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1. Full Text Nervous glucose sensing regulates postnatal â cell proliferation and glucose homeostasis
by Tarussio, David and Metref, Salima and Seyer, Pascal and Mounien, Lourdes and Vallois, David and Magnan, Christophe and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 413 - 424
How glucose sensing by the nervous system impacts the regulation of [beta] cell mass and function during post-natal development and throughout adulthood is... 
Physiological aspects | Homeostasis | Nervous system | Pancreatic beta cells | Research | Studies | Glucose | Pancreas | Insulin | Rodents
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2. Full Text Type II enteropathy-associated T-cell lymphoma features a unique genomic profile with highly recurrent SETD2 alterations
by Roberti, Annalisa and Dobay, Maria Pamela and Bisig, Bettina and Vallois, David and Boéchat, Cloé and Lanitis, Evripidis and Bouchindhomme, Brigitte and Parrens, Marie-Cécile and Bossard, Céline and Quintanilla-Martinez, Leticia and Missiaglia, Edoardo and Gaulard, Philippe and De Leval, Laurence
Nature Communications, ISSN 2041-1723, 09/2016, Volume 7, Issue 1, p. 12602
Enteropathy-associated T-cell lymphoma (EATL), a rare and aggressive intestinal malignancy of intraepithelial T lymphocytes, comprises two disease variants... 
EPIGENETIC REGULATORS | STAT3 MUTATIONS | PROTEIN STABILITY | NK CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | GENES | GAMMA-DELTA-T | CANCER | SOMATIC MUTATIONS | WEB SERVER | Genetic Predisposition to Disease | Histone-Lysine N-Methyltransferase - genetics | Enteropathy-Associated T-Cell Lymphoma - genetics | Intestinal Neoplasms - classification | Genomics | Humans | Mutation | Enteropathy-Associated T-Cell Lymphoma - classification | Gene Expression Regulation, Neoplastic - physiology | Intestinal Neoplasms - genetics
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3. Full Text Nervous glucose sensing regulates postnatal beta cell proliferation and glucose homeostasis
by Tarussio, D and Metref, S and Seyer, P and Mounien, L and Vallois, D and Magnan, C and Foretz, M and Thorens, B
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 413 - 424
How glucose sensing by the nervous system impacts the regulation of beta cell mass and function during postnatal development and throughout adulthood is... 
ADULT-RAT BRAIN | GLUCAGON-SECRETION | MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC-ISLETS | ELECTRICAL-STIMULATION | GLUCOSE-TRANSPORTER-2 GLUT2 | INSULIN-SECRETION | IN-VIVO | GLUT2-NULL MICE | MUSCARINIC ACETYLCHOLINE-RECEPTORS | CEPHALIC-PHASE | Glucose Intolerance - metabolism | Glucose Transporter Type 2 - genetics | Cell Proliferation | Mice, Inbred C57BL | Homeostasis | Pancreas - pathology | Male | Mice, 129 Strain | Ganglia, Parasympathetic - metabolism | Mice, Knockout | Action Potentials | Glucose Transporter Type 2 - deficiency | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Autonomic Fibers, Preganglionic - physiology | Pancreas - innervation | Energy Metabolism | Glucose - metabolism | Female | Mice | Insulin-Secreting Cells - pathology | Insulin Secretion | Ganglia, Parasympathetic - physiopathology
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4. Full Text Hepatic glucose sensing is required to preserve β cell glucose competence
by Seyer, Pascal and Vallois, David and Poitry-Yamate, Carole and Schütz, Frédéric and Metref, Salima and Tarussio, David and Maechler, Pierre and Staels, Bart and Lanz, Bernard and Grueter, Rolf and Decaris, Julie and Turner, Scott and Da Costa, Anabela and Preitner, Frédéric and Minehira, Kaori and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1662 - 1676
Liver glucose metabolism plays a central role in glucose homeostasis and may also regulate feeding and energy expenditure. Here we assessed the impact of... 
MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCYTES | FARNESOID X RECEPTOR | INSULIN-RESISTANCE | FOOD-INTAKE | MIXED MEAL | MICE | FACTOR-I | DIET-INDUCED OBESITY | BILE-ACIDS | RAT MODEL | Glucose Transporter Type 2 - genetics | Cholesterol - blood | Transcriptome | Fluorodeoxyglucose F18 - metabolism | Homeostasis | Mice, 129 Strain | Liver - physiopathology | Glucose Intolerance - blood | Glucose Transporter Type 2 - metabolism | Radiopharmaceuticals - metabolism | Insulin-Secreting Cells - metabolism | Liver - diagnostic imaging | Nuclear Proteins - genetics | Insulin Secretion | Glucose Intolerance - genetics | Down-Regulation | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Insulin Resistance | Bile Acids and Salts - metabolism | Blood Glucose | Lipid Metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Cholesterol - metabolism | Gene Knockout Techniques | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Feces - chemistry | Energy Metabolism | Glucose - physiology | Radionuclide Imaging | Glucose - metabolism | Mice
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5. Full Text Postprandial macrophage-derived IL-1β stimulates insulin, and both synergistically promote glucose disposal and inflammation
by Dror, Erez and Dalmas, Elise and Meier, Daniel T and Wueest, Stephan and Thévenet, Julien and Thienel, Constanze and Timper, Katharina and Nordmann, Thierry M and Traub, Shuyang and Schulze, Friederike and Item, Flurin and Vallois, David and Pattou, Francois and Kerr-Conte, Julie and Lavallard, Vanessa and Berney, Thierry and Thorens, Bernard and Konrad, Daniel and Böni-Schnetzler, Marianne and Donath, Marc Y
Nature Immunology, ISSN 1529-2908, 02/2017, Volume 18, Issue 3, pp. 283 - 292
The deleterious effect of chronic activation of the 1L-1 beta system on type 2 diabetes and other metabolic diseases is well documented. However, a possible... 
ACTIVATION | OBESITY | PANCREATIC-ISLETS | EFFECTOR FUNCTION | DOUBLE-BLIND | RESISTANCE | BETA-CELL | NLRP3 INFLAMMASOME | IMMUNOLOGY | TYPE-2 DIABETES-MELLITUS | NALP3 INFLAMMASOME | Insulin-Secreting Cells - physiology | Macrophages - physiology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Sodium-Glucose Transporter 2 - metabolism | Humans | Mice, Inbred C57BL | Cells, Cultured | Inflammation - immunology | Interleukin-1beta - genetics | Mice, Knockout | Insulin - metabolism | Animals | Interleukin-1beta - metabolism | Diabetes Mellitus, Type 2 - immunology | Postprandial Period | Glucose - metabolism | Mice | Mice, Inbred BALB C
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6. Full Text Activating mutations in genes related to TCR signaling in angioimmunoblastic and other follicular helper T-cell-derived lymphomas
by Vallois, David and Dobay, Maria Pamela D and Morin, Ryan D and Lemonnier, François and Missiaglia, Edoardo and Juilland, Mélanie and Iwaszkiewicz, Justyna and Fataccioli, Virginie and Bisig, Bettina and Roberti, Annalisa and Grewal, Jasleen and Bruneau, Julie and Fabiani, Bettina and Martin, Antoine and Bonnet, Christophe and Michielin, Olivier and Jais, Jean-Philippe and Figeac, Martin and Bernard, Olivier A and Delorenzi, Mauro and Haioun, Corinne and Tournilhac, Olivier and Thome, Margot and Gascoyne, Randy D and Gaulard, Philippe and De Leval, Laurence
Blood, ISSN 0006-4971, 09/2016, Volume 128, Issue 11, pp. 1490 - 1502
Angioimmunoblastic T-cell lymphoma (AITL) and other lymphomas derived from follicular T-helper cells (TFH) represent a large proportion of peripheral T-cell... 
STAT3 MUTATIONS | PROLYMPHOCYTIC LEUKEMIA | NK CELLS | LANDSCAPE | RECURRENT MUTATIONS | KINASE | MISSENSE MUTATION | SEZARY-SYNDROME | HEMATOLOGY | EXPRESSION | RHOA
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7. Full Text Hepatic glucose sensing is required to preserve [beta] cell glucose competence
by Seyer, Pascal and Vallois, David and Poitry-Yamate, Carole and Schuz, Frederic and Metref, Salima and Tarussio, David and Maechler, Pierre and Staels, Bart and Lanz, Bernard and Grueter, Rolf and Decaris, Julie and Turner, Scott and da Costa, Anabela and Preitner, Frederic and Minehira, Kaori and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, p. 1662
  Liver glucose metabolism plays a central role in glucose homeostasis and may also regulate feeding and energy expenditure. Here we assessed the impact of... 
