUSION | TRANSFORMING GENE | IDENTIFICATION | REARRANGEMENTS | RADIATION | IMMUNOHISTOCHEMISTRY | CELL BIOLOGY | Adenocarcinoma - pathology | Prognosis | Adenocarcinoma of Lung | Age Factors | Cytoskeletal Proteins - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Protein-Tyrosine Kinases - genetics | Anaplastic Lymphoma Kinase | Piperidines - pharmacology | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Oncogene Proteins, Fusion - genetics | Sex Factors | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | Neoplasm Staging | Quinazolines - pharmacology | 3T3 Cells | Proto-Oncogene Proteins c-ret - genetics | Care and treatment | Lung cancer | Protein tyrosine kinase | Physiological aspects | Genetic aspects | Research | Lung cancer, Non-small cell | Risk factors | Proteins | Pathology | Cellular biology | Kinases
MEDICINE, RESEARCH & EXPERIMENTAL | ONCOGENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | EML4-ALK FUSION | TRANSFORMING GENE | IDENTIFICATION | REARRANGEMENTS | RADIATION | IMMUNOHISTOCHEMISTRY | CELL BIOLOGY | Adenocarcinoma - pathology | Prognosis | Adenocarcinoma of Lung | Age Factors | Cytoskeletal Proteins - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Protein-Tyrosine Kinases - genetics | Anaplastic Lymphoma Kinase | Piperidines - pharmacology | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Oncogene Proteins, Fusion - genetics | Sex Factors | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | Neoplasm Staging | Quinazolines - pharmacology | 3T3 Cells | Proto-Oncogene Proteins c-ret - genetics | Care and treatment | Lung cancer | Protein tyrosine kinase | Physiological aspects | Genetic aspects | Research | Lung cancer, Non-small cell | Risk factors | Proteins | Pathology | Cellular biology | Kinases
Journal Article
11.
Full Text
Alternatively activated macrophages determine repair of the infarcted adult murine heart
Journal of Clinical Investigation, ISSN 0021-9738, 06/2016, Volume 126, Issue 6, pp. 2151 - 2166
Alternatively activated (also known as M2) macrophages are involved in the repair of various types of organs. However, the contribution of M2 macrophages to...
MEDICINE, RESEARCH & EXPERIMENTAL | THERAPY | MYOCARDIAL-INFARCTION | EPITHELIAL-CELLS | PATHWAY | INFLAMMATION | IN-VIVO | CARDIAC MACROPHAGES | STEADY-STATE | POLARIZATION | FIBROBLASTS | Protein-Serine-Threonine Kinases - deficiency | Myocardium - immunology | Fibroblasts - physiology | Intracellular Signaling Peptides and Proteins - immunology | Mice, 129 Strain | Myocardial Infarction - immunology | Intracellular Signaling Peptides and Proteins - deficiency | Regeneration - genetics | Myocardial Infarction - pathology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Myocardial Infarction - physiopathology | Intracellular Signaling Peptides and Proteins - genetics | Macrophages - immunology | Macrophages - physiology | Macrophages - classification | Connective Tissue - pathology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Connective Tissue - immunology | Myocardium - pathology | Fibroblasts - pathology | Regeneration - physiology | Macrophage Activation | Mice, Knockout | Animals | Interleukin-4 - administration & dosage | Connective Tissue - physiopathology | Fibroblasts - immunology | Protein-Serine-Threonine Kinases - immunology | Mice | Regeneration - immunology | Care and treatment | Biological products | Innovations | Genetic aspects | Macrophages | Phosphotransferases | Health aspects | Heart attack | Studies | Heart | Biomedical research | Research & development--R&D | Fibroblasts | Software | Kinases | Laboratory animals
MEDICINE, RESEARCH & EXPERIMENTAL | THERAPY | MYOCARDIAL-INFARCTION | EPITHELIAL-CELLS | PATHWAY | INFLAMMATION | IN-VIVO | CARDIAC MACROPHAGES | STEADY-STATE | POLARIZATION | FIBROBLASTS | Protein-Serine-Threonine Kinases - deficiency | Myocardium - immunology | Fibroblasts - physiology | Intracellular Signaling Peptides and Proteins - immunology | Mice, 129 Strain | Myocardial Infarction - immunology | Intracellular Signaling Peptides and Proteins - deficiency | Regeneration - genetics | Myocardial Infarction - pathology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Myocardial Infarction - physiopathology | Intracellular Signaling Peptides and Proteins - genetics | Macrophages - immunology | Macrophages - physiology | Macrophages - classification | Connective Tissue - pathology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Connective Tissue - immunology | Myocardium - pathology | Fibroblasts - pathology | Regeneration - physiology | Macrophage Activation | Mice, Knockout | Animals | Interleukin-4 - administration & dosage | Connective Tissue - physiopathology | Fibroblasts - immunology | Protein-Serine-Threonine Kinases - immunology | Mice | Regeneration - immunology | Care and treatment | Biological products | Innovations | Genetic aspects | Macrophages | Phosphotransferases | Health aspects | Heart attack | Studies | Heart | Biomedical research | Research & development--R&D | Fibroblasts | Software | Kinases | Laboratory animals
Journal Article
Journal of Obstetrics and Gynaecology Research, ISSN 1341-8076, 01/2019, Volume 45, Issue 1, pp. 96 - 103
Aim To compare serum levels of angiogenesis‐related factors between 14 women with HELLP (hemolysis, elevated liver enzymes and low platelet count) syndrome and...
