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by Yamagami, Keitaro and Kurogi, Ryota and Kurogi, Ai and Nishimura, Hiroyuki and Nishimura, Shinjitsu and Nishimura, Yasuaki and Nishimura, Kunihiro and Nishimura, Ataru and Onozuka, Daisuke and Ren, Nice and Kada, Akiko and Arimura, Koichi and Ido, Keisuke and Mizoguchi, Masahiro and Sakamoto, Seisaburo and Sakamoto, Tetsuya and Kayama, Takamasa and Suzuki, Nozomi and Suzuki, Michiyasu and Suzuki, Sachio and Suzuki, Hidenori and Suzuki, Ichiro and Suzuki, Susumu and Arai, Yoshinori and Arai, Hajime and Arai, Motohiro and Hagihara, Akihito and Iihara, Koji and Takigami, Masayoshi and Kamiyama, Kenji and Houkin, Kiyohiro and Nishi, Shougo and Yoshimoto, Tetsuyuki and Yoshimoto, Junpei and Kaneko, Sadao and Oka, Hirofumi and Oka, Koji and Ooyama, Hiroshi and Kamada, Kyousuke and Makino, Kenichi and Tokumitsu, Naoki and Sako, Kazuhiro and Izumi, Naoto and Nitta, Hisashi and Nitta, Kazumi and Ootaki, Masahumi and Isobe, Masanori and Nishiya, Mikio and Yamazaki, Takaaki and Mabuchi, Syouