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Cell Death and Differentiation, ISSN 1350-9047, 01/2019, Volume 26, Issue 1, pp. 99 - 114
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 2018, Volume 26, Issue 5, pp. 877 - 889
RIPK1 is an essential downstream component of many pattern recognition and death receptors. RIPK1 can promote the activation of caspase-8 induced apoptosis and... 
NECROSIS-FACTOR RECEPTOR | ACTIVATION | HOMEOSTASIS | NECROPTOSIS | INHIBITION | PHOSPHORYLATION | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CASPASE-8 | FADD | CELL-DEATH | CELL BIOLOGY | Tumor necrosis factor receptors | Neonates | Splenomegaly | Caspase | c-FLIP protein | Lethality | Pattern recognition | Thymus | FADD protein | Caspase-8 | Clonal deletion | Intestine | Cell death | Death receptors | Apoptosis
Journal Article
Immunity, ISSN 1074-7613, 09/2018, Volume 49, Issue 3, pp. 379 - 381
The pore-forming protein GSDMD promotes cytokine release and induces pyroptotic cell death. In this issue of , document how GSDMD triggers potassium efflux to... 
DNA | IMMUNOLOGY | Interferon | Bacterial infections | Biological response modifiers | Cell death | Efflux | Neutrophils | Infections | Punches | Kinases | Immunity | Proteins | Studies | Sensors | Potassium | Deoxyribonucleic acid--DNA | Viral infections | Pore formation | Apoptosis
Journal Article
Journal Article
Journal of Cell Science, ISSN 0021-9533, 03/2019, Volume 132, Issue 5, p. jcs220996
Necroptosis is an inflammatory form of programmed cell death mediated by the pseudokinase mixed-lineage kinase domain-like protein (MLKL). Upon phosphorylation... 
MLKL | Necroptosis | Autophagy | LC3 | PROTEIN | PHOSPHORYLATION | FAMILY-MEMBERS | MIXED LINEAGE KINASE | DOMAIN-LIKE | TNF | CELL-DEATH | CELL BIOLOGY | NECROSIS | DOWNSTREAM | MEDIATES NECROPTOSIS
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 02/2017, Volume 95, Issue 2, pp. 129 - 130
The February 2017 issue contains a Special Feature on Necroptotic death signalling: evolution, mechanisms and disease relevance. In recent years, research into... 
GSDMD | APOPTOSIS | GASDERMIN D | IMMUNOLOGY | CASPASE-11 | PORES | PYROPTOTIC CELL-DEATH | CELL BIOLOGY
Journal Article
Cell, ISSN 0092-8674, 05/2014, Volume 157, Issue 5, pp. 1175 - 1188
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 03/2017, Volume 24, Issue 3, p. 481
  Peptido-mimetic inhibitor of apoptosis protein (IAP) antagonists (Smac mimetics (SMs)) can kill tumour cells by depleting endogenous IAPs and thereby... 
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 02/2018, Volume 25, Issue 2, p. 392
Soluble CD52 is a small glycoprotein that suppresses T-cell activation, but its effect on innate immune cell function is unknown. Here we demonstrate that... 
Shock | Bax protein | Activation | Lymphocytes T | Macrophages | Lipopolysaccharides | Proteins | Cell activation | Clonal deletion | Deletion | Toll-like receptors | Endotoxemia | Immune system | NF-κB protein | Cell survival | Cytokines | Dendritic cells | Secretion | Glycoprotein | Inflammation | T cell receptors | Mcl-1 protein | Signaling | Monocytes | Cell death | Receptor mechanisms | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2017, Volume 114, Issue 6, pp. E961 - E969
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 08/2015, Volume 135, Issue 8, pp. 1936 - 1939
Caspase-8 downregulation is observed in the epidermis of wounded skin, whereas permanent epidermal caspase-8 deletion causes chronic skin inflammation,... 
INTERLEUKIN-1 | APOPTOSIS | ACTIVATION | HOMEOSTASIS | NECROPTOSIS | DERMATITIS | KERATINOCYTES | MICE | NLRP3 INFLAMMASOME | EXPRESSION | DERMATOLOGY | Dermatitis - pathology | Cellular Microenvironment - physiology | Keratinocytes - pathology | Animals | Caspase 1 - physiology | Dermatitis - physiopathology
Journal Article