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1. Full Text Infrequent SMARCB1/INI1 gene alteration in epithelioid sarcoma: a useful tool in distinguishing epithelioid sarcoma from malignant rhabdoid tumor
by Kohashi, Kenichi, MD, PhD and Izumi, Teiyu, MD, PhD and Oda, Yoshinao, MD, PhD and Yamamoto, Hidetaka, MD, PhD and Tamiya, Sadafumi, MD, PhD and Taguchi, Tomoaki, MD, PhD and Iwamoto, Yukihide, MD, PhD and Hasegawa, Tadashi, MD, PhD and Tsuneyoshi, Masazumi, MD, PhD
Human Pathology, ISSN 0046-8177, 2009, Volume 40, Issue 3, pp. 349 - 355
Summary Loss of SMARCB1/INI1 protein expression is considered useful for confirming a histologic diagnosis of malignant rhabdoid tumor. However, loss of... 
Pathology | Malignant rhabdoid tumor | Immunohistochemistry | Epithelioid sarcoma | SMARCB1/INI1 | BLADDER-CANCER | SOFT-TISSUE SARCOMAS | TIME QUANTITATIVE PCR | PATHOLOGY | IMMUNOHISTOCHEMICAL ANALYSIS | CENTRAL-NERVOUS-SYSTEM | CHECKPOINT | HSNF5/INI1 | MUTATIONS | ATYPICAL TERATOID/RHABDOID TUMORS | EXPRESSION | Soft Tissue Neoplasms - diagnosis | Humans | Male | Immunoenzyme Techniques | DNA-Binding Proteins - metabolism | SMARCB1 Protein | Young Adult | DNA Mutational Analysis | Sarcoma - mortality | Biomarkers, Tumor - metabolism | Female | DNA, Neoplasm - analysis | Sarcoma - diagnosis | Sarcoma - genetics | Soft Tissue Neoplasms - genetics | Diagnosis, Differential | Soft Tissue Neoplasms - mortality | Chromosomal Proteins, Non-Histone - metabolism | Kaplan-Meier Estimate | Survival Rate | Gene Dosage | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Chromosomal Proteins, Non-Histone - genetics | Transcription Factors - metabolism | Rhabdoid Tumor - genetics | Biomarkers, Tumor - genetics | Rhabdoid Tumor - diagnosis | Mutation | Genetic research | Universities and colleges | Sarcoma | DNA | Proteins | Polymerase chain reaction | Survival analysis | Genes | Cell cycle | Epigenetics | Genetics
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2. Full Text Extrarenal rhabdoid tumors of soft tissue: Clinicopathological and molecular genetic review and distinction from other soft‐tissue sarcomas with rhabdoid features
by Oda, Yoshinao and Tsuneyoshi, Masazumi
Pathology International, ISSN 1320-5463, 06/2006, Volume 56, Issue 6, pp. 287 - 295
Malignant rhabdoid tumor (MRT) of the soft tissue is a rare and highly aggressive tumor that occurs in infancy or childhood. It predominantly involves a deep... 
immunohistochemistry | proximal‐type epithelioid sarcoma | extrarenal rhabdoid tumor | hSNF5/INI1/SMARCB1 | soft tissue tumor | Immunohistochemistry | Soft tissue tumor | Extrarenal rhabdoid tumor | Proximal-type epithelioid sarcoma | proximal-type epithelioid sarcoma | SYNOVIAL SARCOMA | PROGNOSTIC-FACTORS | EXTRASKELETAL MYXOID CHONDROSARCOMA | PATHOLOGY | RENAL-CELL CARCINOMA | IMMUNOHISTOCHEMICAL ANALYSIS | MALIGNANT FIBROUS HISTIOCYTOMA | EPITHELIOID SARCOMA | POLYMERASE CHAIN-REACTION | CENTRAL-NERVOUS-SYSTEM | ATYPICAL TERATOID/RHABDOID TUMORS | Diagnosis, Differential | Molecular Biology | Biomarkers, Tumor - analysis | Humans | Rhabdoid Tumor - chemistry | Chromosomal Proteins, Non-Histone | Child, Preschool | DNA-Binding Proteins - analysis | Infant | Rhabdoid Tumor - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sarcoma - pathology | SMARCB1 Protein | Sarcoma - chemistry | Rhabdoid Tumor - genetics | Soft Tissue Neoplasms - chemistry | Soft Tissue Neoplasms - pathology | Cytogenetics | Transcription Factors - analysis | Mutation | Sarcoma - genetics | Child | Soft Tissue Neoplasms - genetics | Sarcoma | Genetic research
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3. Full Text Apparent Diffusion Coefficients of Breast Tumors: Clinical Application
by HATAKENAKA, Masamitsu and SOEDA, Hiroyasu and YABUUCHI, Hidetake and MATSUO, Yoshio and KAMITANI, Takeshi and ODA, Yoshinao and TSUNEYOSHI, Masazumi and HONDA, Hiroshi
Magnetic Resonance in Medical Sciences, ISSN 1347-3182, 2008, Volume 7, Issue 1, pp. 23 - 29
Purpose: To evaluate the usefulness of apparent diffusion coefficient (ADC) for the differential diagnosis of breast tumors and to determine the relation... 
