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SCIENTIFIC REPORTS, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 19939 - 14
... structure-specific ceramide alterations in the zebrafish tejia Zhang1, Sunia A. trauger2, charles Vidoudez2, Kim P. Doane1, Brock R. pluimer1 and Randall T. peterson1... 
MUTATION ANALYSIS | GENE | QUANTIFICATION | MULTIDISCIPLINARY SCIENCES | FARBER-DISEASE | FATTY-ACIDS | TANDEM MASS-SPECTROMETRY | IDENTIFICATION | CHROMATOGRAPHY | CHAIN-LENGTH | CELL | CRISPR | Embryogenesis | Developmental stages | Ceramide | Mass spectroscopy | Zebrafish | Liquid chromatography | Genomes | Proteomes | Structure-function relationships | Index Medicus
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2017, Volume 357, Issue 6356, pp. 1156 - 1160
Journal Article
Nature medicine, ISSN 1546-170X, 2014, Volume 20, Issue 10, pp. 1147 - 1156
Astrocytes have complex roles in health and disease, thus it is important to study the pathways that regulate their function. Here we report that... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISTINCT | MONOCYTES | INFILTRATION | CELL BIOLOGY | ADHESION | LACTOSYLCERAMIDE SYNTHASE | REACTIVE ASTROCYTES | MICE | EXPRESSION | T-CELLS | Up-Regulation | Central Nervous System - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Central Nervous System - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Nerve Degeneration - genetics | Galactosyltransferases - genetics | Central Nervous System - immunology | Nerve Degeneration - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | Glial Fibrillary Acidic Protein | Astrocytes - immunology | Female | Encephalomyelitis, Autoimmune, Experimental - genetics | Multiple Sclerosis - metabolism | Mice, Inbred C57BL | Lactosylceramides - metabolism | Chemokine CCL2 - genetics | Mice, Transgenic | Multiple Sclerosis - genetics | Nerve Degeneration - immunology | Galactosyltransferases - metabolism | Glycolipids - metabolism | Immunity, Innate | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Multiple Sclerosis - immunology | Mice, Inbred NOD | Mice | Astrocytes - metabolism | Inflammation | Research | Astrocytes | Analysis | Central nervous system | Enzymes | Nervous system | Cellular biology | Gene expression | Inflammatory diseases
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 4, pp. 1500 - 1505
Advances in human genetics are leading to the discovery of new disease-causing mutations at a remarkable rate. Many such mutations, however, occur in genes... 
Brain | Enzymes | Nervous system diseases | Disease models | Neurodegenerative diseases | Lipids | Mice | Lipid metabolism | Genotypes | Microglia | TOLL-LIKE-RECEPTOR-2 | neuroinflammation | MICROGLIA | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MAST-CELLS | PHENOTYPE | CHEMOTACTIC MIGRATION | MICE | GENERATION | RECEPTORS | lipidomics | Lysophospholipids - metabolism | Microglia - metabolism | Humans | Cataract - pathology | Monoacylglycerol Lipases - deficiency | Polyneuropathies - metabolism | Monoacylglycerol Lipases - genetics | Brain - metabolism | Monoacylglycerol Lipases - metabolism | Ataxia - genetics | Ataxia - metabolism | Retinitis Pigmentosa - physiopathology | Ataxia - pathology | Disease Models, Animal | Ataxia - physiopathology | Mice, Inbred C57BL | Retinitis Pigmentosa - genetics | Retinitis Pigmentosa - metabolism | Behavior, Animal - physiology | Lipid Metabolism | Polyneuropathies - physiopathology | Cataract - metabolism | Mice, Knockout | Phenotype | Animals | Metabolic Networks and Pathways | Polyneuropathies - pathology | Brain - pathology | Polyneuropathies - genetics | Cataract - genetics | Models, Neurological | Mutation | Cataract - physiopathology | Retinitis Pigmentosa - pathology | Gene mutations | Serine | Genomics | Physiological aspects | Nervous system | Development and progression | Genetic aspects | Degeneration | Research | Health aspects | Biological Sciences
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2016, Volume 126, Issue 11, pp. 4372 - 4386
Obese, insulin-resistant states are characterized by a paradoxical pathogenic condition in which the liver appears to be selectively insulin resistant.... 
RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | TRANSCRIPTION FACTOR FOXO1 | FATTY LIVER-DISEASE | OB/OB MICE | ELEMENT-BINDING PROTEIN | HEPATIC STEATOSIS | ADIPOSE-TISSUE | PLASMA TRIGLYCERIDES | Glucose Intolerance - metabolism | Glucose-6-Phosphatase - genetics | Fatty Liver - pathology | Humans | Male | Glucose - biosynthesis | Glucose Intolerance - pathology | Fatty Liver - chemically induced | Glycolysis - drug effects | Glycolysis - genetics | Lipogenesis - genetics | Female | Glucose Intolerance - chemically induced | Insulin - genetics | Nuclear Proteins - genetics | Fatty Liver - genetics | Forkhead Box Protein O1 - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Fructose - toxicity | Insulin Resistance | Glucose - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Glucose-6-Phosphatase - metabolism | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Forkhead Box Protein O1 - genetics | Obesity | Analysis | Insulin resistance | Genetic aspects | Genetic transcription | Research | Risk factors | Protein binding | Glucose | Metabolites | Rodents | Liver
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 11/2016, Volume 12, Issue 11, pp. 900 - 901
... Janetzko1,2, Sunia A Trauger3, Michael B Lazarus1,2,4 & Suzanne Walker2* 201Nature America, Inc. All rights reserved. The essential human enzyme O-linked b-N... 
METASTABLE BINDING-SITES | PROTEIN | MODELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HCF-1 | O-GLCNAC TRANSFERASE | FAMILY | Hydrolysis | Biocatalysis | Humans | Amides - chemistry | Amides - metabolism | Host Cell Factor C1 - chemistry | Host Cell Factor C1 - metabolism | N-Acetylglucosaminyltransferases - metabolism | Enzymes | Carbohydrates | Glycosylation | Catalysis | Proteases
Journal Article
Biophysical Journal, ISSN 0006-3495, 02/2018, Volume 114, Issue 3, pp. 579a - 579a
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 12, pp. e0207940 - e0207940
Journal Article
ISSN 2041-6520, 2/2019, Volume 1, Issue 7, pp. 225 - 233
Some ruthenium-hydride complexes react with O 2 to yield H 2 O 2 , therefore the principle of microscopic reversibility dictates that the reverse reaction is... 
Journal Article
Journal Article