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Journal Article
Molecular Biology Reports, ISSN 0301-4851, 12/2016, Volume 43, Issue 12, pp. 1359 - 1369
Journal Article
Endocrine Research, ISSN 0743-5800, 07/2019, Volume 44, Issue 3, pp. 87 - 102
Purpose: It has been proposed that DHEA influences bone formation through, bioconversion to 17β-estradiol; however, DHEA is converted to Δ5-androstenediol... 
Estrogen receptors | androgen receptors | Δ5-androstenediol | bone | dehydroepiandrosterone
Journal Article
BMC musculoskeletal disorders, ISSN 1471-2474, 2014, Volume 15, Issue 1, pp. 400 - 400
Journal Article
Journal Article
Journal Article
Experimental and Therapeutic Medicine, ISSN 1792-0981, 12/2017, Volume 14, Issue 6, pp. 5464 - 5472
MicroRNAs (miRNAs or miRs) are a class of short non-coding RNAs that serve an important regulatory role in living organisms. These molecules are associated... 
microRNAs | Osteoporosis | Osteoclasts | Monocytes | Expression | MEDICINE, RESEARCH & EXPERIMENTAL | monocytes | NETWORK | expression | ALPHA | osteoporosis | PREDICTION | INTERFERON REGULATORY FACTOR-8 | osteoclasts | GENE-EXPRESSION | BONE | OSTEOCLAST DIFFERENTIATION
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, p. e0143421
Journal Article
BMC Medical Genetics, ISSN 1471-2350, 05/2017, Volume 18, Issue 1, pp. 46 - 6
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0150383
Idiopathic pulmonary fibrosis (IPF) is a progressive and lethal disease of unknown etiology. A growing body of evidence indicates that it may result from an... 
PATHOGENESIS | MATRIX METALLOPROTEINASES | LUNG FIBROSIS | MULTIDISCIPLINARY SCIENCES | EMPHYSEMA | INTERSTITIAL PNEUMONIA | MYOFIBROBLASTS | NEUTROPHIL ELASTASE | GUINEA-PIGS | IDIOPATHIC PULMONARY-FIBROSIS | OCCUPATIONAL-EXPOSURE | Smoking - adverse effects | Idiopathic Pulmonary Fibrosis - genetics | Epithelial Cells - metabolism | Rats, Wistar | Humans | Transcriptome | Integrins - genetics | Male | Integrins - metabolism | Idiopathic Pulmonary Fibrosis - metabolism | Idiopathic Pulmonary Fibrosis - etiology | Pulmonary Alveoli - metabolism | Epithelial-Mesenchymal Transition | Pulmonary Alveoli - cytology | Antigens, Neoplasm - metabolism | Epithelial Cells - cytology | Wnt Signaling Pathway | Fibroblasts - metabolism | Antigens, Neoplasm - genetics | Cell Line | Pulmonary Alveoli - pathology | Signal Transduction | Smoke - adverse effects | Gene Expression Regulation | Epithelial Cells - pathology | Fibroblasts - pathology | Animals | Transforming Growth Factor beta - genetics | Idiopathic Pulmonary Fibrosis - pathology | Fibroblasts - cytology | Mice | Tobacco - adverse effects | Transforming Growth Factor beta - metabolism | Physiological aspects | Genetic aspects | Research | Gene expression | Epithelial cells | Smoking | Cell culture | Deregulation | Wnt protein | Genes | Activation | Smoke | Risk factors | Ecological risk assessment | Signal transduction | Pathways | Etiology | Rodents | Transcription activation | Fibroblasts | Extracellular matrix | Alveoli | Growth factors | Medical research | Secretion | Lung diseases | Exposure | Cigarettes | Epithelium | Environmental risk | Insulin | Signaling | Pulmonary fibrosis | DNA microarrays | Fibrosis | Aberration | Laboratory animals | Cigarette smoke
Journal Article