placental growth factor | reference range | soluble fms‐like tyrosine kinase 1 | hemolysis, elevated liver enzymes and low platelet count syndrome | acute fatty liver of pregnancy | duodenal ulcer | soluble fms-like tyrosine kinase 1 | DIAGNOSIS | HYPERTENSIVE DISORDERS | HEMOLYSIS | PREECLAMPSIA | ANGIOGENIC FACTORS | OBSTETRICS & GYNECOLOGY | PATHOGENESIS | ENZYMES | PERFORATION | Tyrosine | Women | Thrombocytopenia | Amplified fragment length polymorphism | Transformation | Gestation | Kinases | Pregnancy | Serum levels | Angiogenesis | Fatty liver | Placenta | Protein-tyrosine kinase
placental growth factor | reference range | soluble fms‐like tyrosine kinase 1 | hemolysis, elevated liver enzymes and low platelet count syndrome | acute fatty liver of pregnancy | duodenal ulcer | soluble fms-like tyrosine kinase 1 | DIAGNOSIS | HYPERTENSIVE DISORDERS | HEMOLYSIS | PREECLAMPSIA | ANGIOGENIC FACTORS | OBSTETRICS & GYNECOLOGY | PATHOGENESIS | ENZYMES | PERFORATION | Tyrosine | Women | Thrombocytopenia | Amplified fragment length polymorphism | Transformation | Gestation | Kinases | Pregnancy | Serum levels | Angiogenesis | Fatty liver | Placenta | Protein-tyrosine kinase
Journal Article
BMC Genomics, ISSN 1471-2164, 03/2010, Volume 11, Issue 1, pp. 210 - 210
Background: The Solanaceae family includes several economically important vegetable crops. The tomato (Solanum lycopersicum) is regarded as a model plant of...
TRANSCRIPTION FACTORS | COLLECTION | SEQUENCES | DATABASE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ANNOTATION | INFORMATION | GENES | GENETICS & HEREDITY | LIBRARY | CLONES | RESOURCE | DNA, Complementary - analysis | Lycopersicon esculentum - genetics | Gene Library | DNA, Plant - analysis | Genomics | Library collections | Studies | Genomes | Genes | Deoxyribonucleic acid--DNA | Index Medicus
TRANSCRIPTION FACTORS | COLLECTION | SEQUENCES | DATABASE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ANNOTATION | INFORMATION | GENES | GENETICS & HEREDITY | LIBRARY | CLONES | RESOURCE | DNA, Complementary - analysis | Lycopersicon esculentum - genetics | Gene Library | DNA, Plant - analysis | Genomics | Library collections | Studies | Genomes | Genes | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2008, Volume 105, Issue 3, pp. 1003 - 1008
Infection with cagA-positive Helicobacter pylori is associated with gastric adenocarcinoma and gastric mucosa-associated lymphoid tissue (MALT) lymphoma of B...
