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Human Pathology, ISSN 0046-8177, 2010, Volume 41, Issue 12, pp. 1682 - 1693
Journal Article
CLINICAL NEPHROLOGY, ISSN 0301-0430, 01/2012, Volume 77, Issue 1, pp. 32 - 39
Aim: The transcription factor c-Jun is a major component of the activator protein-1 complex involved in renal physiological events, such as inflammation and... 
FIBROSIS | PATHWAYS | APOPTOSIS | chronic antibody-mediated rejection | JNK | PROTEIN-KINASE | kidney transplantation | INDUCTION | MESENCHYMAL TRANSITION | PATHOGENIC ROLE | GBM GLOMERULONEPHRITIS | p-c-Jun | c-Jun | UROLOGY & NEPHROLOGY | STRESS | CHRONIC ALLOGRAFT NEPHROPATHY
Journal Article
MOLECULAR PHARMACOLOGY, ISSN 0026-895X, 03/2003, Volume 63, Issue 3, pp. 478 - 488
In the present study, we examined the roles of G(12), G(13), G(q), and G(i) in endothelin-1-induced hypertrophic responses. Endothelin-1 stimulation activated... 
CELLS | HYPERTROPHY | STRESS FIBER FORMATION | P115 RHOGEF | DISSOCIATION | PROTEIN-COUPLED RECEPTOR | RAS | INVOLVEMENT | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | VENTRICULAR MYOCYTES
Journal Article
PROSTAGLANDINS & OTHER LIPID MEDIATORS, ISSN 1098-8823, 08/2002, Volume 68-9, pp. 95 - 114
The cyclooxygenase (COX, prostaglandin endoperoxide synthase) is a key enzyme in prostaglandin biosynthesis. Two isoforms of COX, COX-1 and COX-2, have been... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | cyclooxygenase | NECROSIS-FACTOR-ALPHA | JUN NH2-TERMINAL KINASE | MESSENGER-RNA STABILITY | CCAAT/ENHANCER-BINDING PROTEINS | CELL BIOLOGY | signaling pathways | NUCLEAR-FACTOR | CAMP-RESPONSE ELEMENT | PROSTAGLANDIN-ENDOPEROXIDE SYNTHASE-2 | VASCULAR ENDOTHELIAL-CELLS | C-JUN | isozyme | NF-KAPPA-B
Journal Article
Respiratory Research, ISSN 1465-9921, 03/2012, Volume 13, Issue 1, pp. 19 - 19
Background: Chronic obstructive pulmonary disease (COPD) is characterized by airflow obstruction and persistent inflammation in the airways and lung... 
IFNγ | Acute exacerbation | COPD | PROMOTER ACTIVITY | SIGNALING PATHWAYS | ACTIVATION | IFN gamma | THYMIC STROMAL LYMPHOPOIETIN | PROINFLAMMATORY CYTOKINE | OBSTRUCTIVE PULMONARY-DISEASE | INTERFERON-GAMMA | RESPIRATORY SYSTEM | acute exacerbation | A VIRUS-INFECTION | NF-KAPPA-B | T-CELLS | Cell Survival - drug effects | MAP Kinase Signaling System - physiology | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Oxidative Stress - physiology | Cells, Cultured | Hydrogen Peroxide - pharmacology | RNA, Messenger - metabolism | Interleukins - metabolism | Bronchi - drug effects | Signal Transduction - drug effects | Proto-Oncogene Proteins c-jun - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Bronchi - metabolism | Signal Transduction - physiology | Oxidative Stress - drug effects | Epithelial Cells - cytology | Bronchi - cytology | Cell Survival - physiology | Interferon-gamma - pharmacology | Oxidative stress | Lung diseases, Obstructive | Messenger RNA | DNA binding proteins | Interleukins | Cell culture | Thoracic surgery | Free radicals | Cytokines | Disease | Pathogenesis | Colleges & universities | RNA polymerase | Medicine | Polymerase chain reaction | Cell growth | Rodents | Nitric oxide | Ligands | Life sciences | Chemokines | Transcription factors | Hydrogen peroxide | Epithelial cells | Gene regulation | MEK inhibitors | Parenchyma | c-Jun protein | Intracellular signalling | MAP kinase | Inflammation | siRNA | Cyclic AMP response element-binding protein | Epithelium | Gene expression | Promoters | Respiratory tract | c-Jun amino-terminal kinase | Chronic obstructive pulmonary disease | Mutation | Research
Journal Article
Transplantation, ISSN 0041-1337, 05/2006, Volume 81, Issue 9, pp. 1325 - 1330
Background. In small intestinal ischemia reperfusion injury, we investigated the pathophysiological role of c-Jun NH2 terminal kinase (JNK) and p38 in order to... 
Journal Article
CIRCULATION RESEARCH, ISSN 0009-7330, 11/2002, Volume 91, Issue 10, pp. 961 - 969
In neonatal cardiomyocytes, activation of the G(q)-coupled alpha(1)-adrenergic receptor (alpha(1)AR) induces hypertrophy by activating mitogen-activated... 
MYOCARDIUM | c-Jun NH2-terminal kinase | CARDIAC & CARDIOVASCULAR SYSTEMS | alpha-adrenergic receptors | ACTIVATED PROTEIN-KINASE | STIMULATION | P115 RHOGEF | hypertrophy | INVOLVEMENT | OVEREXPRESSION | G family G proteins | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | BETA-GAMMA-SUBUNITS | JUN KINASE | RAT VENTRICULAR MYOCYTES | HEMATOLOGY
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2005, Volume 280, Issue 18, p. 18434
In the present study, we examined signal transduction mechanism of reactive oxygen species (ROS) production and the role of ROS in angiotensin II-induced... 
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 11/2002, Volume 91, Issue 10, pp. 961 - 969
ABSTRACT—In neonatal cardiomyocytes, activation of the Gq-coupled α1-adrenergic receptor (α1AR) induces hypertrophy by activating mitogen-activated protein... 
family G proteins | terminal kinase | adrenergic receptors | c-Jun NH | Hypertrophy
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 03/2003, Volume 63, Issue 3, p. 478
In the present study, we examined the roles of G 12 , G 13 , G q , and G i in endothelin-1-induced hypertrophic responses. Endothelin-1 stimulation activated... 
Journal Article
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