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Journal Article
2004, International reviews of immunology, Volume 23, no. 3/4., 221-365
Book
2000, Current molecular medicine, ISBN 9780896037731, xiii, 542
Book
2004, Current directions in autoimmunity, ISBN 9783805576468, Volume 7, vi, 205
Complement protein expression and activation control central aspects of the normal B cell response. Imbalances in these processes invariably lead to autoimmune... 
Autoimmunity | Complement activation
Book
2007, 1st ed., ISBN 9780323044349, xv, 586
This new companion to Hochberg et al.'s Rheumatology masterwork provides new insights into the causes, detection and therapy of this challenging disease. In... 
Lupus Erythematosus, Systemic
Book
2002, ISBN 0896039161, xv, 655
Book
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 04/2019, Volume 143, Issue 4, pp. 1351 - 1352
Additional sophisticated molecular studies have identified distinct checkpoints in B-cell development in which tolerance is broken and the expansion of... 
Autoantibodies | autoimmunity | lupus | checkpoint control | ALLERGY | IMMUNOLOGY | B-CELL TOLERANCE | Immunoglobulin M | Lupus | Plasma | Antigens | Immunoglobulins | Disease | Immunoglobulin G | Arthritis | Patients | Immunological tolerance | Human subjects | Chronic conditions | Systemic lupus erythematosus | Lymphocytes B | Rheumatoid arthritis | Plasma cells | Mice | Autoimmune diseases
Journal Article
Nature Immunology, ISSN 1529-2908, 08/2017, Volume 18, Issue 9, pp. 955 - 956
Poor glycolysis and increased fatty-acid synthesis feed the locomotion machinery in T cells from people with rheumatoid arthritis and allow these cells to... 
RHEUMATOID-ARTHRITIS | IMMUNOLOGY | Autoimmunity | Rheumatoid arthritis | Physiological aspects | Genetic aspects | Research | B cells | T cells | Joints | Risk factors | Lymphocytes | Lymphocytes T
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2009, Volume 183, Issue 7, pp. 4675 - 4681
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 11/2019, Volume 71, Issue 11, pp. 1869 - 1878
Objective Glutaminase 1 (Gls1) is the first enzyme in glutaminolysis. The selective Gls1 inhibitor bis‐2‐(5‐phenylacetamido‐1,3,4‐thiadiazol‐2‐yl)ethyl sulfide... 
Sulfide | Lupus | Encephalomyelitis | Helper cells | Medical services | Lymphocytes T | Disease control | Experimental allergic encephalomyelitis | Glutaminase | Western blotting | CD4 antigen | Proteins | Chronic conditions | Systemic lupus erythematosus | Lymphocytes | Rodents | Glycolysis | Hypoxia | Inhibition | Autoimmune diseases | Differentiation
Journal Article
Nature Reviews Rheumatology, ISSN 1759-4790, 02/2016, Volume 12, Issue 2, p. 74
Journal Article
Current Opinion in Rheumatology, ISSN 1040-8711, 03/2019, Volume 31, Issue 2, pp. 167 - 174
PURPOSE OF REVIEWLupus nephritis (LN) is a serious manifestation of systemic lupus erythematosus and is characterized by proteinuria and renal failure.... 
ECULIZUMAB | ANTIBODIES | COMPLEMENT | ACTIVATION | RECEPTOR | INDUCTION | RHEUMATOLOGY | RESISTANT | systemic lupus erythematosus | LUPUS NEPHRITIS | podocyte | KIDNEY | EXPRESSION | Development and progression | Nephritis | Research | Epithelial cells
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 09/2019
Systemic lupus erythematosus (SLE) is a devastating autoimmune disease, in which hyperactive T cells play a critical role. Understanding molecular mechanisms... 
Journal Article
Arthritis Research & Therapy, ISSN 1478-6354, 2014, Volume 16, Issue Suppl 1, pp. A30 - A30
Journal Article
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