American journal of respiratory cell and molecular biology, 07/2019
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 11/2018, Volume 9, p. 2598
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 07/2017, Volume 8, p. 757
Disruption of the alveolar-capillary barrier and accumulation of pulmonary edema, if not resolved, result in poor alveolar gas exchange leading to hypoxia and...
Acute respiratory distress syndrome | Acute lung injury | Hypercapnia | Alveolar fluid clearance | Na,K-ATPase | channel | Hypoxia | Epithelial Na | hypoxia | NA,K-ATPASE DOWN-REGULATION | hypercapnia | ELEVATED CO2 LEVELS | IMMUNOLOGY | acute lung injury | PULMONARY-EDEMA | RESPIRATORY-DISTRESS-SYNDROME | MECHANICALLY VENTILATED PATIENTS | alveolar fluid clearance | EPITHELIAL-CELLS | A549 CELLS | PKC-ZETA | acute respiratory distress syndrome | K-ATPase | epithelial Na+ channel
Acute respiratory distress syndrome | Acute lung injury | Hypercapnia | Alveolar fluid clearance | Na,K-ATPase | channel | Hypoxia | Epithelial Na | hypoxia | NA,K-ATPASE DOWN-REGULATION | hypercapnia | ELEVATED CO2 LEVELS | IMMUNOLOGY | acute lung injury | PULMONARY-EDEMA | RESPIRATORY-DISTRESS-SYNDROME | MECHANICALLY VENTILATED PATIENTS | alveolar fluid clearance | EPITHELIAL-CELLS | A549 CELLS | PKC-ZETA | acute respiratory distress syndrome | K-ATPase | epithelial Na+ channel
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY, ISSN 1040-0605, 05/2019, Volume 316, Issue 5, pp. L738 - L739
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 11/2013, Volume 305, Issue 10, pp. L665 - L681
In this review we summarize recent major advances in our understanding on the molecular mechanisms, mediators, and biomarkers of acute lung injury (ALI) and...
Zaacute lung injury/acute respiratory distress syndrome | Pulmonary edema | Molecular mechanism | Pharmacological and cell-based therapy | Alveolar-capillary barrier dysfunction | pulmonary edema | AIRWAY EPITHELIAL-CELLS | PHYSIOLOGY | COLONY-STIMULATING FACTOR | MICROVASCULAR ENDOTHELIAL-CELLS | alveolar-capillary barrier dysfunction | MESENCHYMAL STEM-CELLS | LETHAL INFLUENZA INFECTION | RESPIRATORY-DISTRESS-SYNDROME | molecular mechanism | NEUTROPHIL EXTRACELLULAR TRAPS | zaacute lung injury/acute respiratory distress syndrome | RESPIRATORY SYSTEM | pharmacological and cell-based therapy | TRANSEPITHELIAL LIQUID TRANSPORT | PROVIDING PERMISSIVE HYPERCAPNIA | GLYCATION END-PRODUCTS | Acute Lung Injury - physiopathology | Capillaries - physiopathology | Animals | Capillary Permeability | Humans | Cell Communication | Pulmonary Alveoli - physiopathology | Blood-Air Barrier - physiopathology | Acute respiratory distress syndrome | Physiological aspects | Research | Molecular biology
Zaacute lung injury/acute respiratory distress syndrome | Pulmonary edema | Molecular mechanism | Pharmacological and cell-based therapy | Alveolar-capillary barrier dysfunction | pulmonary edema | AIRWAY EPITHELIAL-CELLS | PHYSIOLOGY | COLONY-STIMULATING FACTOR | MICROVASCULAR ENDOTHELIAL-CELLS | alveolar-capillary barrier dysfunction | MESENCHYMAL STEM-CELLS | LETHAL INFLUENZA INFECTION | RESPIRATORY-DISTRESS-SYNDROME | molecular mechanism | NEUTROPHIL EXTRACELLULAR TRAPS | zaacute lung injury/acute respiratory distress syndrome | RESPIRATORY SYSTEM | pharmacological and cell-based therapy | TRANSEPITHELIAL LIQUID TRANSPORT | PROVIDING PERMISSIVE HYPERCAPNIA | GLYCATION END-PRODUCTS | Acute Lung Injury - physiopathology | Capillaries - physiopathology | Animals | Capillary Permeability | Humans | Cell Communication | Pulmonary Alveoli - physiopathology | Blood-Air Barrier - physiopathology | Acute respiratory distress syndrome | Physiological aspects | Research | Molecular biology
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2016, Volume 126, Issue 4, pp. 1566 - 1580
Influenza A viruses (IAV) can cause lung injury and acute respiratory distress syndrome (ARDS), which is characterized by accumulation of excessive fluid...
