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2017, ISBN 9781771122078, xxi, 215 pages
What is the relationship between literature and the society in which it incubates? Are there common political, social, and economic factors that predominate... 
New Brunswick | Canadian literature | New Brunswick Social conditions | History and criticism | History | Literature and society | Social conditions | Regional Studies | LITERARY CRITICISM | SOCIAL SCIENCE | Canadian | Canadian literature-New Brunswick-History and criticism
Book
2006, APPEL DES MOTS(L'), ISBN 2890067785, 0
Book
12/2019, ISBN 9780228000549, 345
.... In The Fiddlehead Moment, Tony Tremblay challenges this potent stereotype by showcasing the work of a group of literary modernists who set out to change the meaning of New Brunswick in the national lexicon... 
New Brunswick | Modernism (Literature) - New Brunswick | Critiques littéraires | Littérature canadienne - Nouveau-Brunswick - Histoire et critique | Modernisme (Littérature) - Nouveau-Brunswick | Canadian literature - New Brunswick - History and criticism | Language & Literature | Canadian literature | History and criticism | Modernism (Literature)
Book
Journal of Clinical Investigation, ISSN 0021-9738, 12/2011, Volume 121, Issue 12, pp. 4758 - 4774
Journal Article
Journal Article
2001, Poésie, ISBN 9782890066571, 64
Book
2001, Poésie, ISBN 2890066576, 62
Book
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e36703
Single nucleotide polymorphisms in the gene encoding the protein tyrosine phosphatase TCPTP (encoded by PTPN2) have been linked with the development of... 
GROWTH-FACTOR RECEPTOR | TC-PTP | STEM-CELLS | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | MARROW MICROENVIRONMENT | EMBRYONIC LETHALITY | DEFICIENT MICE | PROTEIN-TYROSINE-PHOSPHATASE | EGF RECEPTOR | NEGATIVE REGULATOR | Erythropoiesis | Species Specificity | Hyperplasia | Bone Development | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - genetics | Mice, Knockout | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - metabolism | Animals | Mice | Mice, Inbred BALB C | B-Lymphocytes - pathology | Bone Remodeling | B-Lymphocytes - metabolism | Crosses, Genetic | Tyrosine | Autoimmunity | Phosphatases | Patient outcomes | Mortality | T cells | Density | Morbidity | Bones | Genetic aspects | Single nucleotide polymorphisms | Chromosomes | Disease | Homologous recombination | Biochemistry | Single-nucleotide polymorphism | Bone (trabecular) | Phosphatase | Thymus | Proteins | Bone marrow | Clonal selection | Spleen | Medical research | Myeloid cells | Cytokines | Diarrhea | Bone mass | Stem cells | Splenomegaly | Growth rate | Positive selection | Genes | Homeostasis | Homology | Lymphocytes T | Arthritis | Kinases | Lymph nodes | Defects | Posture | Atrophy | Immunology | Epidermal growth factor | Lymphocytes | Rodents | Cell cycle | Bone remodelling | Cystic fibrosis | Lymphocytes B | Growth hormones | Diabetes | Molecular biology | Protein-tyrosine-phosphatase | PTPN2 protein
Journal Article
1996, Poésie, ISBN 2890464172, 54
Book
Journal of Clinical Investigation, ISSN 0021-9738, 03/2019, Volume 129, Issue 3, pp. 1193 - 1210
Genetic variants at the PTPN2 locus, which encodes the tyrosine phosphatase PTPN2, cause reduced gene expression and are linked to rheumatoid arthritis (RA)... 
PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | GENETICS | INFLAMMATION | TRANSCRIPTION FACTOR FOXP3 | REGULATORY T-CELLS | GENERATION | TNF-ALPHA | DIFFERENTIATION | ARTHRITIS | PROTEIN-TYROSINE-PHOSPHATASE | Forkhead Transcription Factors - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Interleukin-6 - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - immunology | Interleukin-17 - immunology | Interleukin-17 - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - genetics | Forkhead Transcription Factors - genetics | T-Lymphocytes, Regulatory - pathology | Arthritis, Rheumatoid - pathology | Mice, Knockout | T-Lymphocytes, Regulatory - immunology | Arthritis, Rheumatoid - genetics | Animals | Interleukin-6 - immunology | Female | Mice | Mice, Inbred BALB C | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | STAT3 Transcription Factor - genetics | Disease Models, Animal | Autoimmunity | Phosphatases | Rheumatoid arthritis | Genetic aspects | Research | Gene expression | T cells | Haplotypes | Chromatin | Animal models | Transcription factors | Disease | CD8 antigen | Cytokine receptors | Lymphocytes T | Dephosphorylation | Kinases | Phosphatase | Interleukin 6 | Proteins | Lymphocytes | Foxp3 protein | Tyrosine | Phenotypes | Cytokines | Immunoregulation | Stat3 protein | Inflammation | T cell receptors | CD4 antigen | Genetic variance | Autoimmune diseases | Protein-tyrosine-phosphatase | PTPN2 protein
Journal Article
Journal Article
2004, Collection L'appel des mots, ISBN 2890067238, 72
Book
Northeastern Naturalist, ISSN 1092-6194, 2015, Volume 22, Issue 2, pp. 236 - 247
.../na101/home/literatum/publisher/bioone/journals/content/nena/2015/045.022.0200/045.022.0200/20150526/045.022.0200.cover.jpg François Morneau, Junior A. Tremblay... 
NATIONAL-PARK | BIODIVERSITY CONSERVATION | ECOLOGY | Endangered & extinct species | Studies | Habitats | Birds | Population | Polls & surveys | Environmental protection
Journal Article
Journal Article