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Gastric Cancer, ISSN 1436-3291, 7/2013, Volume 16, Issue 3, pp. 301 - 308
Journal Article
Diabetology International, ISSN 2190-1678, 3/2017, Volume 8, Issue 1, pp. 52 - 58
We recently reported that vitamin D deficiency aggravates diabetic bone loss in mice. Although vitamin D affects both muscle and bone, the role of the vitamin... 
Medicine & Public Health | Vitamin D | Muscle wasting | Metabolic Diseases | Myoblast | Diabetes | Endocrinology
Journal Article
Journal of Bone and Mineral Metabolism, ISSN 0914-8779, 9/2016, Volume 34, Issue 5, pp. 517 - 525
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92686
Previous studies suggest that fracture healing is impaired in diabetes; however, the underlying mechanism remains unclear. Here, we investigated the roles of... 
FRACTURE | OBESITY | LACKING | RESORPTION | MULTIDISCIPLINARY SCIENCES | RISK | MECHANISMS | MELLITUS | TYPE-1 | DEFICIENCY | CARTILAGE | Bone and Bones - pathology | Body Weight | Femur - pathology | Chondrogenesis - genetics | Bone and Bones - injuries | Diabetes Mellitus, Experimental - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Female | Plasminogen Activator Inhibitor 1 - genetics | Diabetes Mellitus, Experimental - metabolism | Osteogenesis - genetics | Adipogenesis | Disease Models, Animal | Femur - injuries | Gene Expression | Femur - metabolism | Blood Glucose | Osteoclasts - metabolism | Plasminogen Activator Inhibitor 1 - blood | Mice, Knockout | Animals | Biomarkers | Mice | Wound Healing - physiology | RNA | Phosphatases | Alkaline phosphatase | Femur | Collagen (type I) | Streptozocin | mRNA | Defects | Cell adhesion & migration | Cartilage | Osteoporosis | Biomedical materials | Rodents | Extracellular matrix | Tumor necrosis factor-TNF | Biocompatibility | Physiology | Repair | Osteocalcin | Diabetes mellitus | Polyamide-imides | Bone healing | Gene expression | Medicine | Studies | Inhibitors | Plasminogen activator inhibitors | Insulin resistance | Diabetes | Bone | University faculty | Aggrecan | Laboratory animals | Apoptosis
Journal Article
Clinical calcium, ISSN 0917-5857, 03/2015, Volume 25, Issue 3, pp. 381 - 386
Journal Article
Clinical calcium, ISSN 0917-5857, 03/2015, Volume 25, Issue 3, p. 381
Vitamin D deficiency is associated with sarcopenia, which is characterized by the decrease in muscle mass and the muscle weakness. Active form of vitamin D... 
Animals | Aging - physiology | Bone and Bones - metabolism | Humans | Muscle Weakness - drug therapy | Muscle Weakness - metabolism | Muscle, Skeletal - metabolism | Vitamin D - metabolism | Receptors, Calcitriol - metabolism
Journal Article
BMC Cancer, ISSN 1471-2407, 10/2019, Volume 19, Issue 1, pp. 1 - 10
While denosumab has been shown to prevent skeletal-related events in patients with bone metastasis, there is a concern that it may cause atypical femoral... 
Care and treatment | Cancer patients | Usage | Fractures | Causes of | Development and progression | Denosumab | Health aspects | Risk factors | Systematic review | Bone metastasis | Femoral fractures | Retrospective studiess
Journal Article
Journal of Bone and Mineral Metabolism, ISSN 0914-8779, 3/2018, Volume 36, Issue 2, pp. 148 - 156
We recently revealed that plasminogen activator inhibitor-1 (PAI-1), a serine protease inhibitor, is involved in diabetes, osteoporosis and muscle wasting... 
