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Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 863 - 875
Accumulation of unwanted/misfolded proteins in aggregates has been observed in airways of patients with cystic fibrosis (CF), a life-threatening genetic... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | BECLIN 1 | DOWN-REGULATION | ENDOPLASMIC-RETICULUM | MICE | GENE-TRANSFER | TISSUE TRANSGLUTAMINASE | NEURODEGENERATIVE DISEASES | CELL-DEATH | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Sequestosome-1 Protein | Humans | Transglutaminases - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Young Adult | Cystamine - therapeutic use | Inflammation - metabolism | Autophagy - genetics | Nasal Polyps - metabolism | Mice, Inbred CFTR | Respiratory Mucosa - cytology | Cystic Fibrosis - metabolism | Membrane Proteins - genetics | Mice, Transgenic | Protein Transport - genetics | Apoptosis Regulatory Proteins - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Models, Biological | Cystic Fibrosis - genetics | Transglutaminases - metabolism | Adolescent | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | GTP-Binding Proteins | Cystic Fibrosis - drug therapy | Protein Binding - physiology | Microtubule-Associated Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Salicylates - pharmacology | Epithelial Sodium Channels - genetics | Apoptosis Regulatory Proteins - genetics | Adult | Cystamine - pharmacology | Membrane Proteins - metabolism | Acetylcysteine - therapeutic use | Beclin-1 | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cell Line | Small Ubiquitin-Related Modifier Proteins - metabolism | Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Mice, Inbred Strains | Nasal Polyps - drug therapy | Antioxidants - therapeutic use | Animals | Acetylcysteine - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Organometallic Compounds - pharmacology | Complications and side effects | Active oxygen | Gene mutations | Lung diseases | Physiological aspects | Cystic fibrosis | Genetic aspects | Research | Health aspects | Risk factors
Journal Article
Autophagy, ISSN 1554-8627, 11/2012, Volume 8, Issue 11, pp. 1657 - 1672
Channel activators (potentiators) of cystic fibrosis (CF) transmembrane conductance regulator (CFTR), can be used for the treatment of the small subset of CF... 
cystamine | CFTR potentiators | autophagy | cystic fibrosis | therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Therapy | Cystic fibrosis | Autophagy | Cystamine | GAMMA DOWN-REGULATION | OXIDATIVE STRESS | F508DEL-CFTR | PLASMA-MEMBRANE | INTESTINAL EPITHELIAL-CELLS | CELL BIOLOGY | PROTEIN-QUALITY CONTROL | MUTATION | TISSUE TRANSGLUTAMINASE | LUNG INFLAMMATION | CFTR | Inflammation - pathology | Epithelial Cells - metabolism | Sequestosome-1 Protein | Epithelial Cells - drug effects | Humans | Cystic Fibrosis - pathology | Male | Nasal Mucosa - pathology | Molecular Targeted Therapy | Autophagy - drug effects | Salicylates - pharmacology | Cystamine - therapeutic use | Organometallic Compounds - therapeutic use | Cystamine - pharmacology | Female | Cell Membrane - metabolism | Membrane Proteins - metabolism | Child | Cell Membrane - drug effects | Nasal Mucosa - drug effects | Beclin-1 | Epithelium - drug effects | Epithelium - pathology | Lung - pathology | Nasal Polyps - pathology | Epithelium - metabolism | Heat-Shock Proteins - metabolism | Epithelial Cells - pathology | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Salicylates - therapeutic use | Apoptosis Regulatory Proteins - metabolism | Animals | Lung - drug effects | Adolescent | Genistein - pharmacology | Lipopolysaccharides - pharmacology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cystic Fibrosis - drug therapy | Organometallic Compounds - pharmacology | Translational Research Paper
Journal Article
Journal Article
Cell Reports, ISSN 2211-1247, 05/2018, Volume 23, Issue 6, pp. 1767 - 1778
Journal Article
Oncotarget, ISSN 1949-2553, 07/2019, Volume 10, Issue 43, pp. 4492 - 4500
Cystic Fibrosis (CF) is the most frequent lethal monogenetic disease affecting humans. CF is characterized by mutations in cystic fibrosis transmembrane... 
Journal Article
Cell Reports, ISSN 2211-1247, 08/2017, Volume 20, Issue 7, pp. 1667 - 1680
The interleukin 17 (IL-17) cytokine and receptor family is central to antimicrobial resistance and inflammation in the lung. Mice lacking IL-17A, IL-17F, or... 
