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cancer (10) 10
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studies (3) 3
thiazolidinediones (3) 3
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vascular endothelial growth factor a - genetics (3) 3
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PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 12, pp. e28354 - e28354
Background: Ciglitazone belongs to the thiazolidinediones class of antidiabetic drug family and is a high-affinity ligand for the Peroxisome... 
SENSITIZES TUMOR-CELLS | ACTIVATED RECEPTOR-GAMMA | HUMAN UROTHELIAL CELLS | PPAR | SURVIVIN | BIOLOGY | DOWN-REGULATION | C-FLIP | DEATH | LIGAND | PROGRESSION | Apoptosis - drug effects | Humans | Transcriptional Activation - drug effects | Urinary Bladder Neoplasms - enzymology | PPAR gamma - metabolism | G2 Phase - drug effects | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Receptors, Death Domain - metabolism | Thiazolidinediones - therapeutic use | Caspases - metabolism | Neoplasm Grading | Urinary Bladder Neoplasms - pathology | Cell Membrane - metabolism | Inhibitor of Apoptosis Proteins - metabolism | Thiazolidinediones - pharmacology | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Cell Membrane - drug effects | Hypoglycemic Agents - therapeutic use | Urinary Bladder Neoplasms - drug therapy | Enzyme Activation - drug effects | TNF-Related Apoptosis-Inducing Ligand - metabolism | Hypoglycemic Agents - pharmacology | Up-Regulation - drug effects | Xenograft Model Antitumor Assays | Animals | Mitosis - drug effects | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Line, Tumor | Mice | Proteasome Endopeptidase Complex - metabolism | Prevention | Cancer cells | Development and progression | Tumor proteins | Bladder cancer | Thiazolidinediones | Cancer | Apoptosis | Phosphorylation | p53 Protein | Bladder | Activation | Metastasis | Kinases | Cancer therapies | Cyclin B1 | Proteins | Signal transduction | Cell growth | Pathways | Urinary bladder | FLIP protein | Xenografts | Cell cycle | Down-regulation | Diabetes mellitus | Cell division | Caspase | c-FLIP protein | Pharmacology | Survivin | Tumor cell lines | Chemical compounds | Studies | Signaling | Cyclin-dependent kinase inhibitor p21 | Molecular modelling | Cell death | Medical prognosis | Cell lines | Cyclin-dependent kinase inhibitor p27 | Biomarkers | Ligands | TRAIL protein | Tumors | Index Medicus | Up-Regulation | Cell Membrane | Mitosis | Transcriptional Activation | BH3 Interacting Domain Death Agonist Protein | Urinary Bladder Neoplasms | Proteasome Endopeptidase Complex | PPAR gamma | Life Sciences | Hypoglycemic Agents | TNF-Related Apoptosis-Inducing Ligand | G2 Phase | Inhibitor of Apoptosis Proteins | Signal Transduction | Antineoplastic Combined Chemotherapy Protocols | Receptors, Death Domain | CASP8 and FADD-Like Apoptosis Regulating Protein | Caspases | Enzyme Activation
Journal Article
Tumor Biology, ISSN 1010-4283, 11/2016, Volume 37, Issue 11, p. 14789
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s13277-016-5305-6 GW501516... 
Cytochrome c | Bladder cancer | Cancer cells | Apoptosis | Cells | Cell adhesion & migration
Journal Article
Cellular Signalling, ISSN 0898-6568, 02/2014, Volume 26, Issue 2, pp. 433 - 443
Peroxisome Proliferator-Activated Receptor-β (PPARβ) is a ligand-inducible transcription factor activated by both natural (fatty acids and derivatives) and... 
Cervical cancer cells | HuR | PPARβ | p38 MAPK | VEGF | mRNA stability | P38 MAPK | MRNA stability | BLADDER-CANCER | ACTIVATED PROTEIN-KINASE | HPV-16 E6 | FACTOR EXPRESSION | PPAR beta | TUMOR ANGIOGENESIS | RICH ELEMENTS | CELL BIOLOGY | BREAST-CANCER CELLS | GENE-EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | ELAV Proteins - antagonists & inhibitors | Humans | Vascular Endothelial Growth Factor A - metabolism | RNA Stability - drug effects | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Tumor Suppressor Protein p53 - genetics | Vascular Endothelial Growth Factor A - chemistry | Oncogene Proteins, Viral - antagonists & inhibitors | Protein Isoforms - metabolism | PPAR-beta - agonists | RNA Interference | Human papillomavirus 18 - metabolism | Oncogene Proteins, Viral - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | DNA-Binding Proteins | Oncogene Proteins, Viral - metabolism | PPAR-beta - metabolism | Signal Transduction | ELAV Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | PPAR-beta - genetics | Up-Regulation - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | HeLa Cells | Phenoxyacetates - pharmacology | ELAV Proteins - genetics | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Index Medicus | Cellular | Control | Ligands | Activation | Derivatives | Gene expression | Signalling | Cancer
Journal Article
European Urology, ISSN 0302-2838, 2015, Volume 68, Issue 6, pp. 1023 - 1030
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 64, pp. 107744 - 107762
Known activators of the Peroxisome Proliferator-Activated Receptor gamma (PPAR gamma), thiazolidinediones (TZD) induce apoptosis in a variety of cancer cells... 
