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1. Full Text Reduced MEK inhibition preserves genomic stability in naive human embryonic stem cells
by Di Stefano, Bruno and Ueda, Mai and Sabri, Shan and Brumbaugh, Justin and Huebner, Aaron J and Sahakyan, Anna and Clement, Kendell and Clowers, Katie J and Erickson, Alison R and Shioda, Keiko and Gygi, Steven P and Gu, Hongcang and Shioda, Toshi and Meissner, Alexander and Takashima, Yasuhiro and Plath, Kathrin and Hochedlinger, Konrad
Nature methods, ISSN 1548-7091, 09/2018, Volume 15, Issue 9, pp. 732 - 740
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Biochemical Research Methods | Science & Technology | Embryonic Stem Cells - metabolism | Genomic Instability | DNA Methylation | Humans | Transcriptome | Embryonic Stem Cells - enzymology | Proteome | Protein Kinase Inhibitors - pharmacology | MAP Kinase Kinase Kinases - antagonists & inhibitors | Cell proliferation | Cellular signal transduction | Chromosome abnormalities | Research | Stem cells | Cell culture | Phosphorylation | Regulators | Stability | Therapeutic applications | Abnormalities | DNA damage | Embryo cells | MAP kinase | Gene expression | Embryos | DNA repair | Genomic instability | X Chromosomes | Cell cycle | Inhibition | Chromosomes | Deoxyribonucleic acid--DNA | Index Medicus
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2. Full Text MicroRNA-33 and the SREBP Host Genes Cooperate to Control Cholesterol Homeostasis
by S. Hani Najafi-Shoushtari and Fjoralba Kristo and Yingxia Li and Toshi Shioda and David E. Cohen and Robert E. Gerszten and Anders M. Näär
Science (American Association for the Advancement of Science), ISSN 0036-8075, 6/2010, Volume 328, Issue 5985, pp. 1566 - 1569
MicroRNA | Genes | HDL lipoproteins | REPORTS | Cell lines | Stem cells | Journalism | Gene expression regulation | Mice | Macrophages | Cholesterols | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Medical sciences | Up-Regulation | Humans | Homeostasis | MicroRNAs - metabolism | Sterol Regulatory Element Binding Proteins - genetics | ATP-Binding Cassette Transporters - genetics | RNA Interference | ATP-Binding Cassette Transporters - metabolism | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | 3' Untranslated Regions | ATP Binding Cassette Transporter 1 | Sterol Regulatory Element Binding Protein 1 - metabolism | Cell Line | Oligonucleotides, Antisense - pharmacology | Introns | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Sterol Regulatory Element Binding Proteins - metabolism | Cholesterol - metabolism | Macrophages - metabolism | Animals | Diet | Sterol Regulatory Element Binding Protein 1 - genetics | Cholesterol, HDL - blood | MicroRNAs - genetics | Physiological aspects | Genetic aspects | Binding proteins | Cholesterol | Biosynthesis | Cellular biology | Ribonucleic acid--RNA | Gene expression | Index Medicus
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3. Full Text RNA-Seq of single prostate CTCs implicates noncanonical Wnt signaling in antiandrogen resistance
by David T. Miyamoto and Yu Zheng and Ben S. Wittner and Richard J. Lee and Huili Zhu and Katherine T. Broderick and Rushil Desai and Douglas B. Fox and Brian W. Brannigan and Julie Trautwein and Kshitij S. Arora and Niyati Desai and Douglas M. Dahl and Lecia V. Sequist and Matthew R. Smith and Ravi Kapur and Chin-Lee Wu and Toshi Shioda and Sridhar Ramaswamy and David T. Ting and Mehmet Toner and Shyamala Maheswaran and Daniel A. Haber
Science (American Association for the Advancement of Science), ISSN 0036-8075, 9/2015, Volume 349, Issue 6254, pp. 1351 - 1356
