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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2015, Volume 112, Issue 20, pp. E2677 - E2686
Journal Article
Internal Medicine, ISSN 0918-2918, 2019, Volume 58, Issue 13, pp. 1845 - 1849
Objective Oral anticoagulants (OACs), which include direct oral anticoagulants (DOACs) and warfarin, are widely used for the prevention of thromboembolic... 
anticoagulants | atrial fibrillation | thrombosis | neoplasms | stroke | hemorrhage | MEDICINE, GENERAL & INTERNAL | WARFARIN | RIVAROXABAN | EDOXABAN | EKG | Anticoagulants | Cardiac arrhythmia | Stroke | Warfarin | Embolisms | Bleeding | Embolism | Fibrillation | Electrocardiography | Safety | Thromboembolism | Cancer | Original
Journal Article
International Heart Journal, ISSN 1349-2365, 2017, Volume 58, Issue 1, pp. 107 - 114
Cardiac fibrosis is a pathological feature of myocardium of failing heart and plays causative roles in arrhythmia and cardiac dysfunction, but its regulatory... 
Cardiac fibroblasts | EP4 agonist | SUBTYPE | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCYTES | PROTEIN-KINASE | ANGIOTENSIN-II | FAILURE | DIASTOLIC DYSFUNCTION | P42/44 MAPK | MYOCARDIAL FIBROSIS | EXPRESSION | PROSTAGLANDIN E-2 | Protein kinase A | Neonates | Collagen (type I) | Arrhythmia | Transforming growth factor | Kinases | Collagen | Fibrosis | Fibroblasts | Myocardium | Aorta | Agonists | Hypertrophy
Journal Article
Circulation, ISSN 0009-7322, 11/2018, Volume 138, Issue Suppl_1 Suppl 1, pp. A14462 - A14462
IntroductionThe direct oral anticoagulants (DOACs) are widely used for prevention of thromboembolic events in atrial fibrillation (AF) patients. Cancer is... 
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166710
Grb2-associated binder (Gab) docking proteins regulate signals downstream of a variety of growth factors and receptor tyrosine kinases. Neuregulin-1 (NRG-1), a... 
HEART | MULTIDISCIPLINARY SCIENCES | METASTATIC BREAST-CANCER | ERBB2 | GROWTH-FACTOR RECEPTORS | MICE | DILATED CARDIOMYOPATHY | VENTRICULAR MYOCYTES | ADAPTER PROTEIN | TYROSINE PHOSPHATASE SHP2 | CARDIAC DEVELOPMENT | Gene Expression | Phosphorylation | ErbB Receptors - metabolism | Molecular Weight | Alternative Splicing | Signal Transduction | RNA, Messenger - genetics | Phosphoproteins - genetics | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Phosphoproteins - chemistry | Animals | Neuregulin-1 - metabolism | Myocytes, Cardiac - metabolism | Mice | Proto-Oncogene Proteins c-akt - metabolism | Heart failure | Circuit components | Viral antibodies | Tyrosine | Epidermal growth factor | Developmental biology | Analysis | Adenoviruses | Heart cells | Muscles | Antibodies | Heart | Intravenous administration | Neuregulin 1 | Cardiomyopathy | Exons | Galactosidase | Homeostasis | AKT protein | Stimulation | Activation | Kinases | Phosphatase | Experiments | Molecular weight | Proteins | Signal transduction | Receptors | Rodents | Neuregulin | Docking | Physiology | Cardiology | Growth factors | Heart diseases | University graduates | Antigens | Immunoglobulins | Cytokines | Cardiac muscle | ErbB protein | Cardiomyocytes | siRNA | Skeletal muscle | Grb2 protein | Medicine | Polymerase chain reaction | Signaling | Myocardium | Adapter proteins | Cancer | Apoptosis
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2015, Volume 30, Issue 5, pp. 869 - 877
Journal Article
Journal Article