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PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, p. e0186227
Therapeutic agents to the central nervous system (CNS) need to be efficiently delivered to the target site of action at appropriate therapeutic levels.... 
CLEARANCE | OXIDATIVE STRESS | PROTEIN-SYNTHESIS SYSTEM | SUBSTRATE | MULTIDISCIPLINARY SCIENCES | MODEL | EXPRESSION | BRAIN | PEPTIDE | PROGRESSION | Neurons - pathology | Neuroprotective Agents - chemistry | Small Molecule Libraries - administration & dosage | Humans | Central Nervous System - pathology | Imidazoles - administration & dosage | Small Molecule Libraries - chemistry | Animals | alpha-2-HS-Glycoprotein - chemistry | Cattle | Hemopexin - metabolism | Central Nervous System - drug effects | alpha-2-HS-Glycoprotein - metabolism | Mice | Cell Death - drug effects | Hemopexin - chemistry | Oxidative Stress - drug effects | Culture Media - chemistry | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Acetamides - administration & dosage | Care and treatment | Nervous system diseases | Cell death | Amyotrophic lateral sclerosis | Glycoproteins | Research | Diagnosis | Health aspects | Drugs | Neuroprotection | Cell culture | Oxidative stress | Animal models | Biomedical research | Laboratories | Central nervous system | Hemopexin | Neuroblastoma | Serum proteins | Neuroprotective agents | Proteins | Blood-brain barrier | Rodents | Drug dosages | Injuries | Effectiveness | Glycoprotein | Pharmacology | Chemical compounds | Glycan | Neurological diseases | Medicine | Chemistry | Brain research | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87728
Amyotrophic lateral sclerosis (ALS) is an adult-onset motor neuron degenerative disease. Given that oxidative stress and resulting chronic neuronal... 
OXIDATIVE STRESS | ACTIVATION | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | GENES | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | ASTROCYTES | FAMILIAL ALS | NRF2-ARE PATHWAY | Inflammation - pathology | Spinal Cord - metabolism | Amyotrophic Lateral Sclerosis - physiopathology | Imidazoles - chemistry | Muscle, Skeletal - metabolism | Imidazoles - pharmacology | Amyotrophic Lateral Sclerosis - drug therapy | Nerve Tissue Proteins - metabolism | Amyotrophic Lateral Sclerosis - pathology | Inflammation - metabolism | Animals | Inflammation - drug therapy | Spinal Cord - pathology | Amyotrophic Lateral Sclerosis - metabolism | Mice | Spinal Cord - physiopathology | Muscle, Skeletal - pathology | Disease Models, Animal | Inflammation - physiopathology | Neurons | Cell death | Nitric oxide | Amyotrophic lateral sclerosis | Models | Inflammation | Permeability | Brain | Oxidative stress | Spinal cord | Biomedical research | Disease | Toxicity | Genes | DNA damage | Interleukin | Neuromodulation | Modulators | Antioxidants | Neurotoxicity | Rodents | Oxidation | Life sciences | Chemical synthesis | Drug dosages | Deoxyribonucleic acid--DNA | Glutathione | Motor neurons | Cell survival | Preservation | Immunomodulation | Oral administration | Muscles | Pharmacology | Chemical compounds | Nitric-oxide synthase | Skeletal muscle | Medicine | Gliosis | Quadriceps muscle | Brain injury | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 04/2006, Volume 26, Issue 8, pp. 2887 - 2900
Journal Article
American Journal of Cardiology, The, ISSN 0002-9149, 2013, Volume 111, Issue 12, pp. 1688 - 1693
Journal Article
Journal of Epidemiology, ISSN 0917-5040, 2019, Volume 29, Issue 1, pp. 33 - 37
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2012, Volume 53, Issue 11, pp. 2028 - 2042
Antioxidant defense is crucial in restoring cellular redox homeostasis. Recent findings have suggested that oxidative stress plays pivotal roles in the... 
Oxidative stress | Free radicals | Nrf2 | Cu/Zn superoxide dismutase | Transgenic mice | Virtual screening | Amyotrophic lateral sclerosis | BIOCHEMISTRY & MOLECULAR BIOLOGY | INTERACTIONS IN-SILICO | AMYOTROPHIC-LATERAL-SCLEROSIS | APOPTOSIS INHIBITORY PROTEIN | ANTIOXIDANT RESPONSE ELEMENT | TRANSCRIPTION FACTOR NRF2 | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | NEURON DISEASE | HYDROGEN-PEROXIDE | PARKINSONS-DISEASE | GENE-TRANSCRIPTION | Free Radical Scavengers - pharmacology | NF-E2-Related Factor 2 - physiology | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | Amyotrophic Lateral Sclerosis - physiopathology | Apoptosis - drug effects | Humans | Acetamides - pharmacology | Male | Antioxidant Response Elements | Amyotrophic Lateral Sclerosis - drug therapy | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Metabolic Detoxication, Phase II - genetics | Apoptosis Regulatory Proteins - genetics | Enzyme Induction - drug effects | Female | Glutamate-Cysteine Ligase - metabolism | Drug Evaluation, Preclinical | Cell Survival - drug effects | Signal Transduction | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Heme Oxygenase-1 - antagonists & inhibitors | NAD(P)H Dehydrogenase (Quinone) - antagonists & inhibitors | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Amyotrophic Lateral Sclerosis - pathology | Animals | NF-E2-Related Factor 2 - metabolism | Acetylcysteine - pharmacology | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Thiazoles - pharmacology | HeLa Cells | Oxidative Stress - drug effects | Glutamate-Cysteine Ligase - antagonists & inhibitors | Lipid Peroxidation
Journal Article
British Journal of Nutrition, ISSN 0007-1145, 03/2018, Volume 119, Issue 9, pp. 1 - 11
Diabetes mellitus is a metabolic disease spreading worldwide that has been reported to worsen the development and progression of other diseases (cancer,... 
