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PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, pp. e108164 - e108164
Transient Receptor Potential Ankyrin 1 (TRPA1) channels are localized on sensory nerves and several non-neural cells, but data on their functional significance... 
INFLAMMATORY-BOWEL-DISEASE | GENE-RELATED PEPTIDE | ACID-INDUCED COLITIS | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | INTESTINAL POLYPEPTIDE VIP | VANILLOID CAPSAICIN RECEPTORS | SODIUM-INDUCED COLITIS | SUBSTANCE-P | SENSORY NEURONS | TNBS-INDUCED COLITIS | Up-Regulation | Gene Expression | Nerve Tissue Proteins - physiology | Humans | Neuropeptides - metabolism | DNA Primers | Nerve Tissue Proteins - genetics | Colon - metabolism | Mice, Knockout | Calcium Channels - physiology | Animals | Colitis - physiopathology | Transient Receptor Potential Channels - genetics | Base Sequence | TRPA1 Cation Channel | Colitis - metabolism | Polymerase Chain Reaction | Transient Receptor Potential Channels - physiology | Mice | Calcium Channels - genetics | Immunohistochemistry | Inflammatory bowel diseases | Motility | Peptides | Disease | Epithelial cells | Interleukin | Capsaicin receptors | Neuropeptides | mRNA | Macrophages | Medical schools | Transient receptor potential proteins | Receptors | Neurokinin | Intestine | Vasoactive agents | Rodents | Nerves | Sensory neurons | Ion channels | Colon | Localization | Drug therapy | Cytokines | Internal medicine | Substance P | Inflammation | Pharmacology | Ganglia | Pituitary adenylate cyclase-activating polypeptide | Inflammatory bowel disease | Dextran | Crohn's Disease | Brain research | Lymphocytes B | Neurokinin NK1 receptors | γ-Interferon | Pituitary | Interferon | Ankyrin | Chemokines | Ulcerative colitis | Index Medicus
Journal Article
Pharmacological Research, ISSN 1043-6618, 08/2015, Volume 100, pp. 101 - 116
Abstract Transient Receptor Potential (TRP) cation channels, such as TRP Vanilloid 1 and TRP Ankyrin repeat domain 1 (TRPV1 and TRPA1) are nocisensors playing... 
Transient Receptor Potential channel | Nerve terminal | Sphingomyelin | Sensory neuron | Lipid rafts | Methyl β-cyclodextrin
Journal Article
Peptides, ISSN 0196-9781, 02/2012, Volume 33, Issue 2, pp. 307 - 316
► We examined the PACAP-27/38-LI in the activated trigeminovascular system. ► Blood plasma, nerve tissues and liquor were analyzed in two rat models. ►... 
Electrical and chemical stimulation | Trigeminal ganglion | Trigeminal nucleus caudalis | Mass spectrometry | Nitroglycerol | Blood plasma | GENE-RELATED PEPTIDE | INDUCED NEURONAL ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR MESSENGER-RNA | SPINAL-CORD | ELECTRICAL-STIMULATION | VASOACTIVE INTESTINAL POLYPEPTIDE | CAUDAL TRIGEMINAL NUCLEUS | ENDOCRINOLOGY & METABOLISM | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | URINARY-BLADDER PATHWAYS | CEREBRAL DURA-MATER | Immunohistochemistry | Electric Stimulation | Spinal Cord - drug effects | Spinal Cord - metabolism | Vasodilator Agents - pharmacology | Vasomotor System - metabolism | Protein Isoforms - cerebrospinal fluid | Trigeminal Caudal Nucleus - metabolism | Vasomotor System - drug effects | Rats | Trigeminal Ganglion - metabolism | Trigeminal Caudal Nucleus - drug effects | Trigeminal Ganglion - drug effects | Nitroglycerin - pharmacology | Rats, Sprague-Dawley | Protein Isoforms - blood | Animals | Protein Isoforms - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - blood | Pituitary Adenylate Cyclase-Activating Polypeptide - cerebrospinal fluid | Trigeminal Ganglion - physiology | Peptides | Neurosciences | Universities and colleges | Headache | Alterations | Spinal cord | Elevated | Neurons | Stimulation | Activation | Ganglia | Index Medicus
Journal Article
Journal Article
Endocrinology, ISSN 0013-7227, 10/2017, Volume 158, Issue 10, pp. 3249 - 3258
Sex differences exist in chronic pain pathologies, and gonadal estradiol (E2) alters the pain sensation. The nocisensor transient receptor potential vanilloid... 
