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Breast Cancer Research, ISSN 1465-5411, 09/2015, Volume 17, Issue 1, p. 122
Hypoxia is a well-characterized driver of aggressive cancer phenotypes, including metastasis. Accumulating evidence suggests that, in addition to having local... 
BREAST-CANCER | ZOLEDRONIC ACID | METAANALYSIS | ADJUVANT THERAPY | ONCOLOGY | Bone and Bones - pathology | Cell Hypoxia - physiology | Breast Neoplasms - pathology | Neoplasm Metastasis - pathology | Humans | Female | Tumor Microenvironment - physiology | Bone Neoplasms - pathology | Development and progression | Metastasis | Viewpoint
Journal Article
Cancer Cell, ISSN 1535-6108, 10/2013, Volume 24, Issue 4, pp. 410 - 421
Journal Article
Nature, ISSN 0028-0836, 04/2015, Volume 520, Issue 7547, pp. 368 - 372
Drug resistance invariably limits the clinical efficacy of targeted therapy with kinase inhibitors against cancer(1,2). Here we show that targeted therapy with... 
CNS CELL-TYPES | MELANOMA | METASTASIS | MICROENVIRONMENT | TRANSLATIONAL PROFILING APPROACH | MULTIDISCIPLINARY SCIENCES | RAF INHIBITOR RESISTANCE | ACQUIRED-RESISTANCE | KINASE INHIBITORS | DRUG-RESISTANCE | CANCER CHEMORESISTANCE | Lung Neoplasms - drug therapy | Adenocarcinoma - pathology | Clone Cells - drug effects | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Proto-Oncogene Proteins c-fos - deficiency | Adenocarcinoma - metabolism | Neoplasm Metastasis - drug therapy | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Female | Proto-Oncogene Proteins c-akt - metabolism | Melanoma - metabolism | Tumor Microenvironment - drug effects | Cell Survival - drug effects | Melanoma - pathology | Down-Regulation - drug effects | Enzyme Activation - drug effects | Adenocarcinoma - drug therapy | Disease Progression | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Cell Movement - drug effects | Lung Neoplasms - secretion | Melanoma - secretion | Animals | Clone Cells - pathology | Metabolome - drug effects | Signal Transduction - drug effects | Neoplasm Metastasis - pathology | Protein Kinase Inhibitors - therapeutic use | Melanoma - drug therapy | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Adenocarcinoma - secretion | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Drug Resistance, Neoplasm - drug effects | Antimitotic agents | Pharmaceutical research | Care and treatment | Oncology, Experimental | Dosage and administration | Research | Drug therapy | Drug resistance | Antineoplastic agents | Tumors | Cancer | Melanoma | Mutation | Metastasis | Kinases | Cancer therapies
Journal Article
Molecular Oncology, ISSN 1574-7891, 01/2017, Volume 11, Issue 1, pp. 79 - 96
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2013, Volume 19, Issue 1, pp. 50 - 56
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2009, Volume 41, Issue 8, pp. 885 - 890
Journal Article
Cancer discovery, ISSN 2159-8274, 01/2019, Volume 9, Issue 1, pp. 22 - 24
Circulating tumor cells (CTC) are the source of metastases, but only an infi nitesimal fraction of them eventually succeed in colonizing a distant organ. New... 
CANCER PATIENTS | ONCOLOGY
Journal Article