apparent diffusion coefficient | neoplasm | MR imaging | breast | cellularity | Neoplasm | Breast | Cellularity | Apparent diffusion coefficient | Breast Neoplasms - surgery | Fibroadenoma - pathology | Adenocarcinoma, Mucinous - pathology | Breast - surgery | Humans | Middle Aged | Adenocarcinoma, Mucinous - surgery | Aged, 80 and over | Adult | Carcinoma, Ductal, Breast - pathology | Female | Fibroadenoma - diagnosis | Diffusion | Breast - pathology | Carcinoma, Ductal, Breast - surgery | Diagnosis, Differential | Fibroadenoma - surgery | Echo-Planar Imaging - methods | Image Processing, Computer-Assisted - methods | Carcinoma, Ductal, Breast - diagnosis | Adenocarcinoma, Mucinous - diagnosis | Breast Neoplasms - pathology | Adolescent | Aged | Breast Neoplasms - diagnosis | Diffusion Magnetic Resonance Imaging - methods
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4. Full Text N- myc downstream regulated gene-1/Cap43 may play an important role in malignant progression of prostate cancer, in its close association with E-cadherin
by Song, YooHyun, MD and Oda, Yoshinao, MD, PhD and Hori, Mikifumi, MD, PhD and Kuroiwa, Kentaro, MD, PhD and Ono, Mayumi, PhD and Hosoi, Fumihito, MS and Basaki, Yuji, PhD and Tokunaga, Shoji, PhD and Kuwano, Michihiko, MD, PhD and Naito, Seiji, MD, PhD and Tsuneyoshi, Masazumi, MD, PhD
Human Pathology, ISSN 0046-8177, 2010, Volume 41, Issue 2, pp. 214 - 222
Summary N- myc downstream regulated gene-1 (NDRG1)/Cap43 plays an important role in tumor progression and metastases in many kinds of cancers. Recently, it was... 
Pathology | NDRG1/Cap43 | Prognosis | Prostate | Carcinogenesis | E-cadherin | CELLS | PANCREATIC-CANCER | RADICAL PROSTATECTOMY | PATHOLOGY | NDRG1 | BREAST-CANCER | POOR-PROGNOSIS | TUMOR-METASTASIS | DIFFERENTIATION | EXPRESSION | METASTASIS SUPPRESSOR GENE | Immunohistochemistry | Prostatic Neoplasms - metabolism | Cadherins - metabolism | Humans | Cell Cycle Proteins - metabolism | Kaplan-Meier Estimate | Proportional Hazards Models | Male | Intracellular Signaling Peptides and Proteins - metabolism | Prostatic Neoplasms - diagnosis | Chi-Square Distribution | Disease Progression | Disease-Free Survival | Aged | Cell research | Analysis | Oncology, Experimental | Genetic research | Development and progression | Genetic aspects | Research | Universities and colleges | Prostate cancer | Cancer | Medical informatics | Proteins | Medical research | Mortality
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5. Full Text Reduced expression of SMARCB1 INI1 protein in synovial sarcoma
by Kohashi, Kenichi and Oda, Yoshinao and Yamamoto, Hidetaka and Tamiya, Sadafumi and Matono, Hiroshi and Iwamoto, Yukihide and Taguchi, Tomoaki and Tsuneyoshi, Masazumi
Modern Pathology, ISSN 0893-3952, 07/2010, Volume 23, Issue 7, pp. 981 - 990
Synovial sarcoma is classified as a tumor of uncertain differentiation, and some synovial sarcomas have rhabdoid cells. In previous studies, all malignant... 
chromatin remodeling | synovial sarcoma | malignant rhabdoid tumor | SS18-SSX | SMARCB1/INI1 | AGGRESSIVE-BEHAVIOR | SOFT-TISSUE SARCOMAS | FUSION GENE | PATHOLOGY | MALIGNANT RHABDOID TUMORS | SYT | FEATURES | INI1 EXPRESSION | INTERACTS | DISTINCTION | EPITHELIOID SARCOMA | Immunohistochemistry | Biomarkers, Tumor - analysis | Humans | Soft Tissue Neoplasms - metabolism | RNA, Messenger - analysis | Sarcoma, Synovial - metabolism | Sarcoma, Synovial - mortality | Transcription Factors - biosynthesis | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | SMARCB1 Protein | Chromosomal Proteins, Non-Histone - biosynthesis | Soft Tissue Neoplasms - pathology | Survival Analysis | Sarcoma, Synovial - pathology | DNA-Binding Proteins - biosynthesis
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6. Full Text Recent advances in the molecular pathology of soft tissue sarcoma: Implications for diagnosis, patient prognosis, and molecular target therapy in the future
by Oda, Yoshinao and Tsuneyoshi, Masazumi
Cancer Science, ISSN 1347-9032, 02/2009, Volume 100, Issue 2, pp. 200 - 208
In the present paper, recent advances in the molecular pathology of soft tissue sarcomas (STS) and the implications for their prognostic value are reviewed,... 
GROWTH-FACTOR RECEPTOR | FUSION TRANSCRIPT STRUCTURE | HUMAN SYNOVIAL SARCOMA | MALIGNANT RHABDOID TUMOR | ONCOLOGY | MESSENGER-RNA EXPRESSION | BOX-BINDING PROTEIN-1 | SPORADIC DESMOID TUMORS | SUPPRESSOR GENE-MUTATIONS | NERVE SHEATH TUMORS | MYXOID/ROUND-CELL LIPOSARCOMA | Sarcoma - metabolism | Biomarkers, Tumor - metabolism | Sarcoma - diagnosis | Prognosis | Sarcoma - therapy | Humans | Sarcoma | Epidermal growth factor
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7. Full Text Human Nanog pseudogene8 promotes the proliferation of gastrointestinal cancer cells
by Uchino, Keita and Hirano, Gen and Hirahashi, Minako and Isobe, Taichi and Shirakawa, Tsuyoshi and Kusaba, Hitoshi and Baba, Eishi and Tsuneyoshi, Masazumi and Akashi, Koichi
Experimental Cell Research, ISSN 0014-4827, 09/2012, Volume 318, Issue 15, pp. 1799 - 1807
There is emerging evidence that human solid tumor cells originate from cancer stem cells (CSCs). In cancer cell lines, tumor-initiating CSCs are mainly found... 
Nanog | Proliferation | Cancer stem cells | Gastrointestinal cancer | Nanog P8 | INITIATING CELLS | SELF-RENEWAL | PLURIPOTENCY | IDENTIFICATION | TUMORS | CELL BIOLOGY | STEM-LIKE CELLS | ONCOLOGY | GENE NANOG | ES CELLS | CARCINOMA | EXPRESSION | Immunohistochemistry | Neoplasm Transplantation | RNA, Small Interfering - genetics | Cell Proliferation | Gastrointestinal Neoplasms - genetics | Homeodomain Proteins - metabolism | Humans | Molecular Sequence Data | Male | Transplantation, Heterologous | DNA Primers - genetics | Gastrointestinal Neoplasms - pathology | Transfection | Neoplastic Stem Cells - metabolism | Base Sequence | Neoplastic Stem Cells - pathology | Gastrointestinal Neoplasms - metabolism | Recombinant Proteins - metabolism | Nanog Homeobox Protein | Recombinant Proteins - genetics | Sequence Homology, Nucleic Acid | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Pseudogenes | Homeodomain Proteins - antagonists & inhibitors | Cell Line, Tumor | Mice | Medical law | Cancer cells | Stem cells | Cancer | Gene expression | Digestive system | NUCLEOTIDES | INHIBITION | NEOPLASMS | CELL PROLIFERATION | ANIMAL TISSUES | STEM CELLS | TUMOR CELLS | PROTEINS | 60 APPLIED LIFE SCIENCES
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8. Full Text Inflammatory myofibroblastic tumor versus igg4-related sclerosing disease and inflammatory pseudotumor: A comparative clinicopathologic study
by Yamamoto, Hidetaka and Yamaguchi, Hiroshi and Aishima, Shinichi and Oda, Yoshinao and Kohashi, Kenichi and Oshiro, Yumi and Tsuneyoshi, Masazumi
American Journal of Surgical Pathology, ISSN 0147-5185, 09/2009, Volume 33, Issue 9, pp. 1330 - 1340
Inflammatory pseudotumor (IPT) is a heterogeneous group of lesions occurring in various organs, which is histologically characterized by fibroblastic and... 
Immunohistochemistry | IgG4 | Inflammatory pseudotumor | Inflammatory myofibroblastic tumor | SURGERY | LUNG | DIAGNOSIS | PLASMA-CELL GRANULOMA | FUSION | inflammatory myofibroblastic tumor | FIBROUS HISTIOCYTOMA | PATHOLOGY | INFILTRATION | AUTOIMMUNE PANCREATITIS | inflammatory pseudotumor | immunohistochemistry | SPECTRUM | EXPRESSION | EPSTEIN-BARR-VIRUS | Protein-Tyrosine Kinases - metabolism | Humans | Middle Aged | Child, Preschool | Infant | Male | Dacryocystitis - immunology | Retroperitoneal Fibrosis - pathology | Sclerosis | Plasma Cells - pathology | Young Adult | Anaplastic Lymphoma Kinase | Phlebitis - immunology | Immunoglobulin G - immunology | Pancreatitis - immunology | Adult | Female | Phlebitis - pathology | Autoimmune Diseases - pathology | Child | Autoimmune Diseases - immunology | Receptor Protein-Tyrosine Kinases | Cholangitis, Sclerosing - pathology | Sialadenitis - pathology | Granuloma, Plasma Cell - pathology | Cholangitis, Sclerosing - immunology | Pancreatitis - pathology | Retroperitoneal Fibrosis - immunology | Aged | Granuloma, Plasma Cell - metabolism | Chronic Disease | Dacryocystitis - pathology | Sialadenitis - immunology
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9. Full Text CXCR4 and VEGF expression in the primary site and the metastatic site of human osteosarcoma: Analysis within a group of patients, all of whom developed lung metastasis
by Oda, Yoshinao and Yamamoto, Hidetaka and Tamiya, Sadafumi and Matsuda, Shuichi and Tanaka, Kazuhiro and Yokoyama, Ryohei and Iwamoto, Yukihide and Tsuneyoshi, Masazumi
Modern Pathology, ISSN 0893-3952, 05/2006, Volume 19, Issue 5, pp. 738 - 745
The chemokine, CXCL12, and its receptor, CXCR4, have recently been shown to play an important role in metastasis of several kinds of carcinoma. It has also... 
CXCR4 | Lung metastasis | Microvessel density | Osteosarcoma | VEGF | lung metastasis | TUMOR PROGRESSION | CHEMOKINE RECEPTOR CXCR4 | INVOLVEMENT | microvessel density | PATHOLOGY | RELAPSE | CANCER METASTASIS | POOR-PROGNOSIS | SQUAMOUS-CELL CARCINOMA | osteosarcoma | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Antigens, CD34 - analysis | Humans | Lung Neoplasms - metabolism | Middle Aged | Male | Bone Neoplasms - pathology | Bone Neoplasms - metabolism | Neovascularization, Pathologic - pathology | Ki-67 Antigen - analysis | Lung Neoplasms - secondary | Receptors, CXCR4 - biosynthesis | Bone Neoplasms - blood supply | Adolescent | Survival Analysis | Vascular Endothelial Growth Factors - biosynthesis | Adult | Female | Aged | Neovascularization, Pathologic - metabolism | Osteosarcoma - secondary | Child | Osteosarcoma - metabolism
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10. Full Text The Role of S100A6 in Pancreatic Cancer Development and Its Clinical Implication as a Diagnostic Marker and Therapeutic Target
by Kenoki Ohuchida and Kazuhiro Mizumoto and Nami Ishikawa and Kei Fujii and Hiroyuki Konomi and Eishi Nagai and Koji Yamaguchi and Masazumi Tsuneyoshi and Masao Tanaka
Clinical Cancer Research, ISSN 1078-0432, 11/2005, Volume 11, Issue 21, pp. 7785 - 7793
Recent microarray analyses showed that the S100 family contains members that are candidate diagnostic markers or therapeutic targets. In the present study, to... 