Glucose metabolism | Pancreatic beta cells | Genetic aspects | Research | Gene expression | Liver
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8. Full Text Nervous glucose sensing regulates postnatal [beta] cell proliferation and glucose homeostasis
by Tarussio, David and Metref, Salima and Seyer, Pascal and Mounien, Lourdes and Vallois, David and Magnan, Christophe and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, p. 413
Physiological aspects | Homeostasis | Nervous system | Pancreatic beta cells | Research
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9. Nervous glucose sensing regulates postnatal beta cell proliferation and glucose homeostasis
by Tarussio, David and Metref, Salima and Seyer, Pascal and Mounien, Lourdes and Vallois, David and Magnan, Christophe and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 413 - 413
How glucose sensing by the nervous system impacts the regulation of beta cell mass and function during postnatal development and throughout adulthood is... 
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10. Full Text Gluco-incretins regulate beta-cell glucose competence by epigenetic silencing of Fxyd3 expression
by Vallois, David and Niederhäuser, Guy and Ibberson, Mark and Nagaray, Vini and Marselli, Lorella and Marchetti, Piero and Chatton, Jean-Yves and Thorens, Bernard and Diabetes - Islet Patophysiology and Lund University and Diabetes - öpatofysiologi and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e103277
Background/Aims: Gluco-incretin hormones increase the glucose competence of pancreatic beta-cells by incompletely characterized mechanisms. Methods: We... 
TYPE-2 DIABETIC-PATIENTS | PROTEIN | GLUCAGON-LIKE PEPTIDE-1 | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | INSULIN-SECRETION | 7-36 AMIDE | RECEPTOR | CARDIAC NA+/CA2+ EXCHANGER | PROLIFERATION | GASTRIC-INHIBITORY POLYPEPTIDE | Animals, Newborn | Gene Silencing - drug effects | Membrane Proteins - genetics | Humans | Mice, Inbred C57BL | Cells, Cultured | Incretins - metabolism | Neoplasm Proteins - metabolism | Promoter Regions, Genetic - drug effects | Eating - physiology | Mice, Knockout | Insulin-Secreting Cells - metabolism | Animals | Insulin-Secreting Cells - drug effects | Incretins - pharmacology | Glucose - metabolism | Epigenesis, Genetic - drug effects | Membrane Proteins - metabolism | Mice | Neoplasm Proteins - genetics | DNA Methylation - drug effects | Epigenetic inheritance | Pancreatic beta cells | Promoters (Genetics) | Analysis | Glucose | Methylation | Gene expression | Dextrose | Diabetes therapy | Nutrition | Secretion | Diabetes mellitus | Hormones | Kinases | Insulin | Calcium influx | Depolarization | Beta cells | Rodents | Imprinting | DNA methylation | Epigenetics | Membrane potential | Pancreas | Diabetes mellitus (non-insulin dependent) | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
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11. Full Text Postprandial macrophage-derived IL-1[beta] stimulates insulin, and both synergistically promote glucose disposal and inflammation
by Dror, Erez and Dalmas, Elise and Meier, Daniel T and Wueest, Stephan and Thevenet, Julien and Thienel, Constanze and Timper, Katharina and Nordmann, Thierry M and Traub, Shuyang and Schulze, Friederike and Item, Flurin and Vallois, David and Pattou, Francois and Kerr-Conte, Julie and Lavallard, Vanessa and Berney, Thierry and Thorens, Bernard and Konrad, Daniel and Boni-Schnetzler, Marianne and Donath, Marc Y
Nature Immunology, ISSN 1529-2908, 03/2017, Volume 18, Issue 3, p. 283
  The deleterious effect of chronic activation of the IL-1β system on type 2 diabetes and other metabolic diseases is well documented. However, a possible... 
Type 2 diabetes | Glucose metabolism | Obesity | Physiological aspects | Inflammation | Glucose | Macrophages | Insulin | Dextrose
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12. Full Text COUP-TFII controls mouse pancreatic β-cell mass through GLP-1-β-catenin signaling pathways
by Boutant, Marie and Ramos, Oscar Henrique Pereira and Tourrel-Cuzin, Cécile and Movassat, Jamileh and Ilias, Anissa and Vallois, David and Planchais, Julien and Pégorier, Jean-Paul and Schuit, Frans and Petit, Patrice X and Bossard, Pascale and Maedler, Kathrin and Grapin-Botton, Anne and Vasseur-Cognet, Mireille
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e30847
Background: The control of the functional pancreatic beta-cell mass serves the key homeostatic function of releasing the right amount of insulin to keep blood... 
