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Immunological Reviews, ISSN 0105-2896, 07/2017, Volume 278, Issue 1, pp. 87 - 100
Summary CD69 has been known as an early activation marker of lymphocytes; whereas, recent studies demonstrate that CD69 also has critical functions in immune... 
allergy | CD69 | CD69‐Myl9 system | inflammation | Myl9 | platelets | CD69-Myl9 system | RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | NATURAL-KILLER-CELLS | LYMPHOID ORGANS | BONE-MARROW | CD69-DEFICIENT MICE | ACTIVATION ANTIGEN | IMMUNOLOGY | CHRONIC RHINOSINUSITIS | TISSUE-RESIDENT LYMPHOCYTES | MEMORY T-CELLS | T-Lymphocyte Subsets - immunology | Humans | Lectins, C-Type - genetics | Antigens, CD - genetics | Hypersensitivity - drug therapy | Lectins, C-Type - chemistry | Antigens, CD - metabolism | Lectins, C-Type - metabolism | Inflammation - metabolism | Respiratory Hypersensitivity - drug therapy | Hypersensitivity - metabolism | Hypersensitivity - etiology | Antigens, Differentiation, T-Lymphocyte - chemistry | Antigens, CD - chemistry | Protein Interaction Domains and Motifs | Disease Models, Animal | Disease Susceptibility | Signal Transduction | Respiratory Hypersensitivity - etiology | Gene Expression Regulation | Respiratory Hypersensitivity - metabolism | Antigens, Differentiation, T-Lymphocyte - metabolism | Inflammation - etiology | Animals | T-Lymphocyte Subsets - metabolism | Antigens, Differentiation, T-Lymphocyte - genetics | Protein Binding | Myosin Light Chains - metabolism | Inflammation | Asthma | Animal models | Immune response | Pathogenesis | Hypersensitivity | Antibodies | Lymphocytes T | Inflammatory diseases | Recruitment | Diseases | Respiratory tract | CD69 antigen | Cell activation | Lungs | Lymphocytes | Rodents | Myosin | Respiratory tract diseases | Ligands | Activation analysis | Structure-function relationships
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 19, pp. 7409 - 7414
Memory T-helper (Th) lymphocytes are crucial for the maintenance of acquired immunity to eliminate infectious pathogens. We have previously demonstrated that... 
T lymphocytes | Spleen | Immunization | Immune response | Lymphocytes | Stromal cells | Memory | Antibodies | Plasma cells | Endothelial cells | Homing | T-B interaction | Microenvironment | Plasmablast | Trafficking | AFFINITY MATURATION | CD8-T-CELL MEMORY | CD4-T-CELL HELP | MULTIDISCIPLINARY SCIENCES | MOUSE BONE-MARROW | LYMPHOCYTE EGRESS | microenvironment | HEMATOPOIETIC-CELLS | plasmablast | IN-VIVO | ACTIVATION ANTIGEN CD69 | B-CELL ACTIVATION | homing | trafficking | ACUTE INFECTION | Antigens, CD - immunology | Bone Marrow - immunology | Lectins, C-Type - immunology | Male | Green Fluorescent Proteins - genetics | Adoptive Transfer | Lectins, C-Type - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | CD4-Positive T-Lymphocytes - immunology | Lectins, C-Type - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Bone Marrow - metabolism | Bone Marrow Cells - immunology | Female | Membrane Proteins - metabolism | B-Lymphocytes - metabolism | Immunologic Memory - immunology | Green Fluorescent Proteins - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Membrane Proteins - genetics | Mice, Inbred C57BL | Stromal Cells - metabolism | CD4-Positive T-Lymphocytes - metabolism | Antigens, Differentiation, T-Lymphocyte - metabolism | Membrane Proteins - immunology | Mice, Transgenic | Stromal Cells - immunology | Mice, SCID | Mice, Knockout | Microscopy, Confocal | Animals | B-Lymphocytes - immunology | CD4-Positive T-Lymphocytes - transplantation | Antigens, Differentiation, T-Lymphocyte - genetics | Mice | Mice, Inbred BALB C | Bone Marrow Cells - metabolism | Biological Sciences
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 11/2013, Volume 39, Issue 5, pp. 819 - 832
After antigen encounter by CD4 T cells, polarizing cytokines induce the expression of master regulators that control differentiation. Inactivation of the... 
LINEAGE COMMITMENT | BET | MAINTENANCE | METHYLATION | IFN-GAMMA PRODUCTION | REPRESSION | GENE-EXPRESSION | IMMUNOLOGY | TH1 | TRANSCRIPTION FACTOR | GATA-3 | T-Lymphocyte Subsets - immunology | Sequence Deletion | Polycomb Repressive Complex 2 - genetics | T-Lymphocyte Subsets - cytology | Cells, Cultured - cytology | T-Box Domain Proteins - biosynthesis | Male | Histone-Lysine N-Methyltransferase - deficiency | Th2 Cells - immunology | Th1 Cells - immunology | Lymphokines - genetics | Asthma - immunology | Female | Cell Differentiation | Lymphokines - biosynthesis | Cells, Cultured - immunology | GATA3 Transcription Factor - metabolism | Histone-Lysine N-Methyltransferase - genetics | Interferon-gamma Release Tests | Mice, Inbred C57BL | Gene Expression Regulation | Th2 Cells - cytology | Polycomb Repressive Complex 2 - chemistry | T-Box Domain Proteins - genetics | Asthma - genetics | Enhancer of Zeste Homolog 2 Protein | T-Box Domain Proteins - metabolism | Histone-Lysine N-Methyltransferase - chemistry | Animals | Polycomb Repressive Complex 2 - deficiency | Polycomb Repressive Complex 2 - physiology | Immunologic Memory | Cells, Cultured - metabolism | Mice | Protein Processing, Post-Translational | Th1 Cells - cytology | Histones - metabolism | Methylation | Asthma - pathology | Histone-Lysine N-Methyltransferase - physiology | Proteins | Studies | Medical research | Transcription factors | Cytokines | Lymphocytes | Epigenetics | Software | Genomes | Gene expression | Allergies | Asthma
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 01/2020, Volume 10
Journal Article