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Journal of Cellular Physiology, ISSN 0021-9541, 11/2018, Volume 233, Issue 11, pp. 8467 - 8476
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2018, Volume 233, Issue 11, pp. 8467 - 8476
A successful aging could be gained by life satisfaction, social functioning, or psychological resources and, definitely, by increasing resistance to diverse... 
IGF‐1 | mTOR | AMPK | cellular senescence | cellular metabolism | IGF-1 | CARCINOMA CELL-LINES | PHYSIOLOGY | TGF-BETA | EXTENDS LIFE-SPAN | RECEPTOR | CELL BIOLOGY | SREBP ACTIVITY | PREMATURE SENESCENCE | COLORECTAL-CANCER | IN-VIVO | GROWTH | AGE-RELATED DISEASES | Oncology, Experimental | Research | Cancer | Pathways | Aging | Adults | Longevity | Age | Quality of life
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 10/2013, Volume 228, Issue 10, pp. 2054 - 2062
Well-differentiated papillary and follicular thyroid carcinoma are the most frequent types of thyroid cancer and the prognosis is generally favorable however,... 
CADHERIN | BREAST-CANCER CELLS | APOPTOSIS | PHYSIOLOGY | GREEN TEA POLYPHENOLS | PROSTATE-CANCER | DIETARY PHYTOCHEMICALS | GENE-EXPRESSION | RECEPTOR | CHEMOPREVENTION | TRANSCRIPTIONAL REGULATION | CELL BIOLOGY | Cyclin D1 - metabolism | Cadherins - metabolism | Apoptosis - drug effects | Vimentin - metabolism | Homeodomain Proteins - metabolism | Humans | Actins - metabolism | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Cell Movement - genetics | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | Actins - genetics | Matrix Metalloproteinase 9 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | MAP Kinase Signaling System - genetics | Matrix Metalloproteinase 9 - genetics | Vimentin - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Cadherins - genetics | Catechin - pharmacology | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Adhesion - genetics | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Cell Adhesion - drug effects | Down-Regulation - drug effects | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Up-Regulation - drug effects | Cell Movement - drug effects | Thyroid Neoplasms - genetics | MAP Kinase Signaling System - drug effects | Cyclin D1 - genetics | Thyroid Neoplasms - drug therapy | Integrin alpha5 - metabolism | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Catechin - analogs & derivatives | Cell Proliferation - drug effects | Integrin alpha5 - genetics | Twist-Related Protein 1 - metabolism | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Zinc Finger E-box-Binding Homeobox 1 | Catechin | Thyroid cancer | Carcinoma | Growth | Stem cells | Thyroid gland | Muscle proteins | Tumor proteins | Intermediate filament proteins | Green tea | Integrins | Cancer
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 11/2017, Volume 24, Issue 11, pp. 1948 - 1962
High Mobility Group A1 (HMGA1) is an architectural chromatin protein whose overexpression is a feature of malignant neoplasias with a causal role in cancer... 
TRANSFORMATION | HMGA PROTEINS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESISTANCE | REQUIRES | DEATH | TRANSCRIPTIONAL REGULATION | EXPRESSION | MTOR | TRANSGENIC MICE | CELL BIOLOGY | Neoplasms - metabolism | Skin Neoplasms - pathology | Green Fluorescent Proteins - metabolism | Cell Proliferation | Cell Survival | Microtubule-Associated Proteins - metabolism | Humans | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | Autophagy-Related Protein-1 Homolog - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | HMGA1a Protein - metabolism | Autophagy | Gene Knockdown Techniques | Skin Neoplasms - metabolism | Animals | Autophagy-Related Protein 5 - metabolism | Transcription, Genetic | Mice | HeLa Cells | Neoplasms - pathology | Autophagy-Related Protein-1 Homolog - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | TOR protein | Cell proliferation | Chromatin | Deregulation | Transcription | Mobility | Phagosomes | A1 protein | Kinases | DNA repair | Skin cancer | Proteins | Cell growth | Protein folding | Deoxyribonucleic acid--DNA | Cell survival | Stability | Organelles | Survival | High mobility group proteins | Depletion | Cell death | Tumor suppressor genes | Viability | Phagocytosis | Cancer | Apoptosis | Original Paper
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e80728
UVB radiation causes about 90% of non-melanoma skin cancers by damaging DNA either directly or indirectly by increasing levels of reactive oxygen species... 
