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Cement and Concrete Research, ISSN 0008-8846, 03/2012, Volume 42, Issue 3, pp. 539 - 548
The present work offers a general overview about application of titanium dioxide (or titania), TiO , photocatalysis to concrete technology in relation to... 
TiO2 photocatalysis | Particle size | Self-cleaning | Aesthetic durability | Depollution | TiO | photocatalysis | OXIDATION | GASES | DISPERSIONS | RHODAMINE-B | MATERIALS SCIENCE, MULTIDISCIPLINARY | ADSORPTION | LAYERS | CONSTRUCTION & BUILDING TECHNOLOGY | PURIFICATION | DEGRADATION | ELECTRODE | VISIBLE-LIGHT IRRADIATION | Concretes | Photocatalysis | Durability | Cements | Nanostructure | Titanium dioxide | Atmospherics
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 5/2015, Volume 129, Issue 5, pp. 625 - 637
Journal Article
Immunity, ISSN 1074-7613, 07/2015, Volume 43, Issue 1, pp. 92 - 106
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2010, Volume 107, Issue 13, pp. 6058 - 6063
Locus ceruleus (LC)-supplied norepinephrine (NE) suppresses neuroinflammation in the brain. To elucidate the effect of LC degeneration and subsequent NE... 
Brain | Neurons | Antibodies | Norepinephrine | Mice | Amyloids | Alzheimers disease | Phagocytosis | Locus coeruleus | Microglia | Neuroinflammation | Amyloid beta | Neurodegeneration | SENILE DEMENTIA | phagocytosis | COERULEUS | CELLS | PROTEIN | MULTIDISCIPLINARY SCIENCES | neurodegeneration | DOPAMINE-BETA-HYDROXYLASE | amyloid beta | DEGENERATION | neuroinflammation | NEURONS | NORADRENALINE | BRAIN | TRANSGENIC MICE | Inflammation - pathology | Microglia - metabolism | Norepinephrine - pharmacology | Humans | Cell Movement - physiology | Alzheimer Disease - pathology | Brain - metabolism | Inflammation - metabolism | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Microglia - pathology | Female | Transcription, Genetic | Disease Models, Animal | Recombinant Proteins - metabolism | Cell Line | Chemokines - biosynthesis | Phagocytosis - drug effects | Microglia - drug effects | Locus Coeruleus - pathology | Phagocytosis - physiology | Mice, Transgenic | Recombinant Proteins - genetics | Cell Movement - drug effects | Amyloid beta-Protein Precursor - genetics | Animals | Norepinephrine - metabolism | Alzheimer Disease - metabolism | Brain - pathology | Inflammation - genetics | Norepinephrine - deficiency | Alzheimer Disease - genetics | In Vitro Techniques | Cytokines - biosynthesis | Locus Coeruleus - metabolism | Physiological aspects | Development and progression | Research | Alzheimer's disease | Noradrenaline | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 1, pp. 212 - 217
Journal Article
Journal Article
Glia, ISSN 0894-1491, 07/2017, Volume 65, Issue 7, pp. 1176 - 1185
Microglia as principle innate immune cells of the central nervous system (CNS) are the first line of defense against invading pathogens. They are capable of... 
monocytes | inflammation | Rs‐LPS | cytokines | chemokines | TRIF | MyD88 | neutrophils | Rs-LPS | CELLS | ACTIVATION | RECOGNITION | ENDOTOXIN | RHODOBACTER-SPHAEROIDES | NEUROSCIENCES | DIPHOSPHORYL-LIPID-A | RHODOPSEUDOMONAS-SPHAEROIDES | NONTOXIC LIPOPOLYSACCHARIDE | CD14 | INNATE IMMUNITY | Adaptor Proteins, Vesicular Transport - genetics | Myelin Sheath - drug effects | Adaptor Proteins, Vesicular Transport - metabolism | Lipopolysaccharide Receptors - metabolism | Dose-Response Relationship, Drug | Up-Regulation - physiology | Toll-Like Receptor 4 - antagonists & inhibitors | Animals, Newborn | Macrophages - physiology | Brain - cytology | Phagocytosis - drug effects | Cytokines - metabolism | Microglia - drug effects | Myelin Sheath - pathology | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Phagocytosis - physiology | Toll-Like Receptor 4 - genetics | Mice, Transgenic | Toll-Like Receptor 4 - metabolism | Up-Regulation - drug effects | Animals | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Lipopolysaccharide Receptors - genetics | Macrophages - drug effects | Mice | Myeloid Differentiation Factor 88 - metabolism | Photosynthesis | Septic shock | Mitogens | Brain | Central nervous system | CD14 antigen | Lipopolysaccharides | Recruitment | Proteins | Phagocytes | Receptors | Rodents | Toll-like receptors | Bacteria | Immune system | Pathogens | Immune response | Cytokines | TLR4 protein | Microglia | Major histocompatibility complex | Functional morphology | MyD88 protein | Sepsis | Infiltration | Chemokines
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 09/2013, Volume 91, Issue 9, pp. 1133 - 1142
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0161505
Interleukin-1 (IL-1) plays a crucial role in numerous inflammatory diseases via action on its only known signaling IL-1 receptor type 1 (IL-1R1). To... 
SYSTEM | ACTIVATION | IL-1 | MULTIDISCIPLINARY SCIENCES | I RECEPTOR | DEFICIENT | MICE | IL-1-BETA | SECRETION | INDUCTION | AUTOIMMUNE ENCEPHALOMYELITIS | T-Lymphocyte Subsets - immunology | Interleukin-1beta - pharmacology | Cell Proliferation | Cell Survival - genetics | CD4-Positive T-Lymphocytes - immunology | Interleukin-1beta - metabolism | T-Lymphocyte Subsets - drug effects | B-Lymphocytes - metabolism | Receptors, Interleukin-1 Type I - deficiency | Gene Targeting | Interleukin-1 - metabolism | Signal Transduction | Lymphocyte Activation | Antibodies, Monoclonal - pharmacology | CD4-Positive T-Lymphocytes - metabolism | Immunophenotyping | Mice, Transgenic | CD3 Complex - metabolism | Mice, Knockout | Phenotype | Animals | T-Lymphocyte Subsets - metabolism | CD3 Complex - immunology | Receptors, Interleukin-1 Type I - genetics | Biomarkers | Mice | CD4-Positive T-Lymphocytes - drug effects | Cytokines - biosynthesis | Interleukin-1 | Research | Cytokines | T cells | Interleukin | Helper cells | Lymphocytes T | Biology | Interleukin 1 receptors | Kinases | CD28 antigen | Small intestine | Inflammatory diseases | Signal transduction | Lymphocytes | Transgenic animals | Rodents | Interleukin 1 | DNA methylation | Cell survival | Transgenic mice | Cultures | CD3 antigen | Concanavalin A | Survival | CD4 antigen | Medicine | Signaling | Interferon
Journal Article