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Journal Article
Journal Article
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, ISSN 0091-6749, 01/2017, Volume 139, Issue 1, pp. 352 - 352
To the Editor: The identification of polymorphonuclear neutrophils as cellular source of cytokines prompts researchers in the field to investigate whether the... 
CELLS | 6-SULFO LACNAC | ACTIVATION | ALLERGY | TOLL-LIKE RECEPTORS | SUBSETS | ALPHA | IMMUNOLOGY | EXPRESSION | Medical research | Ligands | Cytokines | Neutrophils
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 45, pp. 18257 - 18258
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 08/2017, Volume 8
Interferon lambdas (IFNλs) are recently discovered cytokines acting not only at the first line of defense against viral infections but also at the mucosal... 
CXCL10 | plasmacytoid dendritic cells | IFNα | IL-3 | innate immunity | interferon lambda
Journal Article
The Journal of allergy and clinical immunology, ISSN 0091-6749, 06/2018, Volume 141, Issue 6, pp. 2276 - 2279.e3
[...]human PBMCs were stained with a panel of fluorochrome-conjugated antibodies (listed in the Methods section in this article's Online Repository at... 
Flow cytometry | Antigens | Monocytes | CD16 antigen | Dendritic cells | Cytokines | Software | CD14 antigen | Experiments
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 138, Issue 1, pp. 229 - 240.e3
Background Partial DiGeorge syndrome (pDGS) is caused by deletion of the 22q11.2 region. Within this region lies CrK-like (CRKL) , a gene encoding an adapter... 
Allergy and Immunology | DiGeorge syndrome | proliferation | c-Fos | signal transducer and activator of transcription 5 | IL-2 | CRKL | T-cell receptor activation | CYCLIN D1 EXPRESSION | 22Q11 DELETION SYNDROMES | IMMUNOLOGY | SIGNALING COMPLEX | HEMATOPOIETIC-CELLS | ALLERGY | NONOBESE DIABETIC MICE | GENE-EXPRESSION | VELOCARDIOFACIAL SYNDROME | IL-2 RECEPTOR | NEURAL CREST | ADAPTER PROTEIN | Phosphorylation | Humans | Middle Aged | Child, Preschool | Infant | DiGeorge Syndrome - etiology | Case-Control Studies | Lymphocyte Subsets - immunology | Young Adult | Lymphocyte Subsets - metabolism | STAT5 Transcription Factor - metabolism | RNA Interference | T-Lymphocytes - metabolism | Adult | Nuclear Proteins - genetics | Child | Cell Line | Lymphocyte Activation | Gene Expression Regulation | Gene Silencing | Nuclear Proteins - metabolism | Immunoglobulin Isotypes - blood | Phenotype | DiGeorge Syndrome - diagnosis | DiGeorge Syndrome - metabolism | Adaptor Proteins, Signal Transducing - genetics | Adolescent | Biomarkers | Immunoglobulin Isotypes - immunology | T-Lymphocytes - immunology | Adaptor Proteins, Signal Transducing - metabolism | Cytokines - biosynthesis | Immunological deficiency syndromes | Care and treatment | Interferon | Biological response modifiers | T cells | Fluorescein | Genetic research | Genetic transcription | Heart | Immunoglobulins | Transcription factors | Lymphocytes | Genes | Cell cycle | Infections | Mutation | Adapter proteins | Tumors | Defects
Journal Article
Cytokine, ISSN 1043-4666, 10/2016, Volume 86, p. 124
* IFN[lambda]3 and IL-3 upregulate IL-3R[alpha] and IFN[lambda]R1 expression in human pDCs. * IFN[lambda]3 plus IL-3 synergistically induce ISG mRNA... 
Tumor necrosis factor | RNA | Dendritic cells | Cytokines | Biological response modifiers
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 1, pp. 352 - 355.e6
Journal Article
Frontiers in immunology, ISSN 1664-3224, 2017, Volume 8, p. 1015
Interferon lambdas (IFNλs) are recently discovered cytokines acting not only at the first line of defense against viral infections but also at the mucosal... 
