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Current Drug Targets, ISSN 1389-4501, 05/2012, Volume 13, Issue 5, pp. 623 - 630
Journal Article
Journal of Alzheimer's disease : JAD, ISSN 1387-2877, 02/2019, Volume 68, Issue 1, pp. 323 - 337
We previously demonstrated that microglia play an essential role in clearance of amyloid-β (Aβ) in Alzheimer's disease (AD)-like pathology. Our prior work also... 
Clathrin | TLR4 protein | CD14 antigen | Microglia | Lipopolysaccharides | Receptors | Endocytosis | Internalization | Toll-like receptors | Amyloid | Alzheimer's disease | Phagocytosis | Age
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2017, Volume 55, Issue 3, pp. 1235 - 1247
In response to changes of the central nervous system environment, microglia are capable of acquiring diverse phenotypes for cytotoxic or immune regulation and... 
SOCS3 | IL6 | STAT3 | APPswe/PS1dE9 mice | Inflammation | Amyloid-β | Alzheimer's disease | Microglia | NADPH-OXIDASE | ALZHEIMERS-DISEASE | INTERLEUKIN-6 | NEUROSCIENCES | PEPTIDE | amyloid-beta | AMYLOID-BETA CLEARANCE | inflammation | NEUROINFLAMMATION | TNF-ALPHA | TRANSGENIC MOUSE MODEL | microglia | EXPRESSION | BRAIN | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Neurons - pathology | Humans | Male | Alzheimer Disease - pathology | Amyloid beta-Protein Precursor - metabolism | Microglia - pathology | Female | Alzheimer Disease - immunology | Interleukin-6 - metabolism | Disease Models, Animal | Lipopolysaccharides - toxicity | Amyloid beta-Peptides - toxicity | Mice, Inbred C57BL | Cells, Cultured | Neurons - immunology | Presenilin-1 - genetics | Mice, Transgenic | Amyloid beta-Protein Precursor - genetics | Animals | Presenilin-1 - metabolism | Brain - pathology | Brain - immunology | Suppressor of Cytokine Signaling 3 Protein - metabolism | Oxidative stress | Reactive oxygen species | Interleukin | Central nervous system | Immune clearance | Cytotoxicity | Activation | Lipopolysaccharides | Interleukin 6 | SOCS-3 protein | Rodents | Amyloid | Phenotypes | Cytokines | Neurodegenerative diseases | Immunoregulation | Markers | Tumor necrosis factor-α | Gene expression | Signaling | Mice | In vivo methods and tests | Phagocytosis
Journal Article
Journal Article
Current Drug Targets, ISSN 1389-4501, 05/2012, Volume 13, Issue 5, pp. 623 - 630
Parkinson's disease (PD) is characterized by relatively selective degeneration of dopaminergic neurons in the substantia nigra and loss of dopamine in the... 
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 03/2013, Volume 91, Issue 3, pp. 462 - 471
  To explore a novel therapy against Parkinson's disease through enhancement of [alpha]7 nicotinic acetylcholine receptor (nAChR), we evaluated the... 
Journal Article
Journal Article
JOURNAL OF ALZHEIMERS DISEASE, ISSN 1387-2877, 2019, Volume 72, Issue 3, pp. 867 - 884
Mesenchymal stem cells (MSC) are increasingly being studied as a source of cell therapy for neurodegenerative diseases, and several groups have reported their... 
CLEARANCE | ALZHEIMERS-DISEASE MICE | MEMORY DEFICITS | electron paramagnetic resonance imaging | NEUROSCIENCES | INTRAVENOUS-INFUSION | mesenchymal stem cells | A-BETA | MITOCHONDRIAL DYSFUNCTION | CD14 | TRANSGENIC MOUSE MODEL | Alzheimer's disease | microglia | EXPRESSION | oxidative stress | MARROW STROMAL CELLS | BRAIN
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2019, Volume 67, Issue 3, pp. 1079 - 1087
Alzheimer's disease (AD) is one of the most common neurodegenerative diseases responsible for progressive dementia. Deposition of amyloid-beta (A beta) in the... 
electron paramagnetic resonance | reactive oxygen special | mitochondria | amyloid-β | redox status | Alzheimer's disease | oxidative stress | PATHOLOGY | NEUROSCIENCES | DAMAGE | amyloid-beta | ANTIOXIDANT | HYPOTHESIS | A-BETA | DYSFUNCTION | DEFICITS | EXTRACT
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 01/2015, Volume 44, Issue 2, pp. 409 - 423
We previously reported that activated microglia are involved in amyloid- (A) clearance and that stimulation of 7 nicotinic acetylcholine receptors (nAChR) in... 
Journal Article
Neurology and Clinical Neuroscience, ISSN 2049-4173, 07/2014, Volume 2, Issue 4, pp. 109 - 111
Oculopharyngeal muscular dystrophy is typically characterized by late‐onset, slowly progressive bilateral ptosis and dysphagia. We report a 64‐year‐old... 
myasthenia gravis | diurnal fluctuation | poly(A) binding protein nuclear 1 | ptosis | oculopharyngeal muscular dystrophy
Journal Article
Clinical Neurology, ISSN 0009-918X, 2016, Volume 56, Issue 10, pp. 684 - 689
Journal Article