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Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 413 - 424
How glucose sensing by the nervous system impacts the regulation of [beta] cell mass and function during post-natal development and throughout adulthood is... 
Physiological aspects | Homeostasis | Nervous system | Pancreatic beta cells | Research | Studies | Glucose | Pancreas | Insulin | Rodents
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1662 - 1676
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, p. 1662
  Liver glucose metabolism plays a central role in glucose homeostasis and may also regulate feeding and energy expenditure. Here we assessed the impact of... 
Glucose metabolism | Pancreatic beta cells | Genetic aspects | Research | Gene expression | Liver
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, p. 413
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 413 - 413
How glucose sensing by the nervous system impacts the regulation of beta cell mass and function during postnatal development and throughout adulthood is... 
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e103277
Background/Aims: Gluco-incretin hormones increase the glucose competence of pancreatic beta-cells by incompletely characterized mechanisms. Methods: We... 
TYPE-2 DIABETIC-PATIENTS | PROTEIN | GLUCAGON-LIKE PEPTIDE-1 | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | INSULIN-SECRETION | 7-36 AMIDE | RECEPTOR | CARDIAC NA+/CA2+ EXCHANGER | PROLIFERATION | GASTRIC-INHIBITORY POLYPEPTIDE | Animals, Newborn | Gene Silencing - drug effects | Membrane Proteins - genetics | Humans | Mice, Inbred C57BL | Cells, Cultured | Incretins - metabolism | Neoplasm Proteins - metabolism | Promoter Regions, Genetic - drug effects | Eating - physiology | Mice, Knockout | Insulin-Secreting Cells - metabolism | Animals | Insulin-Secreting Cells - drug effects | Incretins - pharmacology | Glucose - metabolism | Epigenesis, Genetic - drug effects | Membrane Proteins - metabolism | Mice | Neoplasm Proteins - genetics | DNA Methylation - drug effects | Epigenetic inheritance | Pancreatic beta cells | Promoters (Genetics) | Analysis | Glucose | Methylation | Gene expression | Dextrose | Diabetes therapy | Nutrition | Secretion | Diabetes mellitus | Hormones | Kinases | Insulin | Calcium influx | Depolarization | Beta cells | Rodents | Imprinting | DNA methylation | Epigenetics | Membrane potential | Pancreas | Diabetes mellitus (non-insulin dependent) | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
Journal Article
Nature Immunology, ISSN 1529-2908, 03/2017, Volume 18, Issue 3, p. 283
  The deleterious effect of chronic activation of the IL-1β system on type 2 diabetes and other metabolic diseases is well documented. However, a possible... 
Type 2 diabetes | Glucose metabolism | Obesity | Physiological aspects | Inflammation | Glucose | Macrophages | Insulin | Dextrose
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e30847
Background: The control of the functional pancreatic beta-cell mass serves the key homeostatic function of releasing the right amount of insulin to keep blood... 
INSULIN | APOPTOSIS | GLUCOSE-HOMEOSTASIS | CHRONIC EXPOSURE | NEOGENESIS | GLUCAGON-LIKE PEPTIDE-1 | MULTIDISCIPLINARY SCIENCES | GROWTH | GENE-EXPRESSION | CATENIN | TRANSCRIPTION FACTOR-II | Embryo, Mammalian | Cell Count | Humans | Glucagon-Like Peptide 1 - physiology | Male | COUP Transcription Factor II - physiology | Glucagon-Like Peptide 1 - genetics | Insulin-Secreting Cells - metabolism | Organ Size - genetics | Female | Insulin-Secreting Cells - cytology | beta Catenin - physiology | Animals, Newborn | Glucagon-Like Peptide 1 - metabolism | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Pancreas - drug effects | Rats | Mice, Transgenic | Signal Transduction - genetics | Pancreas - metabolism | Pancreas - embryology | beta Catenin - metabolism | beta Catenin - genetics | Organ Size - drug effects | Animals | Insulin-Secreting Cells - drug effects | Signal Transduction - drug effects | COUP Transcription Factor II - genetics | Models, Biological | Signal Transduction - physiology | Mice | COUP Transcription Factor II - metabolism | Pancreas - growth & development | Cell proliferation | Neonates | Transcription factors | Phosphorylation | Wnt protein | Glucagon | Laboratories | Genes | Lymphocytes T | Biology | Feedback loops | Glucose | Cyclin D1 | Homeobox | β-catenin | Signal transduction | Rodents | Extracellular matrix | Pancreas | Bioinformatics | Glucagon-like peptide 1 | Sugar | Hypertension | Ovalbumin | Metabolism | Gene expression | Cell differentiation | Epithelium | Insulin | Ablation | Glucose tolerance | Beta cells | Pathology | Intolerance | Signaling | Cell number | Insulin resistance | Control theory | Diabetes | Liver cirrhosis | Endocrinology | Apoptosis | Cancer
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1662 - 1662
Liver glucose metabolism plays a central role in glucose homeostasis and may also regulate feeding and energy expenditure. Here we assessed the impact of... 
Journal Article
Medecine/Sciences, ISSN 0767-0974, 05/2017, Volume 33, Issue 5, pp. 469 - 473
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, p. 4105
PPAR[beta]/[delta] protects against obesity by reducing dyslipidemia and insulin resistance via effects in muscle, adipose tissue, and liver. However, its... 
Glucose metabolism | Pancreatic beta cells | Genetic aspects | Dosage and administration | Research | Gene expression | Insulin
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4105 - 4117
Journal Article
Medecine sciences : M/S, 05/2017, Volume 33, Issue 5, p. 469
Journal Article