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Science, ISSN 0036-8075, 10/2013, Volume 342, Issue 6156, pp. 373 - 377
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2009, Volume 119, Issue 11, pp. 3437 - 3449
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2012, Volume 122, Issue 4, pp. 1377 - 1392
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 11/2006, Volume 20, Issue 13, pp. 2313 - 2320
Recent studies suggest that moderate red wine consumption reduces the incidence of Alzheimer's disease (AD) clinical dementia. Using Tg2576 mice, which model... 
Polyphenols | APP | Nonamyloidogenic | AD dementia | polyphenols | WINE | DEMENTIA | DEPOSITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | COGNITIVE IMPAIRMENT | AMYLOID PRECURSOR PROTEIN | nonamyloidogenic | CLEAVAGE | SENILE PLAQUES | CELL BIOLOGY | ALCOHOL | BIOLOGY | ASSOCIATION | TRANSGENIC MICE
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 48, pp. 12788 - 12797
Activated protein C (APC) is a serine protease with anticoagulant and direct cytoprotective activities. Early postischemic APC application activates the... 
Angiogenesis | Neuroprotection | Serine protease | Activated protein C | Neurogenesis | Transient cerebral ischemia | FUNCTIONAL RECOVERY | ARTERY OCCLUSION | angiogenesis | FOCAL CEREBRAL-ISCHEMIA | CELL-PROLIFERATION | SUBVENTRICULAR ZONE | serine protease | NEUROSCIENCES | NEURONAL DAMAGE | transient cerebral ischemia | TISSUE-PLASMINOGEN-ACTIVATOR | neuroprotection | IN-VIVO | neurogenesis | EMBOLIC STROKE | activated protein C | SEVERE SEPSIS | Neovascularization, Physiologic - drug effects | Recovery of Function - drug effects | Nerve Regeneration - physiology | Blood-Brain Barrier - physiopathology | Brain - enzymology | Male | Cell Movement - physiology | Protein C - pharmacology | Brain - metabolism | Brain Ischemia - enzymology | Recovery of Function - physiology | Neurogenesis - drug effects | Tissue Plasminogen Activator - toxicity | Brain Infarction - drug therapy | Disease Models, Animal | Brain Infarction - enzymology | Reperfusion Injury - enzymology | Brain - physiopathology | Mice, Inbred C57BL | Cerebral Arteries - physiopathology | Treatment Outcome | Brain Ischemia - physiopathology | Brain Infarction - physiopathology | Blood-Brain Barrier - drug effects | Receptor, PAR-1 - genetics | Blood-Brain Barrier - metabolism | Mice, Knockout | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Animals | Cerebral Arteries - enzymology | Cerebral Arteries - metabolism | Neurogenesis - physiology | Brain Ischemia - drug therapy | Reperfusion Injury - physiopathology | Cell Proliferation - drug effects | Mice | Protein C - metabolism | Enzyme Activation - physiology | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2012, Volume 122, Issue 4, p. 1377
In Alzheimer disease (AD), amyloid [beta] peptide (A[beta]) accumulates in plaques in the brain. Receptor for advanced glycation end products (RAGE) mediates... 
Care and treatment | Amyloid beta-protein | Models | Diagnosis | Research | Properties | Alzheimer's disease | Protein binding
Journal Article
Neurosurgery, ISSN 0148-396X, 01/2010, Volume 66, Issue 1, pp. 165 - 171
Journal Article