INSULIN | APOPTOSIS | GLUCOSE-HOMEOSTASIS | CHRONIC EXPOSURE | NEOGENESIS | GLUCAGON-LIKE PEPTIDE-1 | MULTIDISCIPLINARY SCIENCES | GROWTH | GENE-EXPRESSION | CATENIN | TRANSCRIPTION FACTOR-II | Embryo, Mammalian | Cell Count | Humans | Glucagon-Like Peptide 1 - physiology | Male | COUP Transcription Factor II - physiology | Glucagon-Like Peptide 1 - genetics | Insulin-Secreting Cells - metabolism | Organ Size - genetics | Female | Insulin-Secreting Cells - cytology | beta Catenin - physiology | Animals, Newborn | Glucagon-Like Peptide 1 - metabolism | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Pancreas - drug effects | Rats | Mice, Transgenic | Signal Transduction - genetics | Pancreas - metabolism | Pancreas - embryology | beta Catenin - metabolism | beta Catenin - genetics | Organ Size - drug effects | Animals | Insulin-Secreting Cells - drug effects | Signal Transduction - drug effects | COUP Transcription Factor II - genetics | Models, Biological | Signal Transduction - physiology | Mice | COUP Transcription Factor II - metabolism | Pancreas - growth & development | Cell proliferation | Neonates | Transcription factors | Phosphorylation | Wnt protein | Glucagon | Laboratories | Genes | Lymphocytes T | Biology | Feedback loops | Glucose | Cyclin D1 | Homeobox | β-catenin | Signal transduction | Rodents | Extracellular matrix | Pancreas | Bioinformatics | Glucagon-like peptide 1 | Sugar | Hypertension | Ovalbumin | Metabolism | Gene expression | Cell differentiation | Epithelium | Insulin | Ablation | Glucose tolerance | Beta cells | Pathology | Intolerance | Signaling | Cell number | Insulin resistance | Control theory | Diabetes | Liver cirrhosis | Endocrinology | Apoptosis | Cancer
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13. Full Text Integrative clinicopathological and molecular analyses of angioimmunoblastic T-cell lymphoma and other nodal lymphomas of follicular helper T-cell origin
by Dobay, Maria Pamela and Lemonnier, Francois and Missiaglia, Edoardo and Bastard, Christian and Vallois, David and Jais, Jean-Philippe and Scourzic, Laurianne and Dupuy, Aurélie and Fataccioli, Virginie and Pujals, Anais and Parrens, Marie and Le Bras, Fabien and Rousset, Thérèse and Picquenot, Jean-Michel and Martin, Nadine and Haioun, Corinne and Delarue, Richard and Bernard, Olivier A and Delorenzi, Mauro and de Leval, Laurence and Gaulard, Philippe
Haematologica, ISSN 0390-6078, 03/2017, Volume 102, Issue 4, pp. e148 - e151
HEMATOLOGY | MUTATIONS | FEATURES | Online Only
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14. Full Text RNA fusions involvingCD28are rare in peripheral T-cell lymphomas and concentrate mainly in those derived from follicular helper T cells
by Vallois, David and Dupuy, Aurélie and Lemonnier, Francois and Allen, George and Missiaglia, Edoardo and Fataccioli, Virginie and Ortonne, Nicolas and Clavert, Aline and Delarue, Richard and Rousselet, Marie-Christine and Fabiani, Bettina and Llamas-Gutierrez, Francisco and Ogawa, Seishi and Thome, Margot and Ko, Young Hyeh and Kataoka, Keisuke and Gaulard, Philippe and de Leval, Laurence
Haematologica, ISSN 0390-6078, 08/2018, Volume 103, Issue 8, pp. e360 - e363
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15. Full Text RNA fusions involving CD28 are rare in peripheral t-cell lymphomas and concentrate mainly in those derived from follicular helper T cells
by Vallois, David and Dupuy, Aurélie and Lemonnier, Francois and Allen, George and Missiaglia, Edoardo and Fataccioli, Virginie and Ortonne, Nicolas and Clavert, Aline and Delarue, Richard and Rousselet, Marie-Christine and Fabiani, Bettina and Llamas-Gutierrez, Francisco and Ogawa, Seishi and Thome, Margot and Ko, Young Hyeh and Kataoka, Keisuke and Gaulard, Philippe and De Leval, Laurence
Haematologica, ISSN 0390-6078, 07/2018, Volume 103, Issue 8, pp. e360 - e363
CTLA4-CD28 GENE FUSION | MUTATIONS | SEZARY-SYNDROME | HEMATOLOGY | DIVERSE TYPES | RHOA | Online Only
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16. Hepatic glucose sensing is required to preserve beta cell glucose competence
by Seyer, Pascal and Vallois, David and Poitry-Yamate, Carole and Schuetz, Frederic and Metref, Salima and Tarussio, David and Maechler, Pierre and Staels, Bert and Lanz, Bernard and Grueter, Rolf and Decaris, Julie and Turner, Scott and da Costa, Anabela and Preitner, Frederic and Minehira, Kaori and Foretz, Marc and Thorens, Bernard
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1662 - 1662
Liver glucose metabolism plays a central role in glucose homeostasis and may also regulate feeding and energy expenditure. Here we assessed the impact of... 