ACTIVATION | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | DEATH | CHEMOPREVENTION | MECHANISMS | KAPPA-B | ULTRAVIOLET-RADIATION | CANCER | HUMAN KERATINOCYTES | ERK | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - genetics | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Caspase 8 - metabolism | Phagosomes - radiation effects | Stilbenes - pharmacology | Autophagy - drug effects | Ultraviolet Rays | Proteolysis | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Oxidative Stress - radiation effects | Phagosomes - drug effects | Beclin-1 | Membrane Proteins - genetics | Phagosomes - metabolism | Hydrogen Peroxide - pharmacology | Apoptosis Regulatory Proteins - metabolism | Poly(ADP-ribose) Polymerases - metabolism | Signal Transduction - drug effects | Cell Proliferation - drug effects | Oxidative Stress - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Line, Transformed | Cell Proliferation - radiation effects | Antioxidants | Radiation | Resveratrol | Genetic aspects | Cellular signal transduction | Skin | Cancer | Apoptosis | Cell proliferation | Oxidative stress | Biotechnology | Transformation | Phosphorylation | DNA damage | Colorectal cancer | Inflammatory response | Kinases | Autophagy | Skin cancer | Proteins | Cell cycle | Skin diseases | Pretreatment | Deoxyribonucleic acid--DNA | U.V. radiation | Melanoma | Keratinocytes | Caspase | Poly(ADP-ribose) polymerase | Inflammation | Gene expression | Medicine | Caspase-8 | Phytochemicals | Cell death | Irradiation | Transduction | Viability | Radiation damage | Endocrinology | Phagocytosis | Deoxyribonucleic acid | DNA
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 4843 - 10
Natural selection acts on genetic variants by increasing the frequency of alleles responsible for a cellular function that is favorable in a certain... 
GENOME SEQUENCE | MULTIDISCIPLINARY SCIENCES | BARRIER FUNCTION | NEANDERTHAL | DIVERSITY | SKIN | IDENTIFICATION | ADMIXTURE | EXPRESSION | ANCESTRY | PIGMENTATION | Genetic variance | Nucleotide sequence | Gene frequency | Transcription | U.V. radiation | Exons | Positive selection | Alleles | Genomes | Gene expression | Natural selection
Journal Article
Journal Article
by Castelnuovo, Gianluca and Giusti, Emanuele M and Manzoni, Gian Mauro and Saviola, Donatella and Gatti, Arianna and Gabrielli, Samantha and Lacerenza, Marco and Pietrabissa, Giada and Cattivelli, Roberto and Spatola, Chiara A.M and Corti, Stefania and Novelli, Margherita and Villa, Valentina and Cottini, Andrea and Lai, Carlo and Pagnini, Francesco and Castelli, Lorys and Tavola, Mario and Torta, Riccardo and Arreghini, Marco and Zanini, Loredana and Brunani, Amelia and Capodaglio, Paolo and D'Aniello, Guido E and Scarpina, Federica and Brioschi, Andrea and Priano, Lorenzo and Mauro, Alessandro and Riva, Giuseppe and Repetto, Claudia and Regalia, Camillo and Molinari, Enrico and Notaro, Paolo and Paolucci, Stefano and Sandrini, Giorgio and Simpson, Susan G and Wiederhold, Brenda and Tamburin, Stefano and Agostini, Michela and Alfonsi, Enrico and Aloisi, Anna Maria and Alvisi, Elena and Aprile, Irene and Armando, Michela and Avenali, Micol and Azicnuda, Eva and Barale, Francesco and Bartolo, Michelangelo and Bergamaschi, Roberto and Berlangieri, Mariangela and Berlincioni, Vanna and Berliocchi, Laura and Berra, Eliana and Berto, Giulia and Bonadiman, Silvia and Bonazza, Sara and Bressi, Federica and Brugnera, Annalisa and Brunelli, Stefano and Buzzi, Maria Gabriella and Cacciatori, Carlo and Calvo, Andrea and Cantarella, Cristina and Caraceni, Augusto and Carone, Roberto and Carraro, Elena and Casale, Roberto and Castellazzi, Paola and Castino, Adele and Cerbo, Rosanna and Chiò, Adriano and Ciotti, Cristina and Cisari, Carlo and Coraci, Daniele and Toffola, Elena Dalla and Defazio, Giovanni and De Icco, Roberto and Del Carro, Ubaldo and Dell'Isola, Andrea and De Tanti, Antonio and D'Ippolito, Mariagrazia and Fazzi, Elisa and Ferrari, Adriano and Ferrari, Sergio and Ferraro, Francesco and Formaglio, Fabio and Formisano, Rita and Franzoni, Simone and Gajofatto, Francesca and Gandolfi, Marialuisa and Gardella, Barbara and Geppetti, Pierangelo and Giammò, Alessandro and Gimigliano, Raffaele and Greco, Elena and Ieraci, Valentina and Invernizzi, Marco and Jacopetti, Marco and La Cesa, Silvia and Lobba, Davide and ... and Italian Consensus Conference Pain
Frontiers in Psychology, ISSN 1664-1078, 2016, Volume 7, p. 115
Journal Article