S100A6 | pancreatic cancer | pancreatic juice | PanIN | microdissection | PROTEIN | K-RAS GENE | ONCOLOGY | ADENOCARCINOMA | TELOMERASE ACTIVITY | JUICE | ACTIVATING TRANSCRIPTION FACTOR-3 | CELL-LINES | EXPRESSION | CARCINOMA | TUMOR-STROMAL INTERACTIONS | Immunohistochemistry | Oligonucleotides - chemistry | Up-Regulation | Cell Proliferation | Pancreatic Neoplasms - metabolism | Oligonucleotide Array Sequence Analysis | Pancreatic Neoplasms - diagnosis | Down-Regulation | Humans | Pancreatic Neoplasms - pathology | Cell Cycle Proteins - metabolism | Gene Expression Regulation, Neoplastic | S100 Calcium Binding Protein A6 | S100 Proteins - metabolism | Cell Cycle Proteins - biosynthesis | Pancreas - metabolism | RNA, Messenger - metabolism | Biomarkers, Tumor | Chemotactic Factors - biosynthesis | RNA Interference | Time Factors | Cell Line, Tumor | S100 Proteins - biosynthesis | RNA, Small Interfering - metabolism
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11. Full Text Histologic classification of microscopic portal venous invasion to predict prognosis in hepatocellular carcinoma
by Fujita, Nobuhiro, MD and Aishima, Shinichi, MD, PhD and Iguchi, Tomohiro, MD, PhD and Mano, Yohei, MD and Taketomi, Akinobu, MD, PhD and Shirabe, Ken, MD, PhD and Honda, Hiroshi, MD, PhD and Tsuneyoshi, Masazumi, MD, PhD and Oda, Yoshinao, MD, PhD
Human Pathology, ISSN 0046-8177, 2011, Volume 42, Issue 10, pp. 1531 - 1538
Summary Portal venous invasion is one of the most important prognostic factors after surgical resection of hepatocellular carcinoma. Microscopic portal venous... 
Pathology | Vascular invasion | Hepatocellular carcinoma | Portal venous invasion | SURVIVAL | SYSTEM | RESECTION | HEPATECTOMY | CIRRHOSIS | PATHOLOGY | Prognosis | Carcinoma, Hepatocellular - diagnosis | Neoplasm Invasiveness | Humans | Male | Survival Rate | Disease-Free Survival | Liver Neoplasms - diagnosis | Analysis of Variance | Carcinoma, Hepatocellular - pathology | Female | Liver Neoplasms - pathology | Portal Vein - pathology | Aged | Liver cancer | Analysis | Hepatoma | Studies | Tumors | Veins & arteries | Cancer
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12. Full Text S100P is a novel marker to identify intraductal papillary mucinous neoplasms
by Nakata, Kohei, PhD and Nagai, Eishi, PhD and Ohuchida, Kenoki, PhD and Hayashi, Akifumi, MD and Miyasaka, Yoshihiro, PhD and Aishima, Shinichi, PhD and Oda, Yoshinao, PhD and Mizumoto, Kazuhiro, PhD and Tanaka, Masao, PhD and Tsuneyoshi, Masazumi, PhD
Human Pathology, ISSN 0046-8177, 2010, Volume 41, Issue 6, pp. 824 - 831
Summary Intraductal papillary mucinous neoplasms of the pancreas are subclassified based on morphological features, and different immunohistochemical profiles... 
