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Nature, ISSN 0028-0836, 10/2010, Volume 467, Issue 7318, pp. 972 - 976
Reciprocity of inflammation, oxidative stress and neovascularization is emerging as an important mechanism underlying numerous processes from tissue healing... 
SYSTEM | TOLL-LIKE RECEPTOR-2 | CELLS | PATTERN-RECOGNITION | MULTIDISCIPLINARY SCIENCES | MACULAR DEGENERATION | CD36 | CHRONIC INFLAMMATION | CANCER | INNATE IMMUNITY | Neovascularization, Physiologic - drug effects | Toll-Like Receptor 2 - agonists | Melanoma - blood supply | Humans | Oxidative Stress - physiology | Vascular Endothelial Growth Factor A - metabolism | Inflammation - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Wound Healing - drug effects | Receptors, Scavenger - metabolism | Melanoma - metabolism | Cell Line | Pyrroles - metabolism | Oxidation-Reduction | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Ischemia - metabolism | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Immunity, Innate - immunology | Pyrroles - pharmacology | Animals | Signal Transduction - drug effects | Propionates | Pyrroles - chemistry | Ligands | Mice | Neovascularization, Pathologic - metabolism | Wound Healing - physiology | Aorta - cytology | Hindlimb - metabolism | Myeloid Differentiation Factor 88 - metabolism | rac1 GTP-Binding Protein - metabolism | Aging - metabolism | Cell Movement | Oxidative stress | Complications and side effects | Cell receptors | Physiological aspects | Genetic aspects | Research | Neovascularization | Vascular endothelial growth factor | Angiogenesis | Wound healing | Rodents | Atherosclerosis
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