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1. Full Text Breast cancer-secreted miR-939 downregulates VE-cadherin and destroys the barrier function of endothelial monolayers
by Di Modica, Martina|Regondi, Viola|Sandri, Marco|Iorio, Marilena V.|Zanetti, Adriana|Tagliabue, Elda|Casalini, Patrizia|Triulzi, Tiziana
Cancer Letters, ISSN 0304-3835, 2016, Volume 384, pp. 94 - 100
Abstract Exosomes-secreted microRNAs play an important role in metastatic spread. During this process breast cancer cells acquire the ability to transmigrate... 
Hematology, Oncology and Palliative Medicine | Breast cancer | Metastasis | miR-939 | Exosomes | Triple-negative breast cancer | CELLS | DIAGNOSIS | ANGIOGENESIS | RISK | MICRORNAS | BIOMARKERS | VESSELS | ONCOLOGY | JUNCTIONS | DISTANT METASTASIS | Exosomes - metabolism | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasm Metastasis | Transfection | Time Factors | Transendothelial and Transepithelial Migration | Triple Negative Breast Neoplasms - pathology | Female | Cadherins - genetics | Paracrine Communication | Signal Transduction | Down-Regulation | Proportional Hazards Models | Permeability | Disease-Free Survival | Triple Negative Breast Neoplasms - genetics | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Human Umbilical Vein Endothelial Cells - pathology | MicroRNAs - genetics | Analysis | Molecular biology | Monomolecular films | Endothelium | Proteins | Cloning | Medical prognosis | Mortality | Conflicts of interest | Binding sites | Methods | Tumors | Index Medicus
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2. Full Text FOXP3 Expression and Overall Survival in Breast Cancer
by Andrea Merlo and Patrizia Casalini and Maria Luisa Carcangiu and Chiara Malventano and Tiziana Triulzi and Sylvie Mènard and Elda Tagliabue and Andrea Balsari
Journal of Clinical Oncology, ISSN 0732-183X, 04/2009, Volume 27, Issue 11, pp. 1746 - 1752
Purpose The transcription factor forkhead box P3 (FOXP3) up-or downregulates a large number of genes and has been recently reported to be expressed in tumor... 
RELAPSE | METASTASIS | ONCOLOGY | LYMPH-NODES | REGULATORY T-CELLS | PANCREAS | PERIPHERAL-BLOOD | RADIOTHERAPY | PREVALENCE | SUPPRESSION | TUMOR MICROENVIRONMENT | Breast Neoplasms - surgery | Forkhead Transcription Factors - biosynthesis | Prognosis | Breast Neoplasms - pathology | Humans | Middle Aged | Survival Analysis | Breast Neoplasms - mortality | Female | Breast Neoplasms - metabolism | Index Medicus
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3. Full Text Tumor-Initiating Cells of HER2-Positive Carcinoma Cell Lines Express the Highest Oncoprotein Levels and Are Sensitive to Trastuzumab
by Alessandra Magnifico and Luisa Albano and Stefano Campaner and Domenico Delia and Fabio Castiglioni and Patrizia Gasparini and Gabriella Sozzi and Enrico Fontanella and Sylvie Menard and Elda Tagliabue
Clinical Cancer Research, ISSN 1078-0432, 03/2009, Volume 15, Issue 6, pp. 2010 - 2021
Purpose: The existence of tumor-initiating cells in breast cancer has profound implications for cancer therapy. In this study, we investigated the sensitivity... 
