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by Wu, TS and Lin, WH and Tsai, HJ and Hsueh, CC and Hsu, T and Wang, PC and Lin, HY and Peng, YH and Lu, CT and Lee, LC and Tu, CH and Kung, FC and Shiao, HY and Yeh, TK and Song, JS and Chang, JY and Su, YC and Chen, LT and Chen, CT and Jiaang, WT and Wu, SY
JOURNAL OF MEDICINAL CHEMISTRY, ISSN 0022-2623, 04/2019, Volume 62, Issue 8, pp. 3940 - 3957
Drug resistance due to acquired mutations that constitutively activate c-KIT is a significant challenge in the treatment of patients with gastrointestinal... 
POCKET INHIBITORS | AUTOINHIBITION | CHEMISTRY, MEDICINAL | STRUCTURAL BASIS | IMATINIB | SUNITINIB | DRUG-RESISTANCE | MUTATIONS | GASTROINTESTINAL STROMAL TUMORS | BINDING | RECEPTOR TYROSINE KINASE
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 10/2016, Volume 15, Issue 10, pp. 2323 - 2333
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2005, Volume 102, Issue 4, pp. 1104 - 1109
Journal Article
FRONTIERS IN GENETICS, ISSN 1664-8021, 09/2019, Volume 10
Gastrointestinal stromal tumours (GISTs) are the most common human sarcomas and are typically located in the stomach or small intestine. Although circular RNAs... 
MIGRATION | circRNAs | GIST | CIRCULAR RNAS | PROGNOSIS | CELL INVASION | RISK | PLAT | ETV1 | CANCER | KIT | IMATINIB | GENETICS & HEREDITY | regulatory networks analysis | MUTATIONS
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 36, pp. 13568 - 13573
The nuclear factor E2-related factor 2 (Nrf2) is a master transcriptional activator of genes encoding numerous cytoprotective enzymes that are induced in... 
Tumor cell line | Oxidative stress | Genes | Cell lines | Small interfering RNA | Gene expression regulation | Genetic mutation | Cancer | Tumors | Lung neoplasms | Somatic mutation | Cancer cell microenvironment | Ubiquitin-proteasome system | Multidrug resistant-associated protein | multidrug resistant-associated protein | ACTIVATION | PROTEIN | EFFICACY | UBIQUITIN LIGASE | MULTIDISCIPLINARY SCIENCES | IDENTIFICATION | LUNG-CANCER | ubiquitin-proteasome system | CARCINOGENESIS | POLYMORPHISMS | somatic mutation | cancer cell microenvironment | oxidative stress | GENE-PRODUCT | Neoplasms - metabolism | Protein Structure, Tertiary | Oxidative Stress | Humans | Models, Molecular | Substrate Specificity | Intracellular Signaling Peptides and Proteins - metabolism | Down-Regulation - drug effects | Mutation - genetics | Neoplasms - genetics | Base Sequence | NF-E2-Related Factor 2 - metabolism | Cell Line, Tumor | Cullin Proteins - metabolism | Protein Binding | NF-E2-Related Factor 2 - genetics | Antineoplastic Agents - pharmacology | Active Transport, Cell Nucleus | Gene Expression Regulation, Neoplastic - drug effects | NF-E2-Related Factor 2 - chemistry | Transcription, Genetic - genetics | Kelch-Like ECH-Associated Protein 1 | Genetic aspects | Genetic transcription | Research | Gene mutations | Health aspects | Biological Sciences
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2010, Volume 12, Issue 3, pp. 213 - 223
Impaired selective turnover of p62 by autophagy causes severe liver injury accompanied by the formation of p62-positive inclusions and upregulation of... 
OXIDATIVE STRESS | PROTEIN | MECHANISM | CUL3-BASED E3 LIGASE | STRUCTURAL BASIS | DLG MOTIFS | DEGRADATION | MICE | BETA-CELL MASS | INDUCTION | CELL BIOLOGY | Adaptor Proteins, Signal Transducing - chemistry | Liver - pathology | Microtubule-Associated Proteins - genetics | Cytoskeletal Proteins - genetics | Gene Expression - genetics | Protein Interaction Domains and Motifs - physiology | Sequestosome-1 Protein | Humans | Oxidative Stress - physiology | Crystallography, X-Ray | Hepatocytes - pathology | Autophagy - physiology | Intracellular Signaling Peptides and Proteins - metabolism | Hepatocytes - metabolism | Liver - physiopathology | Heat-Shock Proteins - genetics | Mutation - physiology | Transfection | Organ Size - genetics | Cytoskeletal Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Inclusion Bodies - metabolism | Kelch-Like ECH-Associated Protein 1 | Cell Line | Binding, Competitive - physiology | Heat-Shock Proteins - metabolism | Liver - metabolism | Models, Molecular | Mice, Transgenic | Cytoskeletal Proteins - chemistry | Mice, Knockout | Protein Interaction Mapping | Autophagy-Related Protein 7 | Animals | Models, Biological | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Calorimetry | Signal Transduction - physiology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Heat-Shock Proteins - chemistry | Protein Binding - physiology | Autophagy (Cytology) | Care and treatment | Transcription factors | Liver diseases | Physiological aspects | Genetic aspects | Research
Journal Article
International Journal of Legal Medicine, ISSN 0937-9827, 9/2018, Volume 132, Issue 5, pp. 1287 - 1291
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e17830
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 04/2006, Volume 26, Issue 8, pp. 2887 - 2900
Journal Article