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4107 - 4121
Granulomatosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granulomatous inflammation and the presence of... 
PLASMACYTOID DENDRITIC CELLS | WEGENERS-GRANULOMATOSIS | MEDICINE, RESEARCH & EXPERIMENTAL | ANTINEUTROPHIL CYTOPLASMIC AUTOANTIBODY | HUMAN NEUTROPHILS | TGF-BETA | MACROPHAGE PHAGOCYTOSIS | ACTIVATED NEUTROPHILS | ANCA-ASSOCIATED VASCULITIS | NECROSIS-FACTOR-ALPHA | MEMORY T-CELLS | Granulocyte Colony-Stimulating Factor - physiology | T-Lymphocyte Subsets - immunology | Receptors, Interleukin-1 Type I - physiology | Dendritic Cells - immunology | Humans | Antibodies, Antineutrophil Cytoplasmic - immunology | Peritonitis - pathology | Granulomatosis with Polyangiitis - immunology | Granulomatosis with Polyangiitis - pathology | Myeloblastin - biosynthesis | Neutrophils - pathology | Granulomatosis with Polyangiitis - enzymology | Macrophages - physiology | Lung - pathology | Neutrophils - enzymology | Signal Transduction | Myeloblastin - immunology | Mice, Inbred C57BL | Cytokines - secretion | Neutrophils - immunology | Cellular Microenvironment | Myeloblastin - physiology | Nitric Oxide - physiology | Peritonitis - immunology | Autoantigens - physiology | Cell Membrane - enzymology | Animals | Myeloid Differentiation Factor 88 - deficiency | Autoantigens - immunology | Myeloid Differentiation Factor 88 - physiology | Mice | Apoptosis - physiology | Phagocytosis | Proteins | Ethics | Hypotheses | Consent | Rodents | Cloning | Inflammation | Kinases | Experiments | Apoptosis
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 08/2017, Volume 140, Issue 2, pp. 553 - 564.e4
Gain-of-function (GOF) mutations affecting the coiled-coil domain or the DNA-binding domain of signal transducer and activator of transcription 1 cause chronic... 
signal transducer and activator of transcription 5 | IL-15 | Natural killer cells | cytotoxic activity | signal transducer and activator of transcription 1 | candidiasis | PROLIFERATION | IMMUNOLOGY | CHRONIC MUCOCUTANEOUS CANDIDIASIS | DEFICIENCY | FUNGAL-INFECTIONS | HYPER-IGE SYNDROME | GENE | ALLERGY | UNDERLIE | IL-17 IMMUNITY | CYTOTOXICITY | PRIMARY IMMUNODEFICIENCY | Gene Expression | Phosphorylation | Candidiasis, Chronic Mucocutaneous - genetics | Humans | Middle Aged | Candidiasis, Chronic Mucocutaneous - immunology | Male | STAT1 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | STAT1 Transcription Factor - metabolism | Adolescent | Killer Cells, Natural - immunology | Adult | Female | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | Mutation | Child | Cytokines - pharmacology | Medical colleges | Care and treatment | Killer cells | Bacterial infections | Mycoses | Genetic aspects | Genetic transcription | Analysis | Cell proliferation | Pathogenesis | Helper cells | Cytotoxicity | Stimulation | Infections | Activation | Lymphocytes T | Kinases | Proteins | Fungi | α-Interferon | Interleukin 2 | Lymphocytes | Bacteria | Stat1 protein | Deoxyribonucleic acid--DNA | Binding | Pathogens | Interleukin 2 receptors | Cytokines | Chronic mucocutaneous candidiasis | Functional analysis | Stat5 protein | Interleukin 15 | Patients | Studies | γ-Interferon | Degranulation | Interferon | Viral infections
Journal Article