HER2-overexpressing carcinoma cells | Notch pathway | tumor initiating cells | Trastuzumab | BREAST-CANCER CELLS | STEM-CELLS | EPITHELIAL-CELLS | ONCOLOGY | GROWTH | NOTCH | MAMMARY STEM/PROGENITOR CELLS | SELF-RENEWAL | GENE AMPLIFICATION | HER2 STATUS | TUMORIGENICITY | Receptor, ErbB-2 - genetics | Antibodies, Monoclonal - pharmacology | Humans | RNA, Messenger - analysis | Xenograft Model Antitumor Assays | Antibodies, Monoclonal, Humanized | Animals | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Mice | Receptor, ErbB-2 - antagonists & inhibitors | Receptor, ErbB-2 - analysis | Quinazolines - pharmacology | Receptor, Notch1 - physiology | Index Medicus
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4. Full Text “Omics” and Immunologic Approaches to Optimizing Cure Rates in HER2-Positive Breast Carcinomas
by Tagliabue, Elda and Campiglio, Manuela
Frontiers in Oncology, ISSN 2234-943X, 11/2014, Volume 4
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5. Full Text Biologic and therapeutic role of HER2 in cancer
by Ménard, Sylvie and Pupa, Serenella Marja and Campiglio, Manuela and Tagliabue, Elda
Oncogene, ISSN 0950-9232, 10/2003, Volume 22, Issue 43, pp. 6570 - 6578
Prognostic value | Therapy | Biological role | Predictive value | HER2
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6. Full Text "Omics" and immunologic approaches to optimizing cure rates in HER2-positive breast carcinomas
by Tagliabue, Elda and Campiglio, Manuela
Frontiers in Oncology, ISSN 2234-943X, 2014, Volume 4, pp. 334 - 334
Trastuzumab resistance | Breast cancer | HER2 | Targeted therapy | Omics
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7. Full Text Stromal responses among carcinomas-letter
by Triulzi, Tiziana and Orlandi, Rosaria and Tagliabu, Elda
Clinical Cancer Research, ISSN 1078-0432, 2014, Volume 20, Issue 5, pp. 1396 - 1396
BREAST-CANCER | ONCOLOGY | CHEMOTHERAPY | Prostatic Neoplasms - pathology | Breast Neoplasms - pathology | Stromal Cells - pathology | Colonic Neoplasms - pathology | Humans | Ovarian Neoplasms - pathology | Female | Male | Gene Expression Profiling | Fibroblasts - pathology | Index Medicus
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8. Full Text Pilot Study of the Mechanism of Action of Preoperative Trastuzumab in Patients with Primary Operable Breast Tumors Overexpressing HER2
by Roberto Gennari and Sylvie Menard and Francesco Fagnoni and Luisa Ponchio and Mario Scelsi and Elda Tagliabue and Fabio Castiglioni and Laura Villani and Cesare Magalotti and Nadia Gibelli and Barbara Oliviero and Bettina Ballardini and Gianantonio Da Prada and Alberto Zambelli and Alberto Costa
Clinical Cancer Research, ISSN 1078-0432, 09/2004, Volume 10, Issue 17, pp. 5650 - 5655
Purpose: To elucidate the mechanism by which trastuzumab, a humanized monoclonal antibody against HER2 with proven survival benefit in women with HER2-positive... 
ANTIBODY | ONCOLOGY | CANCER | TARGET-CELLS | CHEMOTHERAPY | Lymphocytes - metabolism | Breast Neoplasms - surgery | Antibody-Dependent Cell Cytotoxicity | Humans | Receptor, ErbB-2 - metabolism | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Remission Induction | Preoperative Care | Breast Neoplasms - therapy | Antibodies, Monoclonal, Humanized | Lymphocytes - pathology | Pilot Projects | Lymphocytes - immunology | Breast Neoplasms - pathology | Female | Neoadjuvant Therapy | Cell Proliferation - drug effects | Trastuzumab | Index Medicus
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9. Full Text CIAP1 regulates the EGFR/Snai2 axis in triple-negative breast cancer cells
by Majorini, Maria Teresa and Manenti, Giacomo and Mano, Miguel and De Cecco, Loris and Conti, Annalisa and Pinciroli, Patrizia and Fontanella, Enrico and Tagliabue, Elda and Chiodoni, Claudia and Colombo, Mario Paolo and Delia, Domenico and Lecis, Daniele
Cell Death and Differentiation, ISSN 1350-9047, 04/2018, Volume 25, Issue 12, pp. 2147 - 2164
Inhibitor of apoptosis (IAP) proteins constitute a family of conserved molecules that regulate both apoptosis and receptor signaling. They are often... 
TO-MESENCHYMAL TRANSITION | STEM-CELLS | ACTIVATION | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LRIG1 | SENSITIVITY | APOPTOSIS PROTEINS | NF-KAPPA-B | SLUG | CARCINOMA | CELL BIOLOGY | IAP protein | Epidermal growth factor receptors | Mesenchyme | MAP kinase | Breast cancer | Tumor cell lines | Gene expression | Kinases | Nodules | Metastases | Proteins | Gene silencing | DIABLO protein | Epidermal growth factor | Protein kinase | Cell lines | Snail protein | Apoptosis | Tumors
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10. Full Text Extracellular matrix proteins as diagnostic markers of breast carcinoma
by Giussani, Marta and Landoni, Elena and Merlino, Giuseppe and Turdo, Federica and Veneroni, Silvia and Paolini, Biagio and Cappelletti, Vera and Miceli, Rosalba and Orlandi, Rosaria and Triulzi, Tiziana and Tagliabue, Elda
Journal of Cellular Physiology, ISSN 0021-9541, 08/2018, Volume 233, Issue 8, pp. 6280 - 6290
We tested the hypothesis that abnormal production of extracellular matrix (ECM) components leads to blood‐released ECM molecules representing tumor circulating... 