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATED PROTEIN-KINASE | APOPTOSIS-INDUCING LIGAND | VIRUS-INFECTION | CYTOKINE RESPONSES | IN-VIVO | ALVEOLAR FLUID CLEARANCE | LIQUID CLEARANCE | UP-REGULATION | RESPIRATORY-DISTRESS-SYNDROME | Respiratory Distress Syndrome, Adult - pathology | Macrophages, Alveolar - pathology | Pulmonary Alveoli - pathology | Humans | Orthomyxoviridae Infections - pathology | TNF-Related Apoptosis-Inducing Ligand - immunology | Interferon Type I - immunology | Paracrine Communication - immunology | Respiratory Mucosa - pathology | Respiratory Distress Syndrome, Adult - immunology | Animals | Respiratory Mucosa - immunology | Pulmonary Edema - immunology | Influenza A virus - immunology | Pulmonary Edema - pathology | Mice | Sodium-Potassium-Exchanging ATPase - immunology | Macrophages, Alveolar - immunology | Orthomyxoviridae Infections - immunology | Lung diseases | Influenza | Physiological aspects | Development and progression | Genetic aspects | Properties | Adenosine triphosphatase | Proteins | Flow cytometry | Edema | Sodium | Respiratory distress syndrome | Rodents | Infections | Communications networks | Gene expression
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATED PROTEIN-KINASE | APOPTOSIS-INDUCING LIGAND | VIRUS-INFECTION | CYTOKINE RESPONSES | IN-VIVO | ALVEOLAR FLUID CLEARANCE | LIQUID CLEARANCE | UP-REGULATION | RESPIRATORY-DISTRESS-SYNDROME | Respiratory Distress Syndrome, Adult - pathology | Macrophages, Alveolar - pathology | Pulmonary Alveoli - pathology | Humans | Orthomyxoviridae Infections - pathology | TNF-Related Apoptosis-Inducing Ligand - immunology | Interferon Type I - immunology | Paracrine Communication - immunology | Respiratory Mucosa - pathology | Respiratory Distress Syndrome, Adult - immunology | Animals | Respiratory Mucosa - immunology | Pulmonary Edema - immunology | Influenza A virus - immunology | Pulmonary Edema - pathology | Mice | Sodium-Potassium-Exchanging ATPase - immunology | Macrophages, Alveolar - immunology | Orthomyxoviridae Infections - immunology | Lung diseases | Influenza | Physiological aspects | Development and progression | Genetic aspects | Properties | Adenosine triphosphatase | Proteins | Flow cytometry | Edema | Sodium | Respiratory distress syndrome | Rodents | Infections | Communications networks | Gene expression
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 01/2014, Volume 111, Issue 3, pp. E374 - E383
TGF-beta is a pathogenic factor in patients with acute respiratory distress syndrome (ARDS), a condition characterized by alveolar edema. A unique TGF-beta...
