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Diabetes, ISSN 0012-1797, 12/2010, Volume 59, Issue 12, pp. 3023 - 3031
Journal Article
Diabetes, ISSN 0012-1797, 09/2009, Volume 58, Issue 9, pp. 1971 - 1977
Journal Article
Journal Article
Wound Repair and Regeneration, ISSN 1067-1927, 01/2013, Volume 21, Issue 1, pp. 88 - 102
Journal Article
Wound Repair and Regeneration, ISSN 1067-1927, 09/2012, Volume 20, Issue 5, pp. 688 - 706
Journal Article
Cardiovascular Research, ISSN 0008-6363, 8/2009, Volume 83, Issue 3, pp. 566 - 574
Aims Polycystic ovary syndrome (PCOS) is associated with insulin resistance (IR), obesity, and cardiovascular complications. Thrombospondin-1 (TSP-1) is a... 
Thrombospondin-1 | Angiogenesis | Polycystic ovary syndrome | Adipokine | Metformin | POLYCYSTIC-OVARY-SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | WOMEN | HUMAN ADIPOSE-TISSUE | OBESITY | INSULIN-RESISTANCE | GLUCOSE | INFLAMMATION | PAI-1
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0123016
Notch signalling is critical for haemopoietic stem cell (HSC) self-renewal and survival. The role of Notch signalling has been reported recently in chronic... 
CHRONIC MYELOGENOUS LEUKEMIA | PROGENITOR CELLS | KINASE-ACTIVITY | LYMPHOMA-CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | BLAST CRISIS | GAMMA-SECRETASE INHIBITORS | TRANSCRIPTION FACTOR | HEMATOPOIETIC STEM | INTRACELLULAR DOMAIN | Transcription Factor HES-1 | Antigens, CD34 - metabolism | Apoptosis - drug effects | Homeodomain Proteins - metabolism | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Receptor, Notch2 - genetics | Imatinib Mesylate - therapeutic use | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplastic Stem Cells - metabolism | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Basic Helix-Loop-Helix Transcription Factors - genetics | RNA, Messenger - genetics | Receptor, Notch2 - metabolism | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Homeodomain Proteins - genetics | Fusion Proteins, bcr-abl - genetics | Protein Kinase Inhibitors - therapeutic use | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Cell Proliferation - drug effects | Cell Cycle - drug effects | Receptor, Notch1 - genetics | Fusion Proteins, bcr-abl - metabolism | BCR protein | Histone deacetylase | Leukemia | Abl gene | BCR gene | Signal transduction | Antagonism | Bone marrow | Inhibition | Fusion protein | Protein-tyrosine kinase | Tyrosine | CD34 antigen | Imatinib | Cell survival | Abl protein | Chronic myeloid leukemia | Valproic acid | Gene expression | Patients | Signaling | Inhibitors | Cell lines | Stem cells | Cells (biology) | Notch protein
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 05/2013, Volume 133, Issue 5, pp. 1340 - 1350
Mammalian target of rapamycin (mTOR) is essential in controlling several cellular functions. This pathway is dysregulated in keloid disease (KD). KD is a... 
MAMMALIAN TARGET | COLLAGEN | PATHWAY | DISEASE | ACUTE MYELOID-LEUKEMIA | KINASE INHIBITOR | ANTITUMOR-ACTIVITY | MTOR INHIBITORS | CANCER-THERAPY | CELL-DEATH | DERMATOLOGY | Keloid - drug therapy | Keloid - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Immunosuppressive Agents - therapeutic use | Middle Aged | Male | Phosphatidylinositol 3-Kinases - metabolism | Mechanistic Target of Rapamycin Complex 2 | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Young Adult | Multiprotein Complexes - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Adult | Female | Proto-Oncogene Proteins c-akt - metabolism | Immunosuppressive Agents - pharmacology | TOR Serine-Threonine Kinases - drug effects | Morpholines - therapeutic use | Multiprotein Complexes - drug effects | Sirolimus - therapeutic use | Keloid - pathology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | Pyrimidines - pharmacology | Enzyme Inhibitors - therapeutic use | Sirolimus - pharmacology | Cell Movement - drug effects | Signal Transduction - drug effects | Pyrimidines - therapeutic use | Adolescent | Aged | Cell Proliferation - drug effects | In Vitro Techniques | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31430
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0123016
Journal Article
Pakistan Journal of Botany, ISSN 0556-3321, 12/2014, Volume 46, Issue 6, pp. 2135 - 2142
Combining ability was studied for yield and yield contributing traits in 5 x 5 diallel cross in Brassica rapa (L.) ssp. dichotoma (Roxb.) Hanelt. Primary... 
Combining ability | Heritability | Yield | Brassica rapa | Heterosis | GENETIC DIVERSITY | NAPUS L | COMPONENTS | PATTERNS | DIALLEL CROSSES | PLANT SCIENCES
Journal Article