Medicine & Public Health | Muscle wasting | Orthopedics | Metabolic Diseases | Insulin-like growth factor-1 | Muscle | Plasminogen activator inhibitor-1 | Glucocorticoid | TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | OLD DRUGS | ATROPHY | MECHANISMS | SKELETAL-MUSCLE | MURF1 | IN-VITRO | ENDOCRINOLOGY & METABOLISM | BONE | ATROGIN-1 | CARDIOVASCULAR RISK | Cell Line | Hemorrhagic Disorders - pathology | Humans | Muscle, Skeletal - metabolism | Mice, Knockout | Muscle Proteins - biosynthesis | Myoblasts - drug effects | Myoblasts - metabolism | Animals | Dexamethasone - pharmacology | Muscle Development - drug effects | Plasminogen Activator Inhibitor 1 - metabolism | Muscle, Skeletal - drug effects | Glucocorticoids - pharmacology | Female | Protein Biosynthesis - drug effects | Plasminogen Activator Inhibitor 1 - deficiency | Muscle, Skeletal - pathology | Hemorrhagic Disorders - metabolism | Insulin-Like Growth Factor I - metabolism | Osteoporosis | Corticosteroids | Protease inhibitors | RNA | Ligases | Physiological aspects | Thrombin | Steroids | Ubiquitin | Dexamethasone | Glucocorticoids | Insulin-like growth factor I | Serine | Diabetes mellitus | Paracrine signalling | AKT protein | Insulin-like growth factors | mRNA | siRNA | Proteinase inhibitors | Ribosomal protein S6 kinase | Rodents | Plasminogen activator inhibitors | Serine proteinase | Gastrocnemius muscle
Journal Article
Gastric Cancer, ISSN 1436-3291, 4/2014, Volume 17, Issue 2, pp. 348 - 353
Journal Article
Journal of Gastroenterology, ISSN 0944-1174, 3/2016, Volume 51, Issue 3, pp. 252 - 259
Hyperbilirubinemia, mild or moderate, is a commonly observed laboratory abnormality in chronic hepatitis C patients treated with simeprevir with pegylated... 
Pegylated interferon plus ribavirin | Simeprevir | Chronic hepatitis C | Medicine & Public Health | Colorectal Surgery | Hepatology | Gastroenterology | Abdominal Surgery | Inosine triphosphate pyrophosphatase | Surgical Oncology | Bilirubin increase | VARIANTS | PHASE-3 | PEGINTERFERON | ALPHA 2A | PROTECT | THERAPY | DOUBLE-BLIND | INFECTION | ANEMIA | GASTROENTEROLOGY & HEPATOLOGY | TREATMENT-NAIVE PATIENTS | Recombinant Proteins - therapeutic use | Prospective Studies | Humans | Middle Aged | Pyrophosphatases - genetics | Polyethylene Glycols - adverse effects | Male | Recombinant Proteins - adverse effects | Polyethylene Glycols - therapeutic use | Simeprevir - therapeutic use | Simeprevir - adverse effects | Hyperbilirubinemia - chemically induced | Female | Drug Therapy, Combination | Hyperbilirubinemia - genetics | Antiviral Agents - therapeutic use | Ribavirin - therapeutic use | Risk Factors | Interferon-alpha - therapeutic use | Hemoglobins - metabolism | Genotype | Treatment Outcome | Hepatitis C, Chronic - drug therapy | Hepatitis C, Chronic - blood | Bilirubin - blood | Ribavirin - adverse effects | Antiviral Agents - adverse effects | Hyperbilirubinemia - blood | Interferon alpha-2 | Aged | Interferon-alpha - adverse effects | Care and treatment | Analysis | Hemoglobin | Interferon | Genetic aspects | Bilirubin | Biological response modifiers | Hepatitis C | Ribavirin
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, p. e0136304
With increasing body weight, macrophages accumulate in adipose tissue. There, activated macrophages secrete numerous proinflammatory cytokines and chemokines,... 
