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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 1, pp. 264 - 269
MicroRNA (miRNAs) are negative regulators of gene expression and can function as tumor suppressors or oncogenes. Expression patterns of miRNAs and their role... 
Tumor cell line | Liver cancer | Cell growth | MicroRNA | Liver | Cell lines | Stem cells | Hepatocellular carcinoma | Signatures | Tumors | Mouse model | AntagomiRs | Hepatocarcinogenesis | DDIT4 | MIR-34A | mouse model | MULTIDISCIPLINARY SCIENCES | CELL LINES | PROLIFERATION | MECHANISMS | hepatocarcinogenesis | IDENTIFICATION | antagomiRs | CANCER | GENE | microRNA | TUMOR-SUPPRESSOR | HUMAN HEPATOCELLULAR-CARCINOMA | MICRORNA EXPRESSION | Liver - pathology | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Gene Expression Profiling | Fibrosis - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Microarray Analysis | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Diagnosis, Differential | Fibrosis - genetics | Reproducibility of Results | Liver Neoplasms - genetics | Tumor Suppressor Protein p53 - metabolism | Transcription Factors - genetics | Mice, Knockout | Transcription Factors - metabolism | Animals | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Liver - physiology | Mice | MicroRNAs - genetics | Fibrosis - pathology | Carcinoma, Hepatocellular - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Development and progression | Genetic aspects | Research | Gene expression | Carcinogenesis | Risk factors | Biological Sciences
Journal Article
Oncologist, ISSN 1083-7159, 03/2017, Volume 22, Issue 3, pp. 359 - 360
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2008, Volume 105, Issue 34, pp. 12417 - 12422
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1062 - 1073
Here, we show that autophagy is activated in the intestinal epithelium in murine and human colorectal cancer and that the conditional inactivation of Atg7 in... 
AUTOPHAGY GENE ATG16L1 | DENDRITIC CELLS | COLORECTAL-CANCER | INFLAMMATION | MOUSE | REGULATORY T-CELLS | APC(MIN) MICE | LUNG-TUMORS | COLON | TUMORIGENESIS | CELL BIOLOGY | Adenocarcinoma - pathology | Cell Proliferation | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Adenoma - prevention & control | Immunity, Mucosal | Adenoma - immunology | Colon - immunology | Male | Dysbiosis | Time Factors | Cell Transformation, Neoplastic - genetics | CD8-Positive T-Lymphocytes - metabolism | Disease Models, Animal | Microbiota - immunology | Colon - pathology | Adenocarcinoma - immunology | Colorectal Neoplasms - microbiology | Tumor Burden | Ubiquitin-Activating Enzymes - metabolism | Colon - metabolism | Mice, Knockout | Host-Pathogen Interactions | Cell Cycle Checkpoints | Adenocarcinoma - prevention & control | Colorectal Neoplasms - immunology | Enzyme Activation | CD8-Positive T-Lymphocytes - immunology | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Adenoma - microbiology | Autophagy | Genes, APC | Female | Adenocarcinoma - genetics | Microtubule-Associated Proteins - deficiency | CD8-Positive T-Lymphocytes - microbiology | Adenocarcinoma - microbiology | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Colorectal Neoplasms - prevention & control | Cell Transformation, Neoplastic - metabolism | Autophagy-Related Protein 7 | Cell Transformation, Neoplastic - immunology | Animals | Adenoma - pathology | Colon - microbiology | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Autophagy (Cytology) | Cellular proteins | Immune response | Colorectal cancer | Development and progression | Genetic aspects | Properties | Gene expression | Health aspects
Journal Article
British Journal of Cancer, ISSN 0007-0920, 10/2016, Volume 115, Issue 8, p. e7
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 04/2019, Volume 247, Issue 4, pp. 435 - 443
Hepatocellular carcinomas (HCCs) are known to be highly heterogenous. Within the extensive histopathological and molecular heterogeneity of HCC, tumors with... 
CTNNB1 | liver | LKB1 | hepatocellular carcinoma | HOMEOSTASIS | KINASE | MOUSE | PATHOLOGY | CANCER | GENE | METABOLISM | ONCOLOGY | TUMOR-SUPPRESSOR | Cluster analysis | Energy metabolism | Wnt protein | Transcription | Liver | Crosstalk | Hepatocellular carcinoma | Epistasis | Hepatoma | Proteins | Genotype & phenotype | LKB1 protein | Heterogeneity | Clonal deletion | Deletion | Polarity | Tumorigenesis | Catenin | Phenotypes | Zonation | Metabolism | Gene expression | Clustering | Signaling | Cell lines | Mutation
Journal Article
Modern Pathology, ISSN 0893-3952, 01/2014, Volume 27, Issue 1, pp. 73 - 86
Intraductal papillary neoplasms of the bile duct are still poorly characterized regarding (1) their molecular alterations during the development to invasive... 
GNAS | bile duct tumors | KRAS | intraductal precursor lesion | CHOLANGIOCARCINOMA | TRACT CANCERS | PANCREAS | PATHOLOGY | GNAS MUTATIONS | BILIARY INTRAEPITHELIAL NEOPLASIA | GROWTH-FACTOR RECEPTOR | IMMUNOHISTOCHEMICAL ANALYSIS | MUCINOUS NEOPLASM | EXPRESSION | CLINICOPATHOLOGICAL FEATURES | Immunohistochemistry | Bile Ducts, Intrahepatic - chemistry | ras Proteins - genetics | Carcinoma in Situ - chemistry | Proto-Oncogene Proteins p21(ras) | Prognosis | Humans | Middle Aged | Male | Carcinoma, Intraductal, Noninfiltrating - genetics | Carcinoma, Intraductal, Noninfiltrating - mortality | Papilloma - chemistry | Tumor Suppressor Protein p53 - analysis | Carcinoma in Situ - genetics | Cyclin-Dependent Kinase Inhibitor p16 - analysis | Neoplasm Grading | Time Factors | DNA Mutational Analysis | Bile Duct Neoplasms - chemistry | Aged, 80 and over | Adult | Female | Papilloma - pathology | Bile Duct Neoplasms - genetics | Papilloma - genetics | Bile Ducts, Intrahepatic - pathology | Signal Transduction | Biomarkers, Tumor - analysis | Carcinoma in Situ - pathology | Neoplasm Invasiveness | Europe | Kaplan-Meier Estimate | Proportional Hazards Models | Proto-Oncogene Proteins - genetics | Papilloma - mortality | Carcinoma, Intraductal, Noninfiltrating - chemistry | Disease Progression | Cholangiocarcinoma - pathology | Carcinoma, Intraductal, Noninfiltrating - pathology | Biopsy | Cholangiocarcinoma - chemistry | Bile Duct Neoplasms - mortality | Cholangiocarcinoma - genetics | Aged | Biomarkers, Tumor - genetics | Bile Duct Neoplasms - pathology | Mutation | Carcinoma in Situ - mortality
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 08/2015, Volume 39, Issue 8, pp. 1156 - 1157
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 12/2015, Volume 39, Issue 12, pp. 1730 - 1741
Journal Article