IL-17F/IL-17RC axis | Th17 immunity | allergy | ABPA | respiratory infections | INTERLEUKIN-17 RECEPTOR | IL-17 RECEPTOR | SEVERE ASTHMA | INFLAMMATION | PROSTATE-CANCER | DOUBLE-BLIND | CYSTIC-FIBROSIS | CHRONIC MUCOCUTANEOUS CANDIDIASIS | CUTTING EDGE | TH17 CELLS | CELL BIOLOGY | Lung - microbiology | Staphylococcal Infections - pathology | Humans | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Hypersensitivity - immunology | Receptors, Interleukin-17 - genetics | Receptors, Interleukin-17 - deficiency | Respiratory Mucosa - pathology | Respiratory Mucosa - microbiology | Aspergillosis - pathology | Hypersensitivity - microbiology | Respiratory Mucosa - immunology | Female | Hypersensitivity - genetics | Protein Isoforms - deficiency | Pseudomonas - immunology | Staphylococcal Infections - microbiology | Disease Models, Animal | Lung - pathology | Staphylococcal Infections - genetics | Aspergillus - immunology | Disease Susceptibility | Signal Transduction | Staphylococcal Infections - immunology | Mice, Inbred C57BL | Aspergillosis - genetics | Gene Expression Regulation | Interleukin-17 - genetics | Epithelial Cells - pathology | Interleukin-17 - deficiency | Pseudomonas Infections - microbiology | Mice, Knockout | Pseudomonas Infections - genetics | Pseudomonas Infections - immunology | Animals | Hypersensitivity - pathology | Epithelial Cells - immunology | Epithelial Cells - microbiology | Aspergillosis - microbiology | Pseudomonas Infections - pathology | Staphylococcus aureus - immunology | Mice | Protein Isoforms - immunology | Lung - immunology | Aspergillosis - immunology | Protein Isoforms - genetics
Journal Article
Journal Article
Gut, ISSN 0017-5749, 03/2010, Volume 59, Issue 3, pp. 311 - 319
BackgroundAn unresolved question in coeliac disease is to understand how some toxic gliadin peptides, in particular p31–43, can initiate an innate response and... 
ACTIVATION | ACID | PERMEABILITY | INFLAMMATION | DISEASE | CROSS-LINKING | ROSIGLITAZONE | GASTROENTEROLOGY & HEPATOLOGY | ASSOCIATION | T-CELLS | MODULATION
Journal Article
Aging, ISSN 1945-4589, 04/2019, Volume 11, Issue 7, pp. 2003 - 2019
In celiac disease (CD), an intolerance to dietary gluten/gliadin, antigenic gliadin peptides trigger an HLA-DQ2/DQ8-restricted adaptive Th1 immune response.... 
Celiac disease | Gluten peptides | Inflammation | Genistein | CFTR | OXIDATIVE STRESS | ACTIVATION | ELECTROGENIC CL-SECRETION | gluten peptides | EPITHELIAL STRESS | AUTOPHAGY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | genistein | inflammation | celiac disease | CYSTIC-FIBROSIS | GLUTEN SENSITIVITY | TISSUE TRANSGLUTAMINASE | T-CELLS
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 5/2013, Volume 190, Issue 10, pp. 5196 - 5206
We have previously reported that TLR4 signaling is increased in lipopolysaccharide (LPS) -stimulated Cystic Fibrosis (CF) macrophages (MΦs), contributing to... 
Journal Article
Pediatric Research, ISSN 0031-3998, 05/2012, Volume 71, Issue 5, pp. 532 - 538
INTRODUCTION: K562(S) agglutination has been used as a rapid and economic tool for the in vitro screening of the toxicity of cereal fractions and prolamins in... 
CONCANAVALIN | MUCOSA | OXIDATIVE STRESS | ACTIVATION | PEPTIDES | PROTEIN | CYTOSKELETON | INFLAMMATION | PEDIATRICS | INTESTINAL EPITHELIAL-CELLS | CELIAC-DISEASE | Gliadin - metabolism | Humans | Transglutaminases - metabolism | K562 Cells | Enzyme Activation | GTP-Binding Proteins | Cell Adhesion | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 03/2010, Volume 59, Issue 3, pp. 311 - 319
  Background An unresolved question in coeliac disease is to understand how some toxic gliadin peptides, in particular p31-43, can initiate an innate response... 
Celiac disease | Lysosomes | Physiological aspects | Development and progression | Natural immunity | Transglutaminases | Research | Inflammatory bowel disease | Oxidative stress | Disease | Peptides | Cytokines | Rodents | Cystic fibrosis | Inflammation | Gene expression | Small intestine
Journal Article
Pediatric Research, ISSN 0031-3998, 05/2012, Volume 71, Issue 5, pp. 529 - 529
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2014, Volume 193, Issue 10, pp. 5347 - 5349
Journal Article
ANNALS OF TRANSLATIONAL MEDICINE, ISSN 2305-5839, 12/2019, Volume 7, Issue Suppl 8, pp. S263 - S263
Journal Article