REPORTS | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Phenylthiohydantoin - therapeutic use | Humans | Transcriptome | Neoplastic Cells, Circulating - metabolism | Male | Wnt-5a Protein | Wnt Proteins - metabolism | Prostate - metabolism | Prostate - pathology | RNA Splicing | Wnt Proteins - genetics | Single-Cell Analysis - methods | Prostate - drug effects | Prostatic Neoplasms - drug therapy | Phenylthiohydantoin - pharmacology | Proto-Oncogene Proteins - metabolism | Prostatic Neoplasms - pathology | Neoplastic Cells, Circulating - drug effects | Signal Transduction | Androgen Antagonists - pharmacology | Proto-Oncogene Proteins - genetics | Phenylthiohydantoin - analogs & derivatives | Sequence Analysis, RNA - methods | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Receptors, Androgen - genetics | Androgen Antagonists - therapeutic use | Cell Line, Tumor | Mice | RNA sequencing | Genetic aspects | Drug resistance | Prostate cancer | Methods | Cancer cells | Metastasis | Ribonucleic acid--RNA | Cancer therapies | Rodents | Index Medicus | Drugs | Heterogeneity | Inhibitors | Pathways | Patients | Prostate | Tumors | Cancer
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4. Full Text Prenatal exposure to bisphenol a disrupts naturally occurring bimodal DNA methylation at proximal promoter of fggy, an obesity-relevant gene encoding a carbohydrate kinase, in gonadal white adipose tissues of CD-1 Mice
by Taylor, Julia A and Shioda, Keiko and Mitsunaga, Shino and Yawata, Shiomi and Angle, Brittany M and Nagel, Susan C and vom Saal, Frederick S and Shioda, Toshi
Endocrinology (Philadelphia), ISSN 0013-7227, 02/2018, Volume 159, Issue 2, pp. 779 - 794
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Phosphotransferases - metabolism | Prenatal Exposure Delayed Effects - enzymology | Humans | Adipose Tissue, White - metabolism | Body Weight - drug effects | Male | Promoter Regions, Genetic - drug effects | Obesity - genetics | Benzhydryl Compounds - adverse effects | Female | Obesity - etiology | Epigenesis, Genetic - drug effects | Phosphotransferases - genetics | Carbohydrate Metabolism | Prenatal Exposure Delayed Effects - etiology | Prenatal Exposure Delayed Effects - metabolism | Obesity - metabolism | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Endocrine Disruptors - adverse effects | Animals | Phenols - adverse effects | Mice | Obesity - enzymology | DNA Methylation - drug effects | Adipose Tissue, White - drug effects | Adipose tissues | Epigenetic inheritance | Medical research | Promoters (Genetics) | Prenatal influences | Bisphenol-A | Medicine, Experimental | Genetic aspects | Research | Health aspects | Gonads | Protein kinases | Endocrine disruptors | Bisulfite | Adipose tissue | Transcription | Toxicity | Body weight | Fuel consumption | Genomes | Males | Kinases | Gene sequencing | DNA methylation | Prenatal experience | Bioinformatics | Deoxyribonucleic acid--DNA | Carbohydrates | Obesity | Phenotypes | Fetuses | Health risks | Exposure | Gene expression | Polymerase chain reaction | Bisphenol A | Epigenetics | Phenols | Females | Index Medicus | Abridged Index Medicus
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5. Full Text Aberrant silencing of imprinted genes on chromosome 12qF1 in mouse induced pluripotent stem cells
by Stadtfeld, Matthias and Apostolou, Effie and Akutsu, Hidenori and Fukuda, Atsushi and Follett, Patricia and Natesan, Sridaran and Kono, Tomohiro and Shioda, Toshi and Hochedlinger, Konrad
Nature (London), ISSN 0028-0836, 05/2010, Volume 465, Issue 7295, pp. 175 - 181
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Cell differentiation, maturation, development, hematopoiesis | Multigene Family - genetics | Cell Line | Embryonic Stem Cells - metabolism | Pluripotent Stem Cells - cytology | Chromosomes, Mammalian - genetics | RNA, Messenger - genetics | Gene Silencing | Intercellular Signaling Peptides and Proteins - genetics | MicroRNAs - metabolism | Gene Expression Profiling | RNA, Messenger - metabolism | Genomic Imprinting - genetics | Pluripotent Stem Cells - metabolism | Proteins - genetics | Animals | Epigenesis, Genetic - genetics | RNA, Long Noncoding | Fibroblasts | Female | Mice | MicroRNAs - genetics | Transcription, Genetic - genetics | Nuclear Proteins - genetics | Physiological aspects | Gene silencing | Transcription factors | Genetic aspects | Research | Stem cells | Proteins | DNA methylation | Gene expression | Index Medicus