Pathology | Mucin expression | S100P | IPMN | Pancreatic cancer | ADENOCARCINOMA | PANCREATIC-CANCER | CONSENSUS | CLASSIFICATION | PATHOLOGY | TUMORS | LESIONS | CARCINOGENESIS | GENE-EXPRESSION | SURGICAL RESECTION | CARCINOMA | Immunohistochemistry | Pancreatic Neoplasms - metabolism | Adenocarcinoma, Mucinous - pathology | Pancreatic Neoplasms - diagnosis | Humans | Middle Aged | Carcinoma, Pancreatic Ductal - metabolism | Male | Carcinoma, Papillary - metabolism | Aged, 80 and over | Carcinoma, Pancreatic Ductal - diagnosis | Female | Diagnosis, Differential | Epithelium - pathology | Calcium-Binding Proteins - biosynthesis | Epithelium - metabolism | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreas - metabolism | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Papillary - pathology | Mucin-1 - biosynthesis | Adenocarcinoma, Mucinous - diagnosis | Adenocarcinoma, Mucinous - metabolism | Aged | Mucin 5AC - biosynthesis | Mucin-2 - biosynthesis | Biomarkers, Tumor - biosynthesis | Carcinoma, Papillary - diagnosis | Mucins | Proteins | Pancreas | Cervical cancer | Index Medicus
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13. Full Text Transcriptional Repressor Snail and Progression of Human Hepatocellular Carcinoma
by Keishi Sugimachi and Shinji Tanaka and Toshifumi Kameyama and Ken-ichi Taguchi and Shin-ichi Aishima and Mitsuo Shimada and Keizo Sugimachi and Masazumi Tsuneyoshi
Clinical Cancer Research, ISSN 1078-0432, 07/2003, Volume 9, Issue 7, pp. 2657 - 2664
Purpose: Snail protein is a suppressive transcriptional factor of E-cadherin that mediates cell-to-cell adhesion, tumor progression, and metastases. We... 
INACTIVATION | METHYLATION | INVASION | CELL-ADHESION SYSTEM | ONCOLOGY | EPITHELIAL-MESENCHYMAL TRANSITIONS | MUTATIONS | BETA-CATENIN | ALTERED EXPRESSION | E-CADHERIN GENE | Immunohistochemistry | Green Fluorescent Proteins | Cadherins - metabolism | Bile Ducts - metabolism | Humans | Middle Aged | Male | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Tissue Distribution | Neoplasm Metastasis | In Situ Hybridization | Transfection | Cloning, Molecular | Female | Liver Neoplasms - pathology | Transcription, Genetic | Retrospective Studies | Snail Family Transcription Factors | Protein Structure, Tertiary | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Neoplasm Invasiveness | Down-Regulation | Reverse Transcriptase Polymerase Chain Reaction | Disease Progression | Transcription Factors - metabolism | Liver Neoplasms - metabolism | Aged | DNA, Complementary - metabolism | Leukocytes - metabolism | Microscopy, Fluorescence | Carcinoma, Hepatocellular - metabolism | Luminescent Proteins - metabolism
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14. Full Text Detection and characterization of vascular endothelial growth factors and their receptors in a series of angiosarcomas
by Itakura, Eijun and Yamamoto, Hidetaka and Oda, Yoshinao and Tsuneyoshi, Masazumi
Journal of Surgical Oncology, ISSN 0022-4790, 01/2008, Volume 97, Issue 1, pp. 74 - 81
Background Angiosarcomas are malignant mesenchymal neoplasms, including sarcomas of presumptive vascular endothelial origin and sarcomas of probable lymphatic... 
VEGF‐C | VEGFR‐3 | vascular neoplasms | skin neoplasms | VEGF‐A | soft tissue neoplasms | Stewart‐Treves syndrome | Stewart-Treves syndrome | Skin neoplasms | VEGFR-3 | Vascular neoplasms | VEGF-C | Soft tissue neoplasms | VEGF-A | SURGERY | TYROSINE KINASE | CUTANEOUS ANGIOSARCOMA | TUMORS | LYMPH-NODE METASTASIS | KAPOSIS-SARCOMA | ONCOLOGY | STEWART-TREVES-SYNDROME | LYMPHANGIOGENESIS | EXPRESSION | SOFT-TISSUE SARCOMA | Immunohistochemistry | Hemangiosarcoma - pathology | Prognosis | Receptors, Vascular Endothelial Growth Factor - analysis | Humans | Middle Aged | Male | Survival Rate | Vascular Endothelial Growth Factor A - analysis | Hemangiosarcoma - chemistry | Hemangiosarcoma - mortality | Aged, 80 and over | Adult | Female | Aged
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