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Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2009, Volume 54, Issue 1, pp. S10 - S19
Inflammatory processes are prominent in various types of human and experimental pulmonary hypertension (PH) and are increasingly recognized as major pathogenic... 
Cardiovascular | Internal Medicine | growth factors | inflammation | pulmonary vascular remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | SARCOMA-ASSOCIATED HERPESVIRUS | FACTOR EXPRESSION | ENDOTHELIAL-CELL APOPTOSIS | SYSTEMIC-SCLEROSIS | HIV-1 NEF | HEPATITIS-C VIRUS | PLASMINOGEN-ACTIVATOR | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | 5-HYDROXYTRYPTAMINE TRANSPORTER GENE | Chemokine CCL5 | Inflammation - pathology | Humans | Scleroderma, Systemic - pathology | Hypertension, Pulmonary - physiopathology | NFATC Transcription Factors - physiology | Chemokines, CX3C - physiology | Cytokines - physiology | Vascular Resistance - physiology | Animals | Scleroderma, Systemic - physiopathology | Antineoplastic Agents - pharmacology | Hypertension, Pulmonary - virology | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Inflammation - physiopathology | Hypertension | Tyrosine | Medical colleges | Lung diseases, Obstructive | Pharmacogenetics | Micropollutants | Dendritic cells | Antibodies | Development and progression | Inflammation | HIV (Viruses) | T cells | Macrophages | Endothelium | Polymerase chain reaction | Viral antibodies | Messenger RNA | Viral proteins | Hepatitis C | Vascular endothelial growth factor | Growth factors | Tumors | Platelet-derived growth factor | Endothelial growth factors | B cells | Pulmonary hypertension | Biomedical research | Cytokines | Pulmonary arteries | Migration | Recruitment | Proteins | Angiogenesis | Lymphocytes | Human immunodeficiency virus--HIV | Medical prognosis | Cell cycle | Chemokines
Journal Article
Chest, ISSN 0012-3692, 2009, Volume 135, Issue 3, pp. 794 - 804
Pulmonary arterial hypertension (PAH) is a deadly disease in which vasoconstriction and vascular remodeling both lead to a progressive increase in pulmonary... 
Pulmonary vascular resistance | Right ventricle | Pulmonary arterial hypertension
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 49, pp. 19725 - 19730
Acute myocardial infarction (AMI) initiates an intense inflammatory response that promotes cardiac dysfunction, cell death, and ventricular remodeling. The... 
Heart | Myocardial infarction | Granulation tissue | Receptors | Ischemia | Cell death | Myocardium | Small interfering RNA | Renovations | Endothelial cells | NLRP3 | Interleukin-1 | Pyroptosis | NALP3 | EXTRACELLULAR ATP | CELLS | interleukin-1 | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | RECEPTOR | pyroptosis | RESPONSES | DANGER SIGNALS | IN-VIVO | LIPOPOLYSACCHARIDE | Myocardial Infarction - genetics | Inflammasomes - metabolism | Apoptosis - drug effects | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | Myocardial Infarction - diagnostic imaging | Receptors, Purinergic P2X7 - genetics | Ventricular Remodeling | RNA Interference | Cytoskeletal Proteins - metabolism | Pyridoxal Phosphate - analogs & derivatives | Disease Models, Animal | Cell Line | Gene Expression | Echocardiography | Pyridoxal Phosphate - pharmacology | Myocardial Infarction - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Inflammasomes - genetics | Carrier Proteins - genetics | Myocytes, Cardiac - pathology | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Fluorescent Antibody Technique | Lipopolysaccharides - pharmacology | Myocytes, Cardiac - metabolism | Receptors, Purinergic P2X7 - metabolism | Mice | Oligomers | Care and treatment | Heart cells | Physiological aspects | Development and progression | Health aspects | Heart attack | Biological Sciences
Journal Article
American journal of respiratory and critical care medicine, ISSN 1535-4970, 2019, Volume 199, Issue 12, pp. 1458 - 1459
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 3, p. e0120157
..., Charles E. Chalfant 3,5,6,7, Norbert F. Voelkel 1,* Introduction Inflammatory cells have been thought to contribute to the pathobiology of pulmonary hypertension (PH... 
CELLS | CYCLOOXYGENASE-2 | INHIBITION | THROMBOXANE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ARTERIAL-HYPERTENSION | PROSTAGLANDIN | EXPRESSION | RAT LUNG | VASOCONSTRICTION | Hypoxia - drug therapy | Arachidonate 5-Lipoxygenase - genetics | Dinoprostone - biosynthesis | Leukotriene D4 - biosynthesis | Humans | Arterioles - enzymology | Prostaglandins F - antagonists & inhibitors | Male | Thromboxane B2 - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Lung - enzymology | Dinoprost - biosynthesis | Cyclooxygenase 2 - genetics | Prostaglandins F - biosynthesis | Hypertension, Pulmonary - drug therapy | Ventricular Function, Right - drug effects | Leukotriene D4 - antagonists & inhibitors | Hypertension, Pulmonary - enzymology | Pyrazoles - pharmacology | Lung - pathology | Hypoxia - enzymology | Arachidonate 5-Lipoxygenase - metabolism | Cyclooxygenase 2 Inhibitors - pharmacology | Diethylcarbamazine - pharmacology | Rats | Hypertension, Pulmonary - genetics | Inflammation | Pulmonary Artery - drug effects | Thromboxane B2 - biosynthesis | Arterioles - drug effects | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Arterioles - pathology | Hypoxia - genetics | Animals | Hypoxia - pathology | Lung - drug effects | Cyclooxygenase 2 - metabolism | Dinoprost - antagonists & inhibitors | Dinoprostone - antagonists & inhibitors | Lipoxygenase Inhibitors - pharmacology | Pulmonary Artery - pathology | COX-2 inhibitors | Research | Metabolites | Analysis | Pulmonary hypertension | Heart | Oxidative stress | Animal models | Pathogenesis | Arterioles | Critical care | Biochemistry | Kinases | Immunity | Antioxidants | Vascular diseases | Rodents | Arachidonate 5-lipoxygenase | Cardiology | Heart failure | Hypertension | Medical research | Pulmonary arteries | Lung diseases | Blood vessels | Medicine | Lungs | Hypoxia | Ventricle | Cyclooxygenase-2 | Lipoxygenase | Molecular biology | Cancer
Journal Article
The American journal of cardiology, ISSN 0002-9149, 2013, Volume 111, Issue 10, pp. 1394 - 1400
Journal Article