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Nature Medicine, ISSN 1078-8956, 05/2017, Volume 23, Issue 5, pp. 568 - 578
Chromosomal instability (CIN), a feature of most adult neoplasms from their early stages onward, is a driver of tumorigenesis. However, several malignancy... 
MOLECULAR CHARACTERIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATED PROTEIN-KINASE | CYCLIN-E | INSTABILITY | PROBE LEVEL DATA | DNA-DAMAGE RESPONSE | HUMAN-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | ONCOGENE-INDUCED SENESCENCE | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | Epithelial Cells - metabolism | Reactive Oxygen Species | Tissue Array Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Immunoblotting | Gene Expression Profiling | Stem Cells - cytology | Methionine Sulfoxide Reductases - genetics | Stem Cells - metabolism | Zinc Finger E-box-Binding Homeobox 1 - genetics | Tumor Suppressor Protein p53 - genetics | Zinc Finger E-box-Binding Homeobox 1 - metabolism | Breast Neoplasms - metabolism | Young Adult | Cell Differentiation - genetics | Flow Cytometry | Chromatin Immunoprecipitation | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Adult | Female | Epithelial Cells - cytology | Real-Time Polymerase Chain Reaction | Genomic Instability - genetics | Mammary Glands, Human - cytology | Tumor Suppressor Protein p53 - metabolism | Sequence Analysis, DNA | Methionine Sulfoxide Reductases - metabolism | Animals | Breast Neoplasms - genetics | Adolescent | Fluorescent Antibody Technique | Cell Line, Tumor | Carcinoma - genetics | Mice, Inbred NOD | Aged | Carcinoma - metabolism | DNA Damage | Life Sciences | Cancer
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2009, Volume 10, Issue 8, pp. 764 - 771
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 6, pp. 607 - 612
Journal Article
Annales de Pathologie, ISSN 0242-6498, 10/2015, Volume 35, Issue 5, pp. 433 - 437
Journal Article
Annales de Pathologie, ISSN 0242-6498, 10/2015, Volume 35, Issue 5, pp. 408 - 412
Journal Article
Thyroid, ISSN 1050-7256, 11/2017, Volume 27, Issue 11, pp. 1441 - 1449
BACKGROUND:The clinical management of thyroid nodules with indeterminate cytology (IC) remains challenging. The role of shear wave elastography (SWE) in this... 
Life Sciences | Human health and pathology | Endocrinology and metabolism
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 08/2010, Volume 19, Issue 15, pp. 3011 - 3020
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, pp. e0184861 - e0184861
Background Molecular alterations of the MAPK pathway are frequently observed in papillary thyroid carcinomas (PTCs). It leads to a constitutive activation of... 
BIOLOGICAL CHARACTERIZATION | APOPTOSIS | CARCINOMA CELLS | PCCL3 CELLS | KINASE PHOSPHATASES | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC IMPLICATIONS | PAPILLARY | FEEDBACK INHIBITION | EXPRESSION | THYROTROPIN | Up-Regulation | Dual Specificity Phosphatase 6 - genetics | Phosphorylation | Cell Proliferation | Dual-Specificity Phosphatases - metabolism | Neoplasm Invasiveness | Humans | Gene Expression Regulation, Neoplastic | Rats | MAP Kinase Signaling System | Thyroid Cancer, Papillary | Dual Specificity Phosphatase 6 - metabolism | Thyroid Neoplasms - genetics | Feedback, Physiological | Animals | Carcinoma, Papillary | Proto-Oncogene Proteins B-raf - genetics | Dual-Specificity Phosphatases - genetics | Cell Line, Tumor | Carcinoma - genetics | Carcinoma - metabolism | Mutation | Cell Movement | Thyroid Neoplasms - metabolism | Genetic aspects | Phosphatases | Thyroid cancer | Research | Analysis | Mitogen-activated protein kinases | Cell culture | Biotechnology | Regulators | Animal models | Leukocyte migration | Genes | Feedback loops | mRNA | Kinases | Inactivation | Phosphatase | Carcinogenesis | Skin cancer | Proteins | Signal transduction | Carcinogens | Negative feedback | Feedback | Thyroid | Deactivation | Extracellular signal-regulated kinase | MAP kinase | siRNA | Tumor cell lines | Signaling | Doxycycline | Control theory | Cell migration | Endocrinology | Papillary thyroid carcinoma | Cancer | Index Medicus | Life Sciences | Human health and pathology | Endocrinology and metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e103744
Insulin-like growth factor 2 (IGF2) overexpression is an important molecular marker of adrenocortical carcinoma (ACC), which is a rare but devastating... 
INSULIN | MALIGNANCY | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | FACTOR SYSTEM | RECEPTOR | LARGE COHORT | TUMORS | EXPRESSION | TUMORIGENESIS | FACTOR-II GENE | Genomic Imprinting | Cell Proliferation | Prognosis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Male | Genetic Loci | RNA, Messenger - metabolism | Gene Knockdown Techniques | Young Adult | Adrenocortical Carcinoma - pathology | Insulin-Like Growth Factor II - genetics | Aged, 80 and over | G1 Phase Cell Cycle Checkpoints | Adult | Female | Adrenocortical Carcinoma - genetics | RNA, Messenger - genetics | Chromosomes, Human, Pair 11 - genetics | DNA Methylation - genetics | Signal Transduction - genetics | Insulin-Like Growth Factor II - metabolism | Phenotype | Adolescent | Cell Line, Tumor | Aged | Epigenetic inheritance | Platelet-derived growth factor | Carcinoma | Growth | Development and progression | Fibroblast growth factors | Comparative analysis | Cancer | Tyrosine | Adrenal glands | Fibroblast growth factor receptor 9 | Therapeutic applications | Cell division | Insulin-like growth factors | Kinases | Gene expression | Insulin | Loci | Proteins | Uniparental disomy | Pathways | Medical prognosis | Chromosome 11 | Tumorigenesis | Mutation | Insulin-like growth factor II | Growth factors | Protein-tyrosine kinase | Tumors | G1 phase | Apoptosis | Life Sciences | Biochemistry, Molecular Biology | Cattle
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 01/2010, Volume 19, Issue 8, pp. 1561 - 1576
Adrenocortical carcinoma is a rare but aggressive cancer with unknown aetiology. Constitutive activation of beta-catenin is the most frequent alteration in... 
GENE | HYPERALDOSTERONISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | FAMILIAL ADENOMATOUS POLYPOSIS | MICE | CHILDHOOD ADRENOCORTICAL TUMORS | MUTATIONS | CONNS-SYNDROME | CARCINOMA | TUMORIGENESIS | EXPRESSION | Life Sciences | Cancer
Journal Article