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1. Full Text Hexavalent Chromium in Drinking Water
by Moffat, Ivy and Martinova, Nadia and Seidel, Chad and Thompson, Chad M
Journal ‐ American Water Works Association, ISSN 0003-150X, 05/2018, Volume 110, Issue 5, pp. E22 - E35
The risk assessment of chromium in drinking water is complex. To better understand this complexity, the essential information on exposure, analytical and... 
drinking water | hexavalent chromium | mode of action | carcinogen | PHARMACOKINETIC MODEL | STEM-CELLS | CARCINOGENIC MODES | PILOT-SCALE | WATER RESOURCES | CANCER | MICE FOLLOWING EXPOSURE | ENGINEERING, CIVIL | ORAL-EXPOSURE | REMOVAL | HUMAN INGESTION | F344 RATS
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2. Hexavalent Chromium in Drinking Water
by Ivy Moffat and Nadia Martinova and Chad Seidel and Chad M Thompson
American Water Works Association. Journal, ISSN 0003-150X, 05/2018, Volume 110, Issue 5, p. E22
The risk assessment of chromium in drinking water is complex. To better understand this complexity, the essential information on exposure, analytical and... 
Drinking water | Analytical methods | Soil erosion | Pollution sources | Methodology | Toxicity | Drinking | Sediment pollution | Industrial pollution | Contamination | Erosion rates | Complexity | Mode of action | Studies | Risks | Hexavalent chromium | Soils | Toxicology | Intestine | Risk assessment | Chromium | Cancer | Soil contamination | Erosion
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3. Full Text Development of an oral reference dose for the perfluorinated compound GenX
by Thompson, Chad M and Fitch, Seneca E and Ring, Caroline and Rish, William and Cullen, John M and Haws, Laurie C
Journal of Applied Toxicology, ISSN 0260-437X, 09/2019, Volume 39, Issue 9, pp. 1267 - 1282
Ammonium 2,3,3,3‐tetrafluoro‐2‐(heptafluoropropoxy)‐propanoate, also known as GenX, is a processing aid used in the manufacture of fluoropolymers. GenX is one... 
risk assessment | benchmark dose (BMD) modeling | GenX | reference dose (RfD) | per‐ and polyfluoroalkyl substances (PFAS) | per- and polyfluoroalkyl substances (PFAS) | WORKSHOP | PERFLUOROALKYL | MOUSE | SUBSTANCES | RISK | FATTY-ACIDS | TUMORS | CARBON-CHAIN LENGTH | MODES | DRINKING-WATER CONTAMINANTS | TOXICOLOGY | Drinking water | Ammonium | Regulators | Water quality standards | Toxicity | Bioassays | Hazard identification | Genotoxicity | Fluoropolymers | Benchmarks | International standardization | Perfluoro compounds | Organic chemistry | Contaminants | Mathematical models | Lesions | Bayesian analysis
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4. Full Text Hexavalent Chromium in Drinking Water
: JOURNAL AWWA
by Moffat, Ivy and Martinova, Nadia and Seidel, Chad and Thompson, Chad M
Journal - American Water Works Association, ISSN 0003-150X, 05/2018, Volume 110, Issue 5, pp. E22 - E35
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5. Full Text Review of transcriptomic responses to hexavalent chromium exposure in lung cells supports a role of epigenetic mediators in carcinogenesis
by Rager, Julia E and Suh, Mina and Chappell, Grace A and Thompson, Chad M and Proctor, Deborah M
Toxicology Letters, ISSN 0378-4274, 05/2019, Volume 305, pp. 40 - 50
Inhalation exposure to hexavalent chromium [Cr(VI)] is associated with increased risk of lung cancer with a mode of action (MOA) postulated to involve... 