breast cancer | diagnosis | biomarker | tumor‐microenvironment | extracellular matrix | tumor-microenvironment | PHYSIOLOGY | COLLAGEN | PROTEOME | BONE-MARROW | SERUM | CELL BIOLOGY | STROMAL CELLS | CANCER PROGRESSION | EXPRESSION | TUMOR MICROENVIRONMENT | Carcinoma | Analysis | Collagen | Genes | Breast cancer | Diagnosis | Gene expression | Cancer | Collagens | Blood plasma | Proteins | Lysates | Breast carcinoma | Fibroblasts | Extracellular matrix | Bioindicators | Conditioning | Matrix protein | Enzyme-linked immunosorbent assay | Benign | Stroma | Patients | Nodules | Blood circulation | Cell lines | Breast | Biomarkers | Skin | Diagnostic systems | Tumors
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11. Full Text Early immune modulation by single-agent trastuzumab as a marker of trastuzumab benefit
by Triulzi, Tiziana and Regondi, Viola and De Cecco, Loris and Cappelletti, Maria Rosa and Di Modica, Martina and Paolini, Biagio and Lollini, Pier Luigi and Di Cosimo, Serena and Sfondrini, Lucia and Generali, Daniele and Tagliabue, Elda
British Journal of Cancer, ISSN 0007-0920, 12/2018, Volume 119, Issue 12, pp. 1487 - 1494
BACKGROUND: Optimising the selection of HER2-targeted regimens by identifying subsets of HER2-positive breast cancer ( BC) patients who need more or less... 
PREOPERATIVE TRASTUZUMAB | THERAPY | EFFICACY | ONCOLOGY | PATHOLOGICAL RESPONSE | RESISTANCE | SUBSETS | INNATE | CELL | HER2-POSITIVE BREAST-CANCER | Animal models | Immunomodulation | Breast cancer | Gene expression | Macrophages | Patients | ErbB-2 protein | Chemotherapy | Major histocompatibility complex | Pathways | Mice | Trastuzumab | Tumors | Cancer
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12. Full Text WNT signaling modulates PD-L1 expression in the stem cell compartment of triple-negative breast cancer
by Castagnoli, Lorenzo and Cancila, Valeria and Cordoba-Romero, Sandra L and Faraci, Simona and Talarico, Giovanna and Belmonte, Beatrice and Iorio, Marilena V and Milani, Matteo and Volpari, Tatiana and Chiodoni, Claudia and Hidalgo-Miranda, Alfredo and Tagliabue, Elda and Tripodo, Claudio and Sangaletti, Sabina and Di Nicola, Massimo and Pupa, Serenella M
Oncogene, ISSN 0950-9232, 2019, Volume 38, Issue 21, pp. 4047 - 4060
Triple-negative breast cancers (TNBCs) are characterized by a poor prognosis and lack of targeted treatments, and thus, new therapeutic strategies are urgently... 
TUMOR-INFILTRATING LYMPHOCYTES | ONCOLOGY | PATHWAY | STABILIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | ONTOLOGY | PROGNOSTIC VALUE | CELL BIOLOGY | Care and treatment | Prognosis | Analysis | Ligands (Biochemistry) | Stem cells | Genetic aspects | Breast cancer | Research | Gene expression | Phenotypes | Wnt protein | PD-1 protein | Tumor cells | Lymphocytes T | CD3 antigen | Aldehyde dehydrogenase | Ataxin | CD44 antigen | PD-L1 protein | Cell lines | Apoptosis | Tumors
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13. Full Text Intratumor lactate levels reflect HER2 addiction status in HER2‐positive breast cancer
by Castagnoli, Lorenzo and Iorio, Egidio and Dugo, Matteo and Koschorke, Ada and Faraci, Simona and Canese, Rossella and Casalini, Patrizia and Nanni, Patrizia and Vernieri, Claudio and Di Nicola, Massimo and Morelli, Daniele and Tagliabue, Elda and Pupa, Serenella M
Journal of Cellular Physiology, ISSN 0021-9541, 02/2019, Volume 234, Issue 2, pp. 1768 - 1779
Despite different molecular tumor profiles indicate that human epidermal growth factor receptor 2 (HER2) messenger RNA (mRNA) levels mirror HER2 addiction and... 
