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Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2017, Volume 76, Issue Suppl 1, p. A19
BackgroundSynovitis-associated pain is an important aspect of arthritis pathology. Inflammatory mediators released by the synovium have been implicated in the... 
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2017, Volume 76, Issue Suppl 1, pp. A19 - A19
BackgroundSynovitis-associated pain is an important aspect of arthritis pathology. Inflammatory mediators released by the synovium have been implicated in the... 
Journal Article
Cellular Signalling, ISSN 0898-6568, 12/2017, Volume 40, p. 190
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1016/j.cellsig.2017.09.010... 
Interleukins | Transforming growth factors | Gene expression | Stem cells | Analysis | Genes | Bone morphogenetic proteins | Genetic transcription
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 01/2014, Volume 355, Issue 1, pp. 163 - 171
Lysyl hydroxylase 2b (LH2b) is known to increase pyridinoline cross-links, making collagen less susceptible to enzymatic degradation. Previously, we observed a... 
Journal Article
Tissue Engineering Part A, ISSN 1937-3341, 04/2011, Volume 17, Issue 7-8, pp. 1157 - 1167
Journal Article
Scientific Reports (Nature Publisher Group), 02/2017, Volume 7, p. 43164
To improve cartilage formation by bone marrow-derived mesenchymal stem cells (BMSCs), the signaling mechanism governing chondrogenic differentiation requires... 
Phosphorylation | Immunology | Mesenchyme | Stem cells | Bone marrow | Cell differentiation
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 4, pp. 2629 - 2629
Journal Article
Developmental Biology, ISSN 0012-1606, 02/2014, Volume 386, Issue 2, pp. 395 - 407
Journal Article
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 01/2014, Volume 355, Issue 1, pp. 163 - 171
Lysyl hydroxylase 2b (LH2b) is known to increase pyridinoline cross-links, making collagen less susceptible to enzymatic degradation. Previously, we observed a... 
PATHWAYS | HUMAN-DISEASE | INDUCTION | Synovium | OSTEOPHYTE FORMATION | OVEREXPRESSION | TGF-beta | GROWTH-FACTOR-BETA | Fibrosis | Lysyl hydroxylase 2b | Osteoarthritis | EXPRESSION | PYRIDINOLINE CROSS-LINKS | TRANSIENT FIBROSIS | TGF-BETA-1 | CELL BIOLOGY | Collagen | Analysis | Heat shock proteins | Bone morphogenetic proteins | Hydroxylases | Transforming growth factors | Gene expression
Journal Article
Rheumatology (United Kingdom), ISSN 1462-0324, 03/2017, Volume 56, Issue 3, pp. 351 - 361
Objective. A crucial feature of OA is cartilage degradation. This process is mediated by pro-inflammatory cytokines, among other factors, via induction of... 
cytokines | IL37 | inflammation | osteoarthritis | cartilage | INTERLEUKIN-1 | MATRIX-METALLOPROTEINASE | DOWN-REGULATION | HUMAN ARTICULAR CHONDROCYTES | RHEUMATOLOGY | TUMOR-NECROSIS-FACTOR | CYTOKINE PRODUCTION | II COLLAGEN | HUMAN OSTEOARTHRITIC CARTILAGE | GENE-EXPRESSION | PROTEOGLYCAN CATABOLISM | Immunohistochemistry | Interleukin-1beta - drug effects | Interleukin-1 - genetics | Interleukin-8 - genetics | Interleukin-1beta - pharmacology | Adenoviridae | Humans | Interleukin-8 - drug effects | Matrix Metalloproteinase 3 - drug effects | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Disintegrins - genetics | Chondrocytes - drug effects | Interleukin-1beta - metabolism | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Chondrocytes - metabolism | Matrix Metalloproteinase 1 - genetics | RNA, Messenger - drug effects | Interleukin-1 - metabolism | Matrix Metalloproteinase 1 - drug effects | Disintegrins - metabolism | Interleukin-6 - genetics | Matrix Metalloproteinase 13 - genetics | Matrix Metalloproteinase 3 - metabolism | Matrix Metalloproteinase 13 - drug effects | Blotting, Western | Matrix Metalloproteinase 13 - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Disintegrins - drug effects | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 3 - genetics | Osteoarthritis
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 2002, Volume 169, Issue 1, pp. 507 - 514
Osteoarthritis has as main characteristics the degradation of articular cartilage and the formation of new bone at the joint edges, so-called osteophytes. In... 
RHEUMATOID-ARTHRITIS | POTENT INHIBITOR | GROWTH-FACTOR-BETA | MESSENGER-RNA | MURINE KNEE-JOINT | ARTICULAR-CARTILAGE | II RECEPTOR | MOUSE PATELLA | HUMAN BONE | IMMUNOLOGY | EXPRESSION | Immunohistochemistry | Recombinant Proteins - analysis | Chondrocytes - immunology | Protein-Serine-Threonine Kinases | Male | Matrix Metalloproteinase 1 - biosynthesis | Recombinant Proteins - biosynthesis | Transforming Growth Factor beta - biosynthesis | Tissue Inhibitor of Metalloproteinases - genetics | RNA, Messenger - biosynthesis | Transforming Growth Factor beta3 | Tissue Inhibitor of Metalloproteinases - biosynthesis | ADAMTS5 Protein | Chondrocytes - pathology | Transforming Growth Factor beta1 | Collagenases - genetics | Collagenases - biosynthesis | Solubility | Protein Isoforms - analysis | Blotting, Western | Cell Differentiation - immunology | Metalloendopeptidases - biosynthesis | Receptors, Transforming Growth Factor beta - biosynthesis | Mice | Metalloendopeptidases - genetics | Receptors, Transforming Growth Factor beta - physiology | Osteogenesis - immunology | Pichia - enzymology | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Chondrocytes - drug effects | Transforming Growth Factor beta - analysis | Receptor, Transforming Growth Factor-beta Type II | Transforming Growth Factor beta - antagonists & inhibitors | Osteoarthritis, Knee - immunology | Matrix Metalloproteinase 1 - genetics | Cartilage, Articular - chemistry | Procollagen N-Endopeptidase | Receptors, Transforming Growth Factor beta - analysis | Electrophoresis, Polyacrylamide Gel | Mice, Inbred C57BL | Osteogenesis - drug effects | ADAM Proteins | Arthritis, Experimental - immunology | Recombinant Proteins - pharmacology | ADAMTS4 Protein | Matrix Metalloproteinase 13 | Cartilage, Articular - pathology | Animals | Protein Isoforms - biosynthesis | Cell Differentiation - drug effects | Osteoarthritis, Knee - pathology | Cartilage, Articular - drug effects | Cartilage, Articular - immunology | Osteoarthritis, Knee - metabolism | Protein Isoforms - antagonists & inhibitors
Journal Article
Journal Article