Cervical cancer cells | TRAIL | PPAR | Thiazolidinedione | Apoptosis | HPV | thiazolidinedione | cervical cancer cells | INDUCED APOPTOSIS | DOWN-REGULATION | apoptosis | TROGLITAZONE | TUMOR-CELLS | INDUCTION | PROLIFERATOR-ACTIVATED-RECEPTOR | CELL BIOLOGY | PPAR-GAMMA-LIGANDS | UP-REGULATION | THIAZOLIDINEDIONES
Journal Article
Journal Article
Oncology Reports, ISSN 1021-335X, 2009, Volume 21, Issue 6, pp. 1495 - 1504
The present investigation was conducted first to determine whether correlation exists between VEGF-A and -B mRNA levels and clinicopathological parameters and... 
Isoform | mRNA | VEGF-A | Bladder cancer | VEGF-B | Prognostic marker | CARCINOMA-CELLS | ANGIOGENESIS | COLON-CANCER | LUNG-CANCER | prognostic marker | LYMPH-NODE METASTASIS | MESSENGER-RNA | ONCOLOGY | bladder cancer | isoform | FACTOR GENE | PROGRESSION | ENDOTHELIAL GROWTH-FACTOR | Carcinoma, Transitional Cell - mortality | Blotting, Southern | Up-Regulation | Alternative Splicing | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Transitional Cell - therapy | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Carcinoma in Situ - genetics | Urinary Bladder Neoplasms - genetics | Time Factors | Urinary Bladder Neoplasms - pathology | Carcinoma, Transitional Cell - genetics | Urinary Bladder Neoplasms - mortality | Carcinoma in Situ - pathology | Neoplasm Invasiveness | Kaplan-Meier Estimate | Treatment Outcome | Carcinoma in Situ - therapy | Reverse Transcriptase Polymerase Chain Reaction | Disease-Free Survival | Blotting, Northern | Carcinoma, Transitional Cell - pathology | Urinary Bladder Neoplasms - therapy | Cell Line, Tumor | Vascular Endothelial Growth Factor B - genetics | Biomarkers, Tumor - genetics | Neoplasm Staging | Carcinoma in Situ - mortality | Index Medicus | Vascular Endothelial Growth Factor A | Vascular Endothelial Growth Factor B | Tumor Markers, Biological | Carcinoma, Transitional Cell | Urinary Bladder Neoplasms | Life Sciences | Carcinoma in Situ | RNA, Messenger | Kaplan-Meiers Estimate | Cancer
Journal Article
Journal Article
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2007, Volume 313, Issue 15, pp. 3239 - 3250
Altered angiogenic response is associated with high-grade cervical dysplasia and with invasive squamous carcinoma of the cervix. Vascular Endothelial Growth... 
Angiogenesis | HPV18 | E6 oncoprotein | VEGF | Cervical cancer | WILD-TYPE P53 | CANCER-CELLS | CERVICAL-CARCINOMA CELLS | angiogenesis | HPV-16 E6 | TUMOR ANGIOGENESIS | cervical cancer | ISOFORM EXPRESSION | CELL BIOLOGY | COLON-CANCER | MESSENGER-RNA | ONCOLOGY | GENE-EXPRESSION | GROWTH-FACTOR EXPRESSION | Oncogene Proteins, Viral - physiology | DNA-Binding Proteins - physiology | Adenocarcinoma - pathology | Alternative Splicing | Humans | Gene Expression Regulation, Neoplastic | Uterine Cervical Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Adenocarcinoma - virology | Tumor Suppressor Protein p53 - physiology | Adenocarcinoma - metabolism | Protein Isoforms - metabolism | Uterine Cervical Neoplasms - metabolism | Human papillomavirus 18 - metabolism | Uterine Cervical Neoplasms - virology | Female | HeLa Cells | Protein Isoforms - genetics | Vascular endothelial growth factor | Papillomavirus infections | Neovascularization | Proteins | Oncology | Human papillomavirus | Biochemistry | Cellular biology | Index Medicus | Adenocarcinoma | Vascular Endothelial Growth Factor A | Uterine Cervical Neoplasms | Oncogene Proteins, Viral | Human papillomavirus 18 | Life Sciences | Tumor Suppressor Protein p53 | Protein Isoforms | RNA, Messenger | Hela Cells | DNA-Binding Proteins | Cancer
Journal Article
Journal Article