Adenocarcinoma | Stomach | Phosphorylation | Helicobacter pylori | Epithelial cells | Transgenic animals | Mice | Infections | Bone marrow cells | Tumors | Transgenic mouse | Bacterial oncoprotein | SHP-2 TYROSINE PHOSPHATASE | PROTEIN | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | transgenic mouse | CELL DEVELOPMENT | CANCER | bacterial oncoprotein | BIOLOGICAL-ACTIVITY | ATROPHIC GASTRITIS | PTPN11 MUTATIONS | INFECTION | MONGOLIAN GERBILS | Gastrointestinal Neoplasms - genetics | Bacterial Proteins - genetics | Gene Expression Regulation, Neoplastic | Helicobacter pylori - genetics | Helicobacter pylori - pathogenicity | Mice, Transgenic | Hematologic Neoplasms - pathology | Antigens, Bacterial - genetics | Cell Transformation, Neoplastic - metabolism | Phosphotyrosine - metabolism | Gastrointestinal Neoplasms - pathology | Animals | Helicobacter pylori - metabolism | Cell Transformation, Neoplastic - genetics | Bacterial Proteins - metabolism | Hematologic Neoplasms - genetics | Cell Transformation, Neoplastic - pathology | Antigens, Bacterial - metabolism | Hematologic Neoplasms - metabolism | Gastrointestinal Neoplasms - metabolism | Gastrointestinal tumors | Causes of | Influence | Genetic aspects | Hematopoietic system | Gene expression | Biological Sciences
Adenocarcinoma | Stomach | Phosphorylation | Helicobacter pylori | Epithelial cells | Transgenic animals | Mice | Infections | Bone marrow cells | Tumors | Transgenic mouse | Bacterial oncoprotein | SHP-2 TYROSINE PHOSPHATASE | PROTEIN | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | transgenic mouse | CELL DEVELOPMENT | CANCER | bacterial oncoprotein | BIOLOGICAL-ACTIVITY | ATROPHIC GASTRITIS | PTPN11 MUTATIONS | INFECTION | MONGOLIAN GERBILS | Gastrointestinal Neoplasms - genetics | Bacterial Proteins - genetics | Gene Expression Regulation, Neoplastic | Helicobacter pylori - genetics | Helicobacter pylori - pathogenicity | Mice, Transgenic | Hematologic Neoplasms - pathology | Antigens, Bacterial - genetics | Cell Transformation, Neoplastic - metabolism | Phosphotyrosine - metabolism | Gastrointestinal Neoplasms - pathology | Animals | Helicobacter pylori - metabolism | Cell Transformation, Neoplastic - genetics | Bacterial Proteins - metabolism | Hematologic Neoplasms - genetics | Cell Transformation, Neoplastic - pathology | Antigens, Bacterial - metabolism | Hematologic Neoplasms - metabolism | Gastrointestinal Neoplasms - metabolism | Gastrointestinal tumors | Causes of | Influence | Genetic aspects | Hematopoietic system | Gene expression | Biological Sciences
Journal Article
Microscopy, ISSN 2050-5698, 11/2015, Volume 64, Issue suppl 1, pp. i131.1 - i131
Journal Article
16.
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Large tricuspid valve vegetation secondary to Austrian syndrome in a young healthy adult
The Annals of Thoracic Surgery, ISSN 0003-4975, 09/2019
Journal Article
IEEE Transactions on Biomedical Engineering, ISSN 0018-9294, 06/2016, Volume 63, Issue 6, pp. 1321 - 1332
Objective: The present study proposes a new epileptic seizure prediction method through integrating heart rate variability (HRV) analysis and an anomaly...
Rail to rail inputs | Frequency-domain analysis | Seizure prediction | Process control | Epilepsy | Heart rate variability analysis | Prediction methods | Feature extraction | Multivariate statistical process control | Heart rate variability | Monitoring | seizure prediction | multivariate statistical process control (MSPC) | heart rate variability analysis | SLEEP | AROUSAL | PRINCIPAL COMPONENT ANALYSIS | ENGINEERING, BIOMEDICAL | TACHYCARDIA | STANDARDS | HUMANS | SYSTEMS | Multivariate Analysis | Humans | Middle Aged | Male | Epilepsy - physiopathology | Epilepsy - diagnosis | Young Adult | Adolescent | Heart Rate - physiology | Signal Processing, Computer-Assisted | Adult | Female | Electrocardiography - classification | Databases, Factual | Statistical process control | Usage | Time-domain analysis | Analysis | Neurophysiology | Heart rate | Seizing | Autonomic nervous system | Sensors | Patients | Anomalies
Rail to rail inputs | Frequency-domain analysis | Seizure prediction | Process control | Epilepsy | Heart rate variability analysis | Prediction methods | Feature extraction | Multivariate statistical process control | Heart rate variability | Monitoring | seizure prediction | multivariate statistical process control (MSPC) | heart rate variability analysis | SLEEP | AROUSAL | PRINCIPAL COMPONENT ANALYSIS | ENGINEERING, BIOMEDICAL | TACHYCARDIA | STANDARDS | HUMANS | SYSTEMS | Multivariate Analysis | Humans | Middle Aged | Male | Epilepsy - physiopathology | Epilepsy - diagnosis | Young Adult | Adolescent | Heart Rate - physiology | Signal Processing, Computer-Assisted | Adult | Female | Electrocardiography - classification | Databases, Factual | Statistical process control | Usage | Time-domain analysis | Analysis | Neurophysiology | Heart rate | Seizing | Autonomic nervous system | Sensors | Patients | Anomalies
Journal Article