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6. Full Text Transcriptomic and Epigenetic Preservation of Genetic Sex Identity in Estrogen-feminized Male Chicken Embryonic Gonads
by Shioda, Keiko and Odajima, Junko and Kobayashi, Misato and Kobayashi, Mutsumi and Cordazzo, Bianca and Isselbacher, Kurt J and Shioda, Toshi
Endocrinology (Philadelphia), ISSN 0013-7227, 01/2021, Volume 162, Issue 1
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Index Medicus | Abridged Index Medicus
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7. Full Text A microfluidic device for label-free, physical capture of circulating tumor cell clusters
by Sarioglu, A. Fatih and Aceto, Nicola and Kojic, Nikola and Donaldson, Maria C and Zeinali, Mahnaz and Hamza, Bashar and Engstrom, Amanda and Zhu, Huili and Sundaresan, Tilak K and Miyamoto, David T and Luo, Xi and Bardia, Aditya and Wittner, Ben S and Ramaswamy, Sridhar and Shioda, Toshi and Ting, David T and Stott, Shannon L and Kapur, Ravi and Maheswaran, Shyamala and Haber, Daniel A and Toner, Mehmet
Nature methods, ISSN 1548-7091, 06/2015, Volume 12, Issue 7, pp. 685 - 691
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Biochemical Research Methods | Science & Technology | Immunohistochemistry | Microfluidic Analytical Techniques | Prostatic Neoplasms - pathology | Sequence Analysis, RNA | Breast Neoplasms - pathology | Humans | Neoplastic Cells, Circulating | Cell Line, Tumor | Female | Male | Technology application | Care and treatment | Analysis | Cancer cells | Health aspects | Microfluidics | Tumors | Medical prognosis | Metastasis | Cancer | Index Medicus
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8. Full Text Cell type of origin influences the molecular and functional properties of mouse induced pluripotent stem cells
by Wagers, Amy J and Eminli, Sarah and Evans, Todd and Liu, Susanna and Kulalert, Warakorn and Li, Yushan and Apostolou, Effie and Tan, Kah Yong and Natesan, Sridaran and Melnick, Ari and Polo, Jose M and Shioda, Toshi and Hochedlinger, Konrad and Stadtfeld, Matthias and Figueroa, Maria Eugenia
Nature biotechnology, ISSN 1087-0156, 08/2010, Volume 28, Issue 8, pp. 848 - 855
Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Biotechnology | Biological and medical sciences | Miscellaneous | Health. Pharmaceutical industry | Industrial applications and implications. Economical aspects | B-Lymphocytes - cytology | Epigenomics | Cells, Cultured | Gene Expression Profiling | Hematopoietic Stem Cells - metabolism | Stem Cells - cytology | Muscle, Skeletal - cytology | Embryoid Bodies - metabolism | Cell Lineage | Animals | Embryoid Bodies - cytology | Hematopoietic Stem Cells - cytology | Transcription, Genetic | Cell Differentiation | Fibroblasts - cytology | Mice | Induced Pluripotent Stem Cells - cytology | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Molecular biology | Gene expression | Rodents | Stem cells | Index Medicus
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9. Full Text Ancestral perinatal obesogen exposure results in a transgenerational thrifty phenotype in mice
by Chamorro-Garcia, Raquel and Diaz-Castillo, Carlos and Shoucri, Bassem M and Käch, Heidi and Leavitt, Ron and Shioda, Toshi and Blumberg, Bruce
Nature communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2012 - 13
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lactation | Mice, Inbred C57BL | Transcriptome | Male | Obesity - genetics | Trialkyltin Compounds - toxicity | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Adipose Tissue - metabolism | DNA Methylation | Phenotype | Animals | Inheritance Patterns - genetics | Female | Prenatal Exposure Delayed Effects - chemically induced | Obesity | Chromatin | Perinatal exposure | Adipose tissue | Body fat | Mitosis | Exposure | Heredity | Meiosis | Metabolism | Gene expression | Diet | Etiology | Leptin | DNA methylation | Mice | Tributyltin | Deoxyribonucleic acid--DNA | Antifouling substances | Index Medicus
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