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Clinical calcium, ISSN 0917-5857, 2018, Volume 28, Issue 4, p. 467
An assessment of the association between the intake of milk or dairy products and bone density or the risk of fractures on the basis of epidemiological studies... 
Bone and Bones | Animals | Bone Density | Calcium, Dietary | Humans | Japan | Dairy Products | Milk
Journal Article
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2016, Volume 31, Issue 5, pp. 940 - 948
Journal Article
Cancer Science, ISSN 1347-9032, 10/2017, Volume 108, Issue 10, pp. 2039 - 2044
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 11/2012, Volume 9, Issue 1, pp. 256 - 256
Introduction: The neuropeptide pituitary adenylate cyclase-activating polypeptide (PACAP) is considered to be a potential therapeutic agent for prevention of... 
DNA microarray | Neuroprotection | Gabra6 | Il6 | Crtam | Ischemia | PACAP | CRMP2 | TARGET | CYCLASE-ACTIVATING POLYPEPTIDE | NEUROENDOCRINE | IMMUNOLOGY | IDENTIFICATION | NEUROSCIENCES | PEPTIDE | DAMAGE | STIMULATES ADENYLATE-CYCLASE | CELL-ASSOCIATED MOLECULE | PROTEINS | EXPRESSION | Oligonucleotide Array Sequence Analysis | Electrophoresis, Gel, Two-Dimensional | Male | Gene Expression Profiling | Interleukins - metabolism | Brain - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Interleukins - genetics | Time Factors | Pituitary Adenylate Cyclase-Activating Polypeptide - pharmacology | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Proteomics - methods | Transcriptome - physiology | Disease Models, Animal | Pituitary Adenylate Cyclase-Activating Polypeptide - genetics | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Gene Expression Regulation - physiology | Functional Laterality | Infarction, Middle Cerebral Artery - pathology | Nerve Tissue Proteins - genetics | Brain - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Proteins - genetics | Animals | Proteins - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Mice | Proteins | Studies | Pituitary gland | Rodents | Proteomics | Economics | Myocardial infarction | Brain | Animal models | Inventories | proteomics | Axonogenesis | Gel electrophoresis | Neuropeptides | Defense mechanisms | Desorption | Inflammation | Gene expression | Western blotting | Pituitary adenylate cyclase-activating polypeptide | Spectrometry | DNA microarrays | Interleukins | Cerebral blood flow | Lasers | Nerves | Cancer
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 04/2011, Volume 193, Issue 2, pp. 409 - 424
Cellular senescence acts as a barrier to cancer progression, and microRNAs (miRNAs) are thought to be potential senescence regulators. However, whether... 
BREAST-CANCER | LUNG-CANCER | TARGET GENES | TUMOR SUPPRESSION | GROWTH ARREST | LET-7 MICRORNA | THYROID-CANCER | IN-VIVO | DOWN-REGULATION | CYCLE ARREST | CELL BIOLOGY | Physiological aspects | Development and progression | Genetic aspects | MicroRNA | Research | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63606
Hemorrhagic shock and resuscitation induces pulmonary inflammation that leads to acute lung injury. Biliverdin, a metabolite of heme catabolism, has been shown... 
OXIDATIVE DAMAGE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DECREASED PROGRESSION | REPERFUSION INJURY | MULTIPLE ORGAN FAILURE | HEME OXYGENASE-1 | MODEL | DYSFUNCTION | RESPIRATORY-DISTRESS-SYNDROME | PROTECTS | Tumor Necrosis Factor-alpha - metabolism | Shock, Hemorrhagic - complications | Tumor Necrosis Factor-alpha - genetics | Biliverdine - administration & dosage | Male | Pulmonary Edema - etiology | Acute Lung Injury - blood | Biliverdine - pharmacology | Inflammation Mediators - metabolism | Pulmonary Edema - pathology | Acute Lung Injury - drug therapy | Lung - metabolism | Neutrophils - metabolism | Deoxyguanosine - analogs & derivatives | Resuscitation | Lung - pathology | Biliverdine - therapeutic use | Neutrophils - drug effects | Rats | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | Shock, Hemorrhagic - blood | Gene Expression Regulation - drug effects | Bilirubin - blood | Animals | Aquaporin 5 - metabolism | Nitric Oxide Synthase Type II - genetics | Lung - drug effects | Deoxyguanosine - metabolism | Acute Lung Injury - etiology | Nitric Oxide Synthase Type II - metabolism | Acute respiratory distress syndrome | Hemorrhagic shock | Nitric oxide | Heme | Mitochondrial DNA | Inflammation | Gene expression | Shock | Intravenous administration | DNA damage | Hemorrhage | Bleeding | Metastases | Prevention | Rodents | Surgery | Blood pressure | Drug dosages | Injuries | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | University graduates | Edema | Medical research | Damage assessment | Biliverdin | Dentistry | Tumor necrosis factor-α | Trauma | Catheters | Nitric-oxide synthase | Medicine | Deoxyguanosine | Lungs | Catabolism | Infiltration | Anesthesiology | Laboratory animals | Veins & arteries | Deoxyribonucleic acid | DNA
Journal Article
Hypertension Research, ISSN 0916-9636, 03/2019, Volume 42, Issue 3, pp. 411 - 418
While the association between 24-h urinary sodium and potassium excretion with blood pressure is well established, the relationships of these ions to spot... 
Association study | Blood pressure | Spot urine | Sodium | Potassium | INTERSALT | CASUAL URINE | PREVENTION | SODIUM/POTASSIUM RATIO | SUFFICIENT | REDUCTION | SALT SENSITIVITY | BODY-MASS INDEX | JAPANESE | PERIPHERAL VASCULAR DISEASE | CARDIOVASCULAR RISK-FACTORS | Urine | Standard deviation
Journal Article