oncogene addiction | breast cancer | HER2 | lactate | glycolysis | ADJUVANT CHEMOTHERAPY | MULTICENTER | PHYSIOLOGY | TRASTUZUMAB | EFFICACY | OPEN-LABEL | HYPOXIA | CELL BIOLOGY | GROWTH | TREATMENT RESPONSE | EXPRESSION | PLUS | Lactates | Breast cancer | DNA microarrays | Analysis | Enrichment | Biotechnology | Nuclear magnetic resonance--NMR | Transcription | Transgenic | Magnetic resonance spectroscopy | Epidermal growth factor | Pathways | Bioindicators | Growth factors | Metabolism | Gene expression | Addictions | Ribonucleic acid--RNA | ErbB-2 protein | Addiction | Magnetic permeability | Cell lines | Glycolysis | Biomarkers | Lactic acid | Trastuzumab | Cancer | Tumors | Original | Original s
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14. Full Text Association of adiposity, dysmetabolisms, and inflammation with aggressive breast cancer subtypes: a cross-sectional study
by Agresti, Roberto and Meneghini, Elisabetta and Baili, Paolo and Minicozzi, Pamela and Turco, Alberto and Cavallo, Ilaria and Funaro, Francesco and Amash, Hade and Berrino, Franco and Tagliabue, Elda and Sant, Milena
Breast Cancer Research and Treatment, ISSN 0167-6806, 5/2016, Volume 157, Issue 1, pp. 179 - 189
Obesity and metabolic syndrome are risk and prognostic factors for breast cancer (BC) and are associated with chronic inflammation. We investigated the... 
Breast cancer subtype | Medicine & Public Health | Adiposity | Oncology | Insulin resistance | Inflammation | Metabolic syndrome | C-REACTIVE PROTEIN | METAANALYSIS | RECEPTOR STATUS | RISK | THERAPY | ONCOLOGY | INSULIN-RESISTANCE | ESTROGEN | WEIGHT | AGE | Cross-Sectional Studies | Obesity - complications | Humans | Middle Aged | Insulin Resistance | Metabolic Syndrome - complications | Breast Neoplasms - metabolism | Waist Circumference | C-Reactive Protein - metabolism | Breast Neoplasms - pathology | Biomarkers, Tumor - metabolism | Adult | Female | Aged | Odds Ratio | Index Medicus
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15. Full Text The PDGFRβ/ERK1/2 pathway regulates CDCP1 expression in triple-negative breast cancer
by Forte, Luca and Turdo, Federica and Ghirelli, Cristina and Aiello, Piera and Casalini, Patrizia and Iorio, Marilena Valeria and D'Ippolito, Elvira and Gasparini, Patrizia and Agresti, Roberto and Belmonte, Beatrice and Sozzi, Gabriella and Sfondrini, Lucia and Tagliabue, Elda and Campiglio, Manuela and Bianchi, Francesca
BMC Cancer, ISSN 1471-2407, 05/2018, Volume 18, Issue 1, pp. 586 - 586
CDCP1, a transmembrane protein with tumor pro-metastatic activity, was recently identified as a prognostic marker in TNBC, the most aggressive breast cancer... 
PDGFRβ | ERK1/2 | CDCP1 | IHC | FISH | TNBC | PDGF-BB | Becaplermin - pharmacology | Up-Regulation | Humans | Middle Aged | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Neoplasm Proteins - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Cell Adhesion Molecules - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | MAP Kinase Signaling System - drug effects | MAP Kinase Signaling System - genetics | Triple Negative Breast Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Triple Negative Breast Neoplasms - pathology | Cell Line, Tumor | Female | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Index Medicus
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16. Full Text Antibody-mediated blockade of JMJD6 interaction with collagen I exerts antifibrotic and antimetastatic activities
by Miotti, Silvia and Gulino, Alessandro and Ferri, Renata and Parenza, Mariella and Chronowska, Agnieszka and Lecis, Daniele and Sangaletti, Sabina and Tagliabue, Elda and Tripodo, Claudio and Colombo, Mario P
FASEB Journal, ISSN 0892-6638, 2017, Volume 31, Issue 12, pp. 5356 - 5370
JMJD6 is known to localize in the nucleus, exerting histone arginine demethylase and lysyl hydroxylase activities. A novel localization of JMJD6 in the... 