Hexavalent chromium | Transcriptomics | Lung cancer | Risk assessment | Epigenetics | Mechanism of action | Mode of action | DOWN-REGULATION | DOUBLE-STRAND BREAKS | TRANSLATION INITIATION | CANCER | DAMAGE | SODIUM DICHROMATE | NICKEL | GENE-EXPRESSION | TOXICOLOGY | CHROMATE | CYCLE | Epigenetic inheritance | Chromatin | MicroRNA | Methyltransferases | DNA damage | Analysis | Oncology, Experimental | Research | Methylation | Genetic translation | Cancer | Index Medicus
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6. Full Text Letter to the Editor Regarding Banu et al. (2018). Chromium Accumulation on Human Placental Oxidative Stress and Apoptosis
by Thompson, Chad M and Suh, Mina and Proctor, Deborah M and Harris, Mark A
Toxicological Sciences, ISSN 1096-6080, 10/2018, Volume 165, Issue 2, pp. 269 - 271
TOXICOLOGY | Pregnancy | Placenta | Oxidative Stress | Humans | Chromium | Female | Apoptosis | Letters to the Editor
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7. Full Text Identifying Attributes That Influence In Vitro-to-In Vivo Concordance by Comparing In Vitro Tox21 Bioactivity Versus In Vivo DrugMatrix Transcriptomic Responses Across 130 Chemicals
by Klaren, William D and Ring, Caroline and Harris, Mark A and Thompson, Chad M and Borghoff, Susan and Sipes, Nisha S and Hsieh, Jui-Hua and Auerbach, Scott S and Rager, Julia E
Toxicological Sciences, ISSN 1096-6080, 01/2019, Volume 167, Issue 1, pp. 157 - 171
Abstract Recent efforts aimed at integrating in vitro high-throughput screening (HTS) data into chemical toxicity assessments are necessitating increased... 
high-throughput screening | DRUG DISCOVERY | toxicokinetics | RECEPTOR | TOXICITY | Tox21 | TOXICOGENOMICS | MODELS | CARCINOGENICITY | ARTIFACTS | DrugMatrix | transcriptomics | TOXICOLOGY | HEPATOTOXICITY | EXPRESSION | EXPOSURE | in vitro-to-in vivo | Tox21 IN VITRO TO IN VIVO CONCORDANCE
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8. Full Text Commentary on New Formaldehyde Studies in Trp53 Haploinsufficient Mice: Further Support for Nonlinear Risks From Inhaled Formaldehyde
by Thompson, Chad M
Dose-Response, ISSN 1559-3258, 5/2018, Volume 16, Issue 2, p. 1559325818777931
dose–response | risk assessment | formaldehyde | mode of action | CARCINOGENICITY | INHALATION | dose-response | RATS | PHARMACOLOGY & PHARMACY | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | EXPOSURE
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9. Full Text Identifying Attributes That InfluenceIn Vitro-to-In VivoConcordance by ComparingIn VitroTox21 Bioactivity VersusIn VivoDrugMatrix Transcriptomic Responses Across 130 Chemicals
by Klaren, William D and Ring, Caroline and Harris, Mark A and Thompson, Chad M and Borghoff, Susan and Sipes, Nisha S and Hsieh, Jui-Hua and Auerbach, Scott S and Rager, Julia E
Toxicological Sciences, ISSN 1096-6080, 01/2019, Volume 167, Issue 1, pp. 157 - 171
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10. Full Text Dose-dependence of chemical carcinogenicity: Biological mechanisms for thresholds and implications for risk assessment
by Clewell, Rebecca A and Clewell, Harvey J and Thompson, Chad M
Chemico-Biological Interactions, ISSN 0009-2797, 03/2019, Volume 301, pp. 112 - 127
Current regulatory practices for chemical carcinogens were established when scientific understanding of the molecular mechanisms of chemical carcinogenesis was... 
