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Neuro-Oncology, ISSN 1522-8517, 04/2019, Volume 21, Issue Supplement_2, pp. ii68 - ii69
Abstract Despite our increased understanding of the genetic make-up and new therapies for pediatric high grade glioma (pHGG) and Diffuse Intrinsic Pontine... 
Journal Article
Journal of Neurosurgery, ISSN 0022-3085, 08/2013, Volume 119, Issue 2, pp. 530 - 531
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2014, Volume 16, Issue suppl 5, pp. v197 - v197
Changes in splicing have been implicated in numerous pathologies including cancer. Previously, our group identified Baf45d as aberrantly spliced in GBM using... 
Journal Article
CNS Oncology, ISSN 2045-0907, 07/2015, Volume 4, Issue 4, pp. 265 - 272
Journal Article
World Neurosurgery, ISSN 1878-8750, 01/2018, Volume 109, pp. e845 - e852
Corticosteroids are routinely used to treat brain tumors. Although steroids have an immediate clinical benefit, their use can lead to a number of relevant... 
Overall survival | Surgery | Glioblastoma | Steroids | SURVIVAL | DEXAMETHASONE | MANAGEMENT | PROGNOSTIC-FACTORS | NEWLY-DIAGNOSED GLIOBLASTOMA | CHEMOTHERAPY | CLINICAL NEUROLOGY | GUIDELINE | MALIGNANT GLIOMA | BRAIN METASTASES | CORTICOSTEROIDS
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e26740
We undertook this study to understand how the transcription factor Sox2 contributes to the malignant phenotype of glioblastoma multiforme (GBM), the most... 
HUMAN GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | NEUROGENESIS | STATE | DIFFERENTIATION | IDENTIFICATION | EXPRESSION | BRAIN | TUMOR-INITIATING CELLS | PLURIPOTENT | Cell Proliferation | Epigenomics | Neoplasm Invasiveness | Humans | Gene Expression Regulation, Neoplastic | Gene Silencing | Promoter Regions, Genetic - genetics | Blotting, Western | Glioma - metabolism | SOXB1 Transcription Factors - metabolism | Glioma - genetics | Azacitidine - pharmacology | DNA Methylation | Phenotype | Gene Amplification | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Glioma - pathology | CpG Islands | Neoplastic Stem Cells - pathology | DNA, Neoplasm - genetics | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Cell Movement | Epigenetic inheritance | Gliomas | Analysis | Brain tumors | Stem cells | Genetic research | Genetic aspects | Methylation | Brain | Biotechnology | Leukocyte migration | Brain stem | Brain cancer | Glioblastoma | Oncology | Genomes | Neurosurgery | Neurogenesis | Proteins | Genotype & phenotype | Neurodegeneration | Glioma cells | DNA methylation | Pituitary gland | Trends | Sox2 protein | CpG islands | Medical research | Invasiveness | Data processing | Gene expression | Glioblastoma multiforme | Esophagus | Studies | Gene amplification | Brain research | Hospitals | Cell lines | Cell migration | Cancer | Tumors
Journal Article
Neurocirugía, ISSN 1130-1473, 11/2017, Volume 28, Issue 6, pp. 260 - 265
Journal Article
Neurocirugía, ISSN 1130-1473, 11/2017, Volume 28, Issue 6, pp. 260 - 265
La patología de craneosinostosis es infrecuente y requiere una intervención quirúrgica muy individualizada, por lo que consideramos que la práctica con modelos... 
Planificación quirúrgica | Craniosynostosis | Impresión 3 D | Neurosurgery | Craneosinostosis | Surgical planning | Neurocirugía | 3D printing
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e77098
Glioblastoma multiforme (GBM)-initiating cells (GICs) represent a tumor subpopulation with neural stem cell-like properties that is responsible for the... 
IN-VITRO | GLIOMA | MULTIDISCIPLINARY SCIENCES | GROWTH | TARGET RECOGNITION | SELF-RENEWAL | MICRORNAS | EXPRESSION | INHIBIT | TUMOR-INITIATING CELLS | BRAIN-TUMORS | Neurons - pathology | Oligonucleotide Array Sequence Analysis | Humans | Astrocytes - pathology | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Myeloid Cell Leukemia Sequence 1 Protein - metabolism | Spheroids, Cellular - pathology | Apoptosis - genetics | MicroRNAs - metabolism | Gene Expression Profiling | Glial Fibrillary Acidic Protein - metabolism | Phosphoproteins - metabolism | Brain Neoplasms - surgery | Cell Differentiation - genetics | Tubulin - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Female | Membrane Proteins - metabolism | Neurons - metabolism | Spheroids, Cellular - metabolism | Brain Neoplasms - genetics | Glioblastoma - surgery | Nestin - metabolism | Animals | Glioblastoma - pathology | Cell Line, Tumor | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Astrocytes - metabolism | MicroRNA | Cell differentiation | Health aspects | Glioblastoma multiforme | Stem cells | Regulators | Neurosciences | Senescence | Pathogenesis | Brain cancer | Glioblastoma | Differentiation (biology) | Oncology | mRNA | Neurosurgery | Cancer therapies | Proteins | Medical research | Markers | MiRNA | Gene expression | Mcl-1 protein | Chemotherapy | Overexpression | Ribonucleic acids | MicroRNAs | Medical prognosis | Cell lines | Neural stem cells | Apoptosis | Tumors
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2014, Volume 16, Issue Suppl 5, pp. v197 - v197
Changes in splicing have been implicated in numerous pathologies including cancer. Previously, our group identified Baf45d as aberrantly spliced in GBM using... 
Abstracts
Journal Article
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