Extracellular matrix | In vivo treatment | Peptide library | Monoclonal antibody | JmjC family | FIBROSIS | LOCALIZATION | PHOSPHATIDYLSERINE RECEPTOR | METASTASIS | SPARC | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYDROXYLATION | in vivo treatment | CELL BIOLOGY | peptide library | APOPTOTIC CELLS | BIOLOGY | FIBRONECTIN | BINDING-SITE | monoclonal antibody | CONTAINING PROTEIN 6 | extracellular matrix | Immunohistochemistry | Collagen Type I - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Extracellular Matrix - metabolism | Osteonectin - metabolism | Receptors, Cell Surface - metabolism | Signal Transduction - genetics | Peptide Library | Mice, Knockout | Xenograft Model Antitumor Assays | Extracellular Matrix - genetics | Animals | Cell Nucleus - metabolism | Collagen Type I - genetics | Osteonectin - genetics | Cell Line, Tumor | Protein Binding | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Antibodies, Monoclonal - metabolism | Receptors, Cell Surface - genetics | Collagen (type I) | Target recognition | Peptides | Lung | Procollagen-lysine 5-dioxygenase | Nuclei | Fibronectin | Metastases | Breast carcinoma | Fibrillogenesis | Arginine | Enzymatic activity | Localization | Recombinant | Secretion | Tumor cells | Hydroxylase | Immunodeficiency | Stroma | Substrates | Collagen | Fibrosis | Monoclonal antibodies | Position (location) | Tumors | Index Medicus
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17. Full Text High efficacy of CpG-ODN, Cetuximab and Cisplatin combination for very advanced ovarian xenograft tumors
by Sommariva, Michele and de Cesare, Michelandrea and Meini, Alessandra and Cataldo, Alessandra and Zaffaroni, Nadia and Tagliabue, Elda and Balsari, Andrea
Journal of Translational Medicine, ISSN 1479-5876, 01/2013, Volume 11, Issue 1, pp. 25 - 25
Background: To mimic clinical treatment situations in advanced human ovarian disease, we tested the efficacy of CpG-oligodeoxynucleotides (CpG-ODN), synthetic... 
CpG-ODN | TLR9 | Ascites | Cisplatin | Monoclonal Antibody | Ovarian cancer | MEDICINE, RESEARCH & EXPERIMENTAL | NUDE-MICE | MEDIATED CYTOTOXICITY | TLR9 AGONISTS | CANCER-CELL-LINES | TOLL-LIKE RECEPTORS | LYMPHOMA | EXPRESSION | CARCINOMA | ENDOTHELIAL GROWTH-FACTOR | INNATE IMMUNITY | Antibody-Dependent Cell Cytotoxicity | Humans | Ovarian Neoplasms - pathology | Cisplatin - pharmacology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Cisplatin - administration & dosage | Xenograft Model Antitumor Assays | Animals | Antibodies, Monoclonal, Humanized - administration & dosage | Flow Cytometry | Oligonucleotides - pharmacology | Mice, Nude | Antibodies, Monoclonal, Humanized - pharmacology | CpG Islands | Female | Mice | Oligonucleotides - administration & dosage | Cetuximab | Phagocytosis | Drug Screening Assays, Antitumor | Ovarian Neoplasms - immunology | Proteins | Cancer therapies | Rodents | Tumors | Index Medicus
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18. Full Text Abstract 3699: Activation of NK cells cytotoxicity mediated by alveolar macrophages in the lung against murine melanoma metastases by combined aerosol immunotherapy
by Sommariva, Michele and Noci, Valentino Le and Tagliabue, Elda and Balsari, Andrea and Sfondrini, Lucia
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 3699 - 3699
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19. Full Text The landscape of d16HER2 splice variant expression across HER2-positive cancers
by Volpi, Chiara Costanza and Pietrantonio, Filippo and Gloghini, Annunziata and Fucà, Giovanni and Giordano, Silvia and Corso, Simona and Pruneri, Giancarlo and Antista, Maria and Cremolini, Chiara and Fasano, Elena and Saggio, Serena and Faraci, Simona and Di Bartolomeo, Maria and de Braud, Filippo and Di Nicola, Massimo and Tagliabue, Elda and Pupa, Serenella Maria and Castagnoli, Lorenzo
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 3545 - 12
The HER2 splice variant characterized by the deletion of exon 16 and denominated as d16HER2, is associated with HER2-positive breast cancer (BC)... 
BREAST-CANCER | THERAPY | TRASTUZUMAB | EFFICACY | GASTRIC-CANCER | MULTIDISCIPLINARY SCIENCES | AMERICAN-PATHOLOGISTS | OPEN-LABEL | HER2 | PERTUZUMAB | GUIDELINE | Alternative splicing | Clonal deletion | Colorectal carcinoma | Xenografts | Breast cancer | mRNA | Hybridization | Exosomes | Gastric cancer | ErbB-2 protein | Trastuzumab | Cancer
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