ISCHEMIA-REPERFUSION INJURY | METABOLIC DISEASE | ALTERNATIVELY ACTIVATED MACROPHAGES | ADIPONECTIN | MULTIDISCIPLINARY SCIENCES | RESISTANCE | MICE | ARACHIDONIC-ACID | E-2 | LEPTIN RELEASE | ADIPOSE-TISSUE | Inflammation - pathology | Adipose Tissue - cytology | Male | Receptors, Prostaglandin E, EP4 Subtype - physiology | Adipose Tissue - metabolism | Inflammation - metabolism | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Diabetes Mellitus, Experimental - physiopathology | Disease Models, Animal | Receptors, Prostaglandin E, EP4 Subtype - agonists | Cytokines - metabolism | Signal Transduction | Obesity - complications | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Insulin Resistance | Methyl Ethers - pharmacology | Macrophages - cytology | Reverse Transcriptase Polymerase Chain Reaction | Chemokines - genetics | Inflammation - etiology | Macrophages - metabolism | Animals | Diabetes Mellitus, Type 2 - physiopathology | Macrophages - drug effects | Chemokines - metabolism | Mice | Macrophage Activation - drug effects | Adipose Tissue - drug effects | Obesity | Complications and side effects | Prostaglandins E | Physiological aspects | Inflammation | Research | Insulin | Risk factors | Polarization | Adipose tissue | Body weight | Prostaglandin E2 | Fuel consumption | Activation | Adipocytes | Glucose | Macrophages | Adiponectin | Receptors | Cell activation | Bioaccumulation | Rodents | Peroxidase | Diabetes mellitus (non-insulin dependent) | Phenotypes | Cytokines | Diabetes mellitus | Body weight gain | Glucose tolerance | Signaling | Food intake | Insulin resistance | Chemokines | Peritoneum
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0123982
Macrophages play crucial roles in repair process of various tissues. However, the details in the role of macrophages during bone repair still remains unknown.... 
CELLS | OSTEOCLASTS | GENE | ANGIOGENESIS | INFLAMMATORY PROTEIN-1-ALPHA | MULTIDISCIPLINARY SCIENCES | OSTEOBLASTS | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | RECEPTOR | ENDOTHELIAL GROWTH-FACTOR | UROKINASE | Fibrinolysis - drug effects | Femur - pathology | Tissue Plasminogen Activator - metabolism | Cartilage - pathology | Femur - blood supply | Cell Count | Chemokine CCL4 - genetics | Male | Plasminogen - metabolism | Cartilage - drug effects | RNA, Messenger - metabolism | Chemokine CCL3 - genetics | Time Factors | Chemokine CCL3 - metabolism | Chemokine CCL4 - metabolism | Chemokine CCL2 - metabolism | Wound Healing - drug effects | Phagocytosis - drug effects | Osteoblasts - drug effects | RNA, Messenger - genetics | Antibodies, Neutralizing - pharmacology | Chemokine CCL2 - genetics | Femur - drug effects | Osteoclasts - metabolism | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Macrophages - metabolism | Animals | Urokinase-Type Plasminogen Activator - metabolism | Sp7 Transcription Factor | Macrophages - drug effects | Mice | Osteoblasts - metabolism | Osteoclasts - drug effects | Tissue Plasminogen Activator - deficiency | Urokinase-Type Plasminogen Activator - deficiency | Plasminogen - deficiency | RNA | Macrophages | Femur | Multiple myeloma | Tissues | Defects | Recruitment | Proteins | Angiogenesis | Urokinase | Elevation | Physiology | Life sciences | Repair | Vascular endothelial growth factor | Immunoglobulins | Cytokines | Roles | IL-1β | Medicine | Injury prevention | Fractures | Bone | University faculty | Laboratory animals | Chemokines | Phagocytosis | Neutralization
Journal Article
Physiological Reports, ISSN 2051-817X, 10/2016, Volume 4, Issue 19, pp. e12979 - n/a
Journal Article