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Leukemia, ISSN 0887-6924, 11/2013, Volume 27, Issue 11, pp. 2165 - 2176
Journal Article
Immunity, ISSN 1074-7613, 09/2016, Volume 45, Issue 3, pp. 610 - 625
Journal Article
Journal Article
Leukemia, ISSN 0887-6924, 06/2019, Volume 33, Issue 9, pp. 2241 - 2253
Journal Article
Leukemia, ISSN 0887-6924, 01/2018, Volume 32, Issue 1, p. 61
T-cell acute lymphoblastic leukemia (T-ALL) results from leukemic transformation of T-cell precursors arrested at specific differentiation stages, including an... 
Transcription factors | Genetic aspects | Research | Acute lymphocytic leukemia | T cells | Risk factors | Subpopulations | Deregulation | Transformation | Acute lymphatic leukemia | Maturation | Transcription | Leukemia | Cortex | Nkx2.3 protein | T cell receptors | Lymphocytes T | Lymphatic leukemia | Cell surface | Thymus | Proteins | T-cell receptor | Immunogenetics | Tlx1 protein | Msx2 protein
Journal Article
Journal Article
The Journal of Molecular Diagnostics, ISSN 1525-1578, 01/2019, Volume 21, Issue 1, pp. 111 - 122
Celiac disease is a chronic inflammation of the small intestine with villous atrophy that can become refractory to a gluten-free diet. Two categories of... 
GAMMA | DESIGN | SPRUE | REFRACTORY CELIAC-DISEASE | RECEPTOR | CLASSIFICATION | T-CELL LYMPHOMA | TIME | INTESTINE | PATHOLOGY | ABERRANT INTRAEPITHELIAL LYMPHOCYTES
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2012, Volume 21, Issue 4, pp. 563 - 576
Journal Article
Leukemia, ISSN 0887-6924, 02/2013, Volume 27, Issue 2, pp. 305 - 314
Constitutively activated FLT3 signaling is common in acute myeloid leukemia, and is currently under evaluation for targeted therapy, whereas little data is... 
MOLECULAR-MECHANISM | PTK INHIBITORS | FLT3 | TYROSINE KINASE | ACUTE MYELOID-LEUKEMIA | DRUG-RESISTANCE | ACUTE LYMPHOBLASTIC LEUKEMIAS | CONSTITUTIVELY ACTIVATED FLT3 | ONCOLOGY | MUTATIONS | HEMATOLOGY | T-ALL | multitarget therapy | LINEAGE | receptor kinase | GENE-EXPRESSION SIGNATURES | fms-Like Tyrosine Kinase 3 - antagonists & inhibitors | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Humans | Middle Aged | Child, Preschool | Infant | Male | Young Adult | fms-Like Tyrosine Kinase 3 - genetics | Flow Cytometry | Polymerase Chain Reaction | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Tumor Cells, Cultured | Child | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Signal Transduction | Survival Rate | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - enzymology | Mutation - genetics | Receptor Protein-Tyrosine Kinases - metabolism | fms-Like Tyrosine Kinase 3 - metabolism | Drug Synergism | Drug Resistance, Neoplasm - genetics | Receptor Protein-Tyrosine Kinases - genetics | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Adolescent | Aged | Protein Kinase Inhibitors - pharmacology | DNA, Neoplasm - genetics | Gene mutations | Leukemia | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | T cells | Phosphotransferases
Journal Article
The Journal of Experimental Medicine, ISSN 0022-1007, 01/2014, Volume 211, Issue 9, pp. 1821 - 1832
textabstractV(D)J recombination of TCR loci is regulated by chromatin accessibility to RAG1/2 proteins, rendering RAG1/2 targeting a potentially important... 
RECOMBINATION SIGNAL SEQUENCES | TRANSCRIPTION FACTORS | ALLELIC EXCLUSION | ALPHA/DELTA LOCUS | MEDICINE, RESEARCH & EXPERIMENTAL | T-CELL DIFFERENTIATION | ADULT HEMATOPOIESIS | LYMPHOCYTE DEVELOPMENT | 12/23 RULE | V(D)J RECOMBINATION | GENE REARRANGEMENT | IMMUNOLOGY
Journal Article