Antidiabetic rice | Mutant rice lines | C-peptide | Insulin secretion | LOW-CARBOHYDRATE DIET | AMYLOSE | NUTRITION & DIETETICS | ENDOSPERM | RESISTANT STARCH | NUTRITION EDUCATION | GLUCOSE | JAPANESE | CELL | SPECTROMETRY | CONSUMPTION | Ornithine - blood | Area Under Curve | Oryza - metabolism | 1,4-alpha-Glucan Branching Enzyme - deficiency | Glycylglycine - blood | Male | Diabetes Mellitus, Type 2 - metabolism | Starch Synthase - metabolism | Oryza - classification | Insulin Secretion - genetics | 1,4-alpha-Glucan Branching Enzyme - metabolism | Oryza - genetics | Starch Synthase - genetics | 1,4-alpha-Glucan Branching Enzyme - genetics | Gene Expression Regulation, Plant | Insulin Secretion - drug effects | Glucose Tolerance Test | Diabetes Mellitus, Type 2 - prevention & control | Rats | Blood Glucose | Pancreas - metabolism | Oryza - enzymology | Rats, Sprague-Dawley | Hypoglycemic Agents - pharmacology | Gene Expression Regulation, Enzymologic | Insulin - metabolism | Animals | Mutation | Starch Synthase - deficiency | Animal Feed | Cluster Analysis | Cluster analysis | Animal models | Glucose | Powder | Elevation | Vascular diseases | Metabolites | Dementia disorders | Starch synthase | Pancreas | Diabetes mellitus (non-insulin dependent) | Enzyme-linked immunosorbent assay | Food | Rice | Cultivars | Carbohydrates | Enzymes | Vascular dementia | Incubation | Starch | Secretion | Diabetes mellitus | Flour | Medical screening | Insulin | Diet | Scientific imaging | Plasma levels | Diabetes | Ornithine | Mass spectrometry | Wheat | Deficient mutant | Metabolic disorders | Cancer
Journal Article
Experimental Neurology, ISSN 0014-4886, 2011, Volume 232, Issue 1, pp. 41 - 52
Journal Article
Journal of Nutrition Education and Behavior, ISSN 1499-4046, 07/2019, Volume 51, Issue 7, pp. S8 - S9
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 09/2017, Volume 377, Issue 12, pp. 1119 - 1131
Patients with myocardial infarction and a high-sensitivity CRP level of 2 mg or more per liter were assigned to one of three canakinumab doses or placebo. The... 
C-REACTIVE PROTEIN | MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | MYOCARDIAL-INFARCTION | STATIN THERAPY | CLONAL HEMATOPOIESIS | INFLAMMATION | CARDIOVASCULAR-DISEASE | INTERLEUKIN-1-BETA INHIBITION | RISK | CORONARY-ARTERY-DISEASE | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Secondary Prevention | Infection - etiology | Incidence | Dose-Response Relationship, Drug | Anti-Inflammatory Agents - adverse effects | C-Reactive Protein - metabolism | Lipids - blood | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Anti-Inflammatory Agents - administration & dosage | Female | Neutropenia - chemically induced | Interleukin-1beta - antagonists & inhibitors | Stroke - prevention & control | Atherosclerosis - drug therapy | Double-Blind Method | Interleukin-1beta - immunology | Atherosclerosis - blood | Myocardial Infarction - drug therapy | Antibodies, Monoclonal - administration & dosage | Aged | Myocardial Infarction - prevention & control | Myocardial infarction | Cerebral infarction | Stroke | Heart attacks | C-reactive protein | Cytokines | Clinical trials | Cardiovascular disease | Innate immunity | Inflammation | Angina | Angina pectoris | Thrombosis | Cholesterol | Disease prevention | Proteins | Arteriosclerosis | Atherosclerosis | Cardiovascular diseases | Health risk assessment | Drug therapy | Drug dosages | Klinisk medicin | Clinical Medicine
Journal Article