Alveolar epithelium | Fluid homeostasis | MOLECULAR CHARACTERIZATION | fluid homeostasis | BIOPHYSICAL PROPERTIES | MULTIDISCIPLINARY SCIENCES | CELL-SURFACE EXPRESSION | RESPIRATORY-DISTRESS-SYNDROME | alveolar epithelium | REACTIVE OXYGEN | NA+ CHANNEL | TRANSFORMING GROWTH-FACTOR-BETA-1 | DISEASE | NADPH OXIDASES | UBIQUITIN LIGASE NEDD4L | Rabbits | Reactive Oxygen Species | Humans | Middle Aged | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Ions | Male | Epithelial Sodium Channels - metabolism | Respiratory Distress Syndrome, Adult - metabolism | Phospholipase D - metabolism | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Animals | Perfusion | Pulmonary Alveoli - metabolism | Adult | Female | Acute Lung Injury - metabolism | Aged | Lung - metabolism | Mice | Transforming Growth Factor beta - metabolism | Biological Sciences | PNAS Plus
Alveolar epithelium | Fluid homeostasis | MOLECULAR CHARACTERIZATION | fluid homeostasis | BIOPHYSICAL PROPERTIES | MULTIDISCIPLINARY SCIENCES | CELL-SURFACE EXPRESSION | RESPIRATORY-DISTRESS-SYNDROME | alveolar epithelium | REACTIVE OXYGEN | NA+ CHANNEL | TRANSFORMING GROWTH-FACTOR-BETA-1 | DISEASE | NADPH OXIDASES | UBIQUITIN LIGASE NEDD4L | Rabbits | Reactive Oxygen Species | Humans | Middle Aged | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Ions | Male | Epithelial Sodium Channels - metabolism | Respiratory Distress Syndrome, Adult - metabolism | Phospholipase D - metabolism | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Animals | Perfusion | Pulmonary Alveoli - metabolism | Adult | Female | Acute Lung Injury - metabolism | Aged | Lung - metabolism | Mice | Transforming Growth Factor beta - metabolism | Biological Sciences | PNAS Plus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 05/2011, Volume 183, Issue 9, pp. 1147 - 1152
In this Update, we provide an overview of selected studies published in 2010 in the American Journal of Respiratory and Critical Care Medicine and other...
PREDICTION SCORE | CRITICAL ILLNESS | MITOCHONDRIAL BIOGENESIS | RESPIRATORY SYSTEM | SEPTIC SHOCK | II CELLS | RANDOMIZED CONTROLLED-TRIAL | MESENCHYMAL STEM-CELLS | INTENSIVE-CARE | RESPIRATORY-DISTRESS-SYNDROME | ENDOTHELIAL BARRIER FUNCTION | CRITICAL CARE MEDICINE | Critical Care - methods | Acute Lung Injury - diagnosis | Acute Lung Injury - physiopathology | Animals | Acute Lung Injury - therapy | Humans | Mice | Pulmonary, Sleep, and Critical Care Updates
PREDICTION SCORE | CRITICAL ILLNESS | MITOCHONDRIAL BIOGENESIS | RESPIRATORY SYSTEM | SEPTIC SHOCK | II CELLS | RANDOMIZED CONTROLLED-TRIAL | MESENCHYMAL STEM-CELLS | INTENSIVE-CARE | RESPIRATORY-DISTRESS-SYNDROME | ENDOTHELIAL BARRIER FUNCTION | CRITICAL CARE MEDICINE | Critical Care - methods | Acute Lung Injury - diagnosis | Acute Lung Injury - physiopathology | Animals | Acute Lung Injury - therapy | Humans | Mice | Pulmonary, Sleep, and Critical Care Updates
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0172116
A hallmark of acute respiratory distress syndrome (ARDS) is accumulation of protein-rich edema in the distal airspaces and its removal is critical for patient...
CLEARANCE | ACTIVATION | INHIBITION | PROTECTION | HUR | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ACUTE LUNG INJURY | MECHANISMS | BINDING PROTEIN | EXPRESSION | Respiratory Distress Syndrome, Adult - pathology | A549 Cells | Respiratory Distress Syndrome, Adult - chemically induced | Humans | RNA, Messenger - genetics | ELAV-Like Protein 1 - genetics | Glycogen Synthase Kinase 3 beta - genetics | Hydrochloric Acid - toxicity | RNA Stability | RNA, Messenger - metabolism | Respiratory Distress Syndrome, Adult - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Respiratory Distress Syndrome, Adult - genetics | Animals | ELAV-Like Protein 1 - metabolism | Mice | Disease Models, Animal | Post-transcription | Pediatrics | Laboratories | Stabilization | Gene regulation | Lung cancer | Critical care | Mitochondrial DNA | Kinases | Autophagy | Datasets | Hydrochloric acid | Proteins | Inhibition | Protein transport | Alveoli | Binding | HuR protein | Edema | Antigens | Departments | Glycogen | Mortality | Drosophila | Glycogen synthase kinase 3 | Albumin | siRNA | Gene expression | Ribonucleic acid--RNA | Embryos | College campuses | Medicine | Signaling | RNA-binding protein | Respiratory distress syndrome | Anesthesiology | Molecular biology | mRNA stability | RNA | Ribonucleic acid
CLEARANCE | ACTIVATION | INHIBITION | PROTECTION | HUR | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ACUTE LUNG INJURY | MECHANISMS | BINDING PROTEIN | EXPRESSION | Respiratory Distress Syndrome, Adult - pathology | A549 Cells | Respiratory Distress Syndrome, Adult - chemically induced | Humans | RNA, Messenger - genetics | ELAV-Like Protein 1 - genetics | Glycogen Synthase Kinase 3 beta - genetics | Hydrochloric Acid - toxicity | RNA Stability | RNA, Messenger - metabolism | Respiratory Distress Syndrome, Adult - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Respiratory Distress Syndrome, Adult - genetics | Animals | ELAV-Like Protein 1 - metabolism | Mice | Disease Models, Animal | Post-transcription | Pediatrics | Laboratories | Stabilization | Gene regulation | Lung cancer | Critical care | Mitochondrial DNA | Kinases | Autophagy | Datasets | Hydrochloric acid | Proteins | Inhibition | Protein transport | Alveoli | Binding | HuR protein | Edema | Antigens | Departments | Glycogen | Mortality | Drosophila | Glycogen synthase kinase 3 | Albumin | siRNA | Gene expression | Ribonucleic acid--RNA | Embryos | College campuses | Medicine | Signaling | RNA-binding protein | Respiratory distress syndrome | Anesthesiology | Molecular biology | mRNA stability | RNA | Ribonucleic acid
Journal Article
PLoS Pathogens, ISSN 1553-7366, 06/2016, Volume 12, Issue 6, p. e1005544
Influenza Virus (IV) pneumonia is associated with severe damage of the lung epithelium and respiratory failure. Apart from efficient host defense, structural...
PROGENITOR CELLS | AIRWAY STEM-CELLS | INJURY | MICROBIOLOGY | ADULT-MOUSE LUNG | RESPIRATORY-DISTRESS-SYNDROME | ALVEOLAR | ACTS UPSTREAM | VIROLOGY | MICE | H1N1 INFLUENZA | EXPRESSION | PARASITOLOGY | Immunohistochemistry | Stem Cells - virology | Epithelial Cells - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Cell Separation | Orthomyxoviridae Infections - metabolism | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Pneumonia, Viral - pathology | Epithelial Cells - pathology | Mice, Transgenic | Stem Cells - metabolism | Pneumonia, Viral - metabolism | Animals | Flow Cytometry | Epithelial Cells - virology | Fluorescent Antibody Technique | Influenza A virus - pathogenicity | Mice | Orthomyxoviridae Infections - virology | Pneumonia, Viral - virology | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Influenza viruses | Physiological aspects | Fibroblast growth factors | Cellular signal transduction | Research | Epithelial cells | Pneumonia | Respiratory distress syndrome | Rodents | Influenza | Stem cells | Viruses | Infections | Experiments
PROGENITOR CELLS | AIRWAY STEM-CELLS | INJURY | MICROBIOLOGY | ADULT-MOUSE LUNG | RESPIRATORY-DISTRESS-SYNDROME | ALVEOLAR | ACTS UPSTREAM | VIROLOGY | MICE | H1N1 INFLUENZA | EXPRESSION | PARASITOLOGY | Immunohistochemistry | Stem Cells - virology | Epithelial Cells - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Cell Separation | Orthomyxoviridae Infections - metabolism | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Pneumonia, Viral - pathology | Epithelial Cells - pathology | Mice, Transgenic | Stem Cells - metabolism | Pneumonia, Viral - metabolism | Animals | Flow Cytometry | Epithelial Cells - virology | Fluorescent Antibody Technique | Influenza A virus - pathogenicity | Mice | Orthomyxoviridae Infections - virology | Pneumonia, Viral - virology | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Influenza viruses | Physiological aspects | Fibroblast growth factors | Cellular signal transduction | Research | Epithelial cells | Pneumonia | Respiratory distress syndrome | Rodents | Influenza | Stem cells | Viruses | Infections | Experiments
Journal Article
DMM Disease Models and Mechanisms, ISSN 1754-8403, 02/2017, Volume 10, Issue 2, pp. 185 - 196
Progress in developing new therapies for bronchopulmonary dysplasia (BPD) is sometimes complicated by the lack of a standardised animal model. Our objective...
Animal model | Structure | Hyperoxia | Alveolarisation | BPD | STEREOLOGY | NEONATAL EXPOSURE | CYCLOOXYGENASE-2 | REGENERATION | LUNG INJURY | ADULT MICE | PATHOLOGY | CELL BIOLOGY | AIRWAY | INFLUENZA-A VIRUS | ALVEOLARIZATION | Animals, Newborn | Hyperoxia - pathology | Lung - pathology | Oxygen - administration & dosage | Reference Standards | Animals | Hyperoxia - complications | Mice, Inbred C57BL | Bronchopulmonary Dysplasia - pathology | Bronchopulmonary Dysplasia - complications | Disease Models, Animal | Resource
Animal model | Structure | Hyperoxia | Alveolarisation | BPD | STEREOLOGY | NEONATAL EXPOSURE | CYCLOOXYGENASE-2 | REGENERATION | LUNG INJURY | ADULT MICE | PATHOLOGY | CELL BIOLOGY | AIRWAY | INFLUENZA-A VIRUS | ALVEOLARIZATION | Animals, Newborn | Hyperoxia - pathology | Lung - pathology | Oxygen - administration & dosage | Reference Standards | Animals | Hyperoxia - complications | Mice, Inbred C57BL | Bronchopulmonary Dysplasia - pathology | Bronchopulmonary Dysplasia - complications | Disease Models, Animal | Resource
Journal Article
Chest, ISSN 0012-3692, 2012, Volume 141, Issue 3, pp. 763 - 771
Ubiquitination is a posttranslational modification that regulates a variety of cellular functions depending on timing, subcellular localization, and type of...
Pulmonary/Respiratory | PLASMA PROTEASOME LEVEL | NA,K-ATPASE ENDOCYTOSIS | RESPIRATORY SYSTEM | ALVEOLAR FLUID CLEARANCE | K-ATPASE | CIRCULATING PROTEASOMES | MECHANICAL VENTILATION | MEDIATED DEGRADATION | 26S PROTEASOME | INFLAMMATORY RESPONSE | RESPIRATORY-DISTRESS-SYNDROME | CRITICAL CARE MEDICINE | Acute Lung Injury - physiopathology | Sodium-Potassium-Exchanging ATPase - physiology | Proteolysis | Humans | Ubiquitination - physiology | Epithelial Sodium Channels - physiology | Pulmonary Alveoli - physiopathology | Proteasome Endopeptidase Complex - physiology | Acute respiratory distress syndrome | Physiological aspects | Development and progression | Care and treatment | Ubiquitin-proteasome system | Translating Basic Research into Clinical Practice
Pulmonary/Respiratory | PLASMA PROTEASOME LEVEL | NA,K-ATPASE ENDOCYTOSIS | RESPIRATORY SYSTEM | ALVEOLAR FLUID CLEARANCE | K-ATPASE | CIRCULATING PROTEASOMES | MECHANICAL VENTILATION | MEDIATED DEGRADATION | 26S PROTEASOME | INFLAMMATORY RESPONSE | RESPIRATORY-DISTRESS-SYNDROME | CRITICAL CARE MEDICINE | Acute Lung Injury - physiopathology | Sodium-Potassium-Exchanging ATPase - physiology | Proteolysis | Humans | Ubiquitination - physiology | Epithelial Sodium Channels - physiology | Pulmonary Alveoli - physiopathology | Proteasome Endopeptidase Complex - physiology | Acute respiratory distress syndrome | Physiological aspects | Development and progression | Care and treatment | Ubiquitin-proteasome system | Translating Basic Research into Clinical Practice
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 04/2012, Volume 46, Issue 4, pp. 417 - 421
Patients with severe acute and chronic lung diseases develop derangements in gas exchange that may result in increased levels of CO2 (hypercapnia), the effects...