FEMALE RATS | TEMPOROMANDIBULAR-JOINT | MECHANICAL HYPERALGESIA | IN-VIVO | ENDOCRINOLOGY & METABOLISM | DORSAL-ROOT GANGLIA | PRIMARY SENSORY NEURONS | SEX-DIFFERENCES | TRPV1 RECEPTORS | OVARIECTOMIZED RATS | ESTROGEN-RECEPTOR | Gene Expression - drug effects | Capsaicin - pharmacology | Mechanoreceptors - physiology | Nociceptors - physiology | Proestrus - physiology | RNA, Messenger - analysis | Male | TRPV Cation Channels - drug effects | Sensory Receptor Cells - physiology | Female | Estradiol - pharmacology | TRPV Cation Channels - physiology | Ovariectomy | Mice, Inbred C57BL | Cells, Cultured | Drug Tolerance | Hot Temperature | Sex Characteristics | TRPV Cation Channels - genetics | Mice, Knockout | Up-Regulation - drug effects | Animals | Mechanoreceptors - drug effects | Pain - physiopathology | Estrus - physiology | Mice | Nociceptors - drug effects | Sensory Receptor Cells - chemistry | Capsaicin | Capsaicin receptors | Estrus cycle | Estradiol | Transient receptor potential proteins | Pain | Rodents | Gender aspects | Sensory neurons | TrkA protein | TrkA receptors | Pretreatment | Pain sensitivity | Pain perception | Estrus | Desensitization | Thresholds | Incubation | Neurons | Tropomyosin | Sex differences | Gene expression | Sex hormones | Ribonucleic acids | In vivo methods and tests | Receptor mechanisms | Index Medicus | Abridged Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 15916 - 8
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e61684 - e61684
Objective: Substance P, encoded by the Tac1 gene, is involved in neurogenic inflammation and hyperalgesia via neurokinin 1 (NK1) receptor activation. Its... 
SENSORY NERVE-TERMINALS | RHEUMATOID-ARTHRITIS | SYNOVIAL-FLUID | NK1 | MULTIDISCIPLINARY SCIENCES | ANIMAL-MODELS | HEMOKININ-1 | DEFICIENT MICE | TARGETED DELETION | EXPRESSION | SUBSTANCE-P-LIKE | Hyperalgesia - chemically induced | Arthritis, Experimental - genetics | Edema - genetics | Receptors, Neurokinin-1 - deficiency | Male | Arthritis, Experimental - metabolism | Hyperalgesia - pathology | Arthritis, Experimental - pathology | Tachykinins - genetics | Arthritis, Experimental - chemically induced | Interleukin-1beta - biosynthesis | Tarsus, Animal - pathology | Joints - metabolism | Protein Precursors - genetics | Hyperalgesia - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Joints - pathology | Inflammation | Tachykinins - deficiency | Freund's Adjuvant | Mice, Knockout | Edema - metabolism | Protein Precursors - deficiency | Animals | Substance P - deficiency | Hyperalgesia - genetics | Substance P - genetics | Mice | Edema - chemically induced | Tarsus, Animal - metabolism | Edema - pathology | Receptors, Neurokinin-1 - genetics | Plethysmography | Neuropeptides | Arthritis | B cells | Genes | Neurophysiology | Tachykinin receptors | Neurosciences | Peptides | Nervous system | Experiments | Cartilage | Receptors | Immunology | Pain | Health care networks | Rodents | Tumor necrosis factor-TNF | Drug therapy | Enzyme-linked immunosorbent assay | Pain perception | Swelling | Substance P | Pharmacology | IL-1β | Gene expression | Joint diseases | Strain | White blood cells | Medicine | Brain research | Neurokinin NK1 receptors | Rheumatoid arthritis | Infiltration | Genetic engineering | Receptor mechanisms | Tachykinin | Index Medicus
Journal Article