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Journal of Physics G: Nuclear and Particle Physics, ISSN 0954-3899, 11/2004, Volume 30, Issue 11, pp. 1517 - 1763
ALICE is a general-purpose heavy-ion experiment designed to study the physics of strongly interacting matter and the quark-gluon plasma in nucleus-nucleus... 
BOSE-EINSTEIN CORRELATIONS | THERMAL HADRON-PRODUCTION | DYNAMICAL PARTON DISTRIBUTIONS | MONTE-CARLO MODEL | QUARK-GLUON PLASMA | HEAVY-ION COLLISIONS | TOTAL CROSS-SECTION | PHYSICS, NUCLEAR | NUCLEUS-NUCLEUS COLLISIONS | PLUS PB COLLISIONS | RADIATIVE ENERGY-LOSS | PHYSICS, PARTICLES & FIELDS
Journal Article
Radiation Protection Dosimetry, ISSN 0144-8420, 03/2014, Volume 163, Issue 2, pp. 141 - 147
Journal Article
STEM CELLS, ISSN 1066-5099, 09/2014, Volume 32, Issue 9, pp. 2373 - 2385
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e89232 - e89232
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 37, pp. 14058 - 14063
NF-κB is constitutively activated in primary human thyroid tumors, particularly in those of anaplastic type. The inhibition of NF-κB activity in the human... 
Cell growth | Thyroid cancer | Epithelial cells | Cell lines | Neutrophils | Small interfering RNA | Iron | Tumors | Apoptosis | Cancer | IRON UPTAKE | EPITHELIAL-CELLS | METABOLISM | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | CARCINOMAS | CANCER | EXPRESSION | PROGRESSION | MOLECULAR-MECHANISMS | INNATE IMMUNITY | Biological Sciences
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e70909 - e70909
APE1/Ref-1 is a main regulator of cellular response to oxidative stress via DNA-repair function and co-activating activity on the NF-κB transcription factor.... 
Propionates - pharmacology | Interleukin-8 - genetics | Promoter Regions, Genetic | Gene Expression | Liver Neoplasms - genetics | Interleukin-8 - biosynthesis | Humans | Transcriptional Activation - drug effects | NF-kappa B - metabolism | Hep G2 Cells | Benzoquinones - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | DNA-(Apurinic or Apyrimidinic Site) Lyase - genetics | DNA-(Apurinic or Apyrimidinic Site) Lyase - metabolism | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - metabolism | Oxidation-Reduction - drug effects | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Fatty Acids - metabolism | Carcinoma, Hepatocellular - metabolism | Oxidative stress | Toxicity | Pathogenesis | Liver | Trafficking | Genotoxicity | Chemoresistance | Cytotoxicity | Inflammatory response | Alcohol | Lipids | Activation | DNA repair | Cancer therapies | Accumulation | Blockage | Interleukin 6 | Proteins | Liver cancer | Rodents | Tumor necrosis factor-TNF | Inhibition | Damage accumulation | Interleukin 8 | Deoxyribonucleic acid--DNA | Enzyme-linked immunosorbent assay | NF-κB protein | Liver diseases | Cytokines | Medical treatment | Interleukin 12 | Tumor cell lines | Tumor necrosis factor-α | Gene expression | Fatty acids | Chemotherapy | Overexpression | Hepatocytes | Plasmids | Pancreatic cancer | Cell lines | Tumors | Cancer | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, pp. e0143289 - e0143289
Objective Human Hepatocellular Carcinoma (HCC) is the fifth most frequent neoplasm worldwide and the most serious complication of long-standing chronic liver... 
LIVER-CANCER | CELLS | REACTIVE OXYGEN | BASE EXCISION-REPAIR | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | TIME RT-PCR | FACTOR-I | HUMAN HEPATOCELLULAR-CARCINOMA | APURINIC/APYRIMIDINIC ENDONUCLEASE | EXPRESSION | Liver Neoplasms - genetics | Humans | Middle Aged | RNA, Messenger - genetics | Oxidative Stress - genetics | bcl-2-Associated X Protein - metabolism | Apoptosis - genetics | Hepatocytes - pathology | Male | Up-Regulation - genetics | Hepatocytes - metabolism | RNA, Messenger - metabolism | DNA-(Apurinic or Apyrimidinic Site) Lyase - genetics | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Female | Liver Neoplasms - pathology | Transcription, Genetic | Carcinoma, Hepatocellular - metabolism | Oxidative stress | Transcription factors | Physiological aspects | Development and progression | Genetic aspects | Research | Hepatoma | Apoptosis | Regulations | Deregulation | Bax protein | Transcription | Liver | Gene regulation | Science | Hepatocellular carcinoma | Kinases | Tissues | Liver cancer | Oxidation resistance | Rodents | Hepatology | Surgery | Localization | Deoxyribonucleic acid--DNA | Stresses | Free radicals | Liver diseases | Inflammation | Gene expression | Survival | Stress | Studies | Cirrhosis | Overexpression | Hepatocytes | Hedgehog protein | Medical prognosis | Endonuclease | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 05/2016, Volume 55, Issue 5, pp. 688 - 704
The apurinic/apyrimidinic endonuclease 1 (APE1) is a protein central to the base excision DNA repair pathway and operates in the modulation of gene expression... 
combination therapy | NPM1 | small molecule | protein/protein interaction | APE1 | Protein/protein interaction | Small molecule | Combination therapy | APE1/REF-1 | CRITICAL LYSINE RESIDUES | protein interaction | RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEPATOCELLULAR-CARCINOMA | PROTEIN INTERACTIONS | NUCLEOPHOSMIN | ONCOLOGY | protein | N-TERMINAL DOMAIN | DNA-REPAIR | EXPRESSION | Neoplasms - metabolism | Cell Survival - drug effects | Small Molecule Libraries - pharmacology | Humans | Nuclear Proteins - metabolism | High-Throughput Screening Assays | DNA-(Apurinic or Apyrimidinic Site) Lyase - metabolism | MCF-7 Cells | Nuclear Proteins - antagonists & inhibitors | Protein Binding - drug effects | Cell Line, Tumor | Female | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | DNA-(Apurinic or Apyrimidinic Site) Lyase - antagonists & inhibitors | HeLa Cells | Neoplasms - pathology | Proteins | Chemotherapy | Pharmacology | DNA repair | Tumors | Cell proliferation | Drugs | Surgical implants | Ovarian carcinoma | Liver | High-throughput screening | Genomes | Drug development | Assaying | Ovarian cancer | Anticancer properties | Biomedical materials | Antitumor agents | Bioactive compounds | Modulation | Biocompatibility | Localization | Deoxyribonucleic acid--DNA | Cell survival | Aggressive behavior | Preservation | Maturation | Tumor cells | rRNA | Light emitting diodes | Chemiluminescence | Gene expression | Screening | Inhibitors | Medical prognosis | Cells (biology) | Aberration | Position (location) | Protein interaction | Endonuclease | Cancer | Index Medicus
Journal Article
Journal Article
Intensive Care Medicine, ISSN 0342-4642, 1/2018, Volume 44, Issue 1, pp. 22 - 37
Journal Article