RESPONSE RELATIONSHIPS | INHALED FORMALDEHYDE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN CROSS-LINKS | CELL-PROLIFERATION | MICE FOLLOWING EXPOSURE | HEXAVALENT CHROMIUM | IN-VITRO | GENE MUTATION ASSAY | CANCER-RISK | PHARMACOLOGY & PHARMACY | F344 RATS | TOXICOLOGY | Dose-Response Relationship, Drug | Animals | Risk Assessment | Humans | Social Control, Formal | Carcinogens - toxicity | Case studies | Research | Gene mutations | Risk assessment | Oncology, Experimental | Cancer | Index Medicus
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11. Full Text Benchmark Dose Modeling Estimates of the Concentrations of Inorganic Arsenic That Induce Changes to the Neonatal Transcriptome, Proteome, and Epigenome in a Pregnancy Cohort
by Rager, Julia E and Auerbach, Scott S and Chappell, Grace A and Martin, Elizabeth and Thompson, Chad M and Fry, Rebecca C
Chemical Research in Toxicology, ISSN 0893-228X, 10/2017, Volume 30, Issue 10, pp. 1911 - 1920
Prenatal inorganic arsenic (iAs) exposure influences the expression of critical genes and proteins associated with adverse outcomes in newborns, in part... 
RESPONSES | CHEMISTRY, MEDICINAL | GENE-EXPRESSION | RATS | TOXICOLOGY | BIRTH | MICRORNAS | HEALTH | RISK-ASSESSMENT | CHEMISTRY, MULTIDISCIPLINARY | EXPOSURE | Epigenomics | Models, Chemical | Humans | Transcriptome | Benchmarking | Male | Proteome | Arsenic - analysis | Pregnancy | Young Adult | Adolescent | Adult | Female | Infant, Newborn | Cohort Studies | Index Medicus
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12. Full Text An Adverse Outcome Pathway (AOP) for forestomach tumors induced by non-genotoxic initiating events
by Proctor, Deborah M and Suh, Mina and Chappell, Grace and Borghoff, Susan J and Thompson, Chad M and Wiench, Karin and Finch, Lavorgie and Ellis-Hutchings, Robert
Regulatory Toxicology and Pharmacology, ISSN 0273-2300, 07/2018, Volume 96, pp. 30 - 40
The utility of rodent forestomach tumor data for hazard and risk assessment has been examined for decades because humans do not have a forestomach, and these... 
Ethyl acrylate | Cytotoxicity | Forestomach tumors | Adverse outcome pathway (AOP) | Butylated hydroxyanisole (BHA) | RAT FORESTOMACH | MEDICINE, LEGAL | INDUCED GASTRIC TOXICITY | CELL-PROLIFERATION | CAFFEIC ACID | RODENT FORESTOMACH | PHARMACOLOGY & PHARMACY | F344 RATS | TOXICOLOGY | BUTYLATED HYDROXYANISOLE | RISK-ASSESSMENT | B6C3F1 MICE | Animals | Risk Assessment | Acrylates - pharmacology | Humans | Stomach Neoplasms - chemically induced | Stomach Neoplasms - pathology | Cell Proliferation - drug effects | Adverse Outcome Pathways | Acrylates - adverse effects | Index Medicus
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13. Full Text Assessment of the mode of action underlying development of forestomach tumors in rodents following oral exposure to ethyl acrylate and relevance to humans
by Thompson, Chad M and Suh, Mina and Proctor, Deborah M and Chappell, Grace and Borghoff, Susan and Ellis-Hutchings, Robert and Wiench, Karin and Finch, Lavorgie
Regulatory Toxicology and Pharmacology, ISSN 0273-2300, 07/2018, Volume 96, pp. 178 - 189
Chronic repeated gavage dosing of high concentrations of ethyl acrylate (EA) causes forestomach tumors in rats and mice. For two decades, there has been... 
Ethyl acrylate | Forestomach | Carcinogenesis | Mode of action (MOA) | Risk assessment | MEDICINE, LEGAL | METHYL-METHACRYLATE | INDUCED GASTRIC TOXICITY | HA-RAS MICE | IMPROVED GENOTOXICITY TESTS | MOUSE LYMPHOMA-CELLS | NONGENOTOXIC CHEMICALS | COMPUTATIONAL FLUID-DYNAMICS | PHARMACOLOGY & PHARMACY | TOXICOLOGY | BUTYLATED HYDROXYANISOLE | TRIPROPYLENE GLYCOL DIACRYLATE | RISK-ASSESSMENT | Animals | Acrylates - chemistry | Administration, Oral | Humans | Rats | Stomach Neoplasms - chemically induced | Molecular Structure | Acrylates - toxicity | Acrylates - administration & dosage | Index Medicus
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