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17. Du neuf dans l'oncogenèse moléculaire des lymphomes: rôle du gène SETD2 dans les lymphomes T de l'intestin
by Vallois, David and Roberti, Annalisa and Bisig, Bettina and Gaulard, Philippe and De Leval, Laurence
Medecine/Sciences, ISSN 0767-0974, 05/2017, Volume 33, Issue 5, pp. 469 - 473
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18. Full Text PPAR[beta]/[delta] affects pancreatic [beta] cell mass and insulin secretion in mice.(Research article)(peroxisome proliferator-activated receptors)
by Iglesias, Jose and Barg, Sebastian and Vallois, David and Lahiri, Shawon and Roger, Catherine and Yessoufou, Akadiri and Pradevand, Sylvain and McDonald, Angela and Bonal, Claire and Reimann, Frank and Gribble, Fiona and Debril, Marie-Bernard and Metzger, Daniel and Chambon, Pierre and Herrera, Pedro and Rutter, Guy A and Prentki, Marc and Thorens, Bernard and Wahli, Walter
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, p. 4105
PPAR[beta]/[delta] protects against obesity by reducing dyslipidemia and insulin resistance via effects in muscle, adipose tissue, and liver. However, its... 
Glucose metabolism | Pancreatic beta cells | Genetic aspects | Dosage and administration | Research | Gene expression | Insulin
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19. Full Text PPARβ/δ affects pancreatic β cell mass and insulin secretion in mice
by Iglesias, José and Barg, Sebastian and Vallois, David and Lahiri, Shawon and Roger, Catherine and Yessoufou, Akadiri and Pradevand, Sylvain and McDonald, Angela and Bonal, Claire and Reimann, Frank and Gribble, Fiona and Debril, Marie-Bernard and Metzger, Daniel and Chambon, Pierre and Herrera, Pedro and Rutter, Guy A and Prentki, Marc and Thorens, Bernard and Wahli, Walter and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för medicinsk cellbiologi
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4105 - 4117
PPAR beta/delta protects against obesity by reducing dyslipidemia and insulin resistance via effects in muscle, adipose tissue, and liver. However, its... 
MEDICINE, RESEARCH & EXPERIMENTAL | GRANULE DYNAMICS | 2ND-PHASE | DIFFERENTIAL EXPRESSION | RAB3 PROTEINS | PROTEIN-KINASE-D | DELTA | IN-VIVO | ACID OXIDATION | ACTIN NETWORK | ACTIVATED RECEPTOR-BETA/DELTA | Protein Kinase C - genetics | PPAR-beta - metabolism | Actins - metabolism | Glucose - genetics | Male | Gene Expression Profiling | PPAR-beta - genetics | Exocytosis - physiology | Actins - genetics | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Mice, Mutant Strains | Protein Kinase C - metabolism | Glucose - metabolism | Female | Golgi Apparatus - metabolism | Insulin-Secreting Cells - cytology | Mice | PPAR delta - metabolism | Insulin - genetics | Insulin Secretion | PPAR delta - genetics | Golgi Apparatus - genetics | Cell and Molecular Biology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Cell- och molekylärbiologi | Medicinska och farmaceutiska grundvetenskaper
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20. Molecular oncogenesis of lymphomas: role of the SETD2 gene in intestinal T-cell lymphomas
by Vallois, David and Roberti, Annalisa and Bisig, Bettina and Gaulard, Philippe and de Leval, Laurence
Medecine sciences : M/S, 05/2017, Volume 33, Issue 5, p. 469
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