Innate immunity | Alveolar epithelial barrier function | CO | Na,K-ATPase | COPD | ACTIVATED PROTEIN-KINASE | ALVEOLAR EPITHELIAL-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CO2 | ACIDOSIS | alveolar epithelial barrier function | TIDAL VOLUMES | PLASMA-MEMBRANE | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | innate immunity | VENTILATION STRATEGY | RESPIRATORY SYSTEM | HOST-DEFENSE | CARBON-DIOXIDE | Humans | Lung | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - cytology | Acute Lung Injury - metabolism | Cell Membrane - metabolism | Hypercapnia - etiology | Immunity, Innate | Carbon Dioxide - blood | Pulmonary Edema - etiology | Respiration, Artificial
Innate immunity | Alveolar epithelial barrier function | CO | Na,K-ATPase | COPD | ACTIVATED PROTEIN-KINASE | ALVEOLAR EPITHELIAL-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CO2 | ACIDOSIS | alveolar epithelial barrier function | TIDAL VOLUMES | PLASMA-MEMBRANE | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | innate immunity | VENTILATION STRATEGY | RESPIRATORY SYSTEM | HOST-DEFENSE | CARBON-DIOXIDE | Humans | Lung | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - cytology | Acute Lung Injury - metabolism | Cell Membrane - metabolism | Hypercapnia - etiology | Immunity, Innate | Carbon Dioxide - blood | Pulmonary Edema - etiology | Respiration, Artificial
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 06/2013, Volume 110, Issue 25, pp. E2308 - E2316
Alveolar fluid clearance driven by active epithelial Na+ and secondary Cl- absorption counteracts edema formation in the intact lung. Recently, we showed that...
Epithelial Cl | Pulmonary edema | transport | pulmonary edema | ION-TRANSPORT | MULTIDISCIPLINARY SCIENCES | VOLUME | EPITHELIAL ION | NA+ ABSORPTION | RESPIRATORY-DISTRESS-SYNDROME | epithelial Cl- transport | ACUTE PULMONARY-EDEMA | FUROSEMIDE | II CELLS | CHANNELS | CFTR | Male | Sodium-Potassium-Chloride Symporters - metabolism | Pulmonary Edema - etiology | Chlorides - metabolism | Pulmonary Alveoli - metabolism | Pulmonary Edema - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Heart Failure - complications | Mice, Inbred CFTR | Rabbits | Diuretics - pharmacology | Mice, Inbred C57BL | Rats | Sodium-Potassium-Chloride Symporters - genetics | Body Fluids - secretion | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Heart Failure - metabolism | Hydrostatic Pressure | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Solute Carrier Family 12, Member 2 | Body Fluids - metabolism | Amiloride - pharmacology | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | Respiratory Mucosa - metabolism | Furosemide - pharmacology | Biological Sciences | epithelial Cl− transport | PNAS Plus
Epithelial Cl | Pulmonary edema | transport | pulmonary edema | ION-TRANSPORT | MULTIDISCIPLINARY SCIENCES | VOLUME | EPITHELIAL ION | NA+ ABSORPTION | RESPIRATORY-DISTRESS-SYNDROME | epithelial Cl- transport | ACUTE PULMONARY-EDEMA | FUROSEMIDE | II CELLS | CHANNELS | CFTR | Male | Sodium-Potassium-Chloride Symporters - metabolism | Pulmonary Edema - etiology | Chlorides - metabolism | Pulmonary Alveoli - metabolism | Pulmonary Edema - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Heart Failure - complications | Mice, Inbred CFTR | Rabbits | Diuretics - pharmacology | Mice, Inbred C57BL | Rats | Sodium-Potassium-Chloride Symporters - genetics | Body Fluids - secretion | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Heart Failure - metabolism | Hydrostatic Pressure | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Solute Carrier Family 12, Member 2 | Body Fluids - metabolism | Amiloride - pharmacology | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | Respiratory Mucosa - metabolism | Furosemide - pharmacology | Biological Sciences | epithelial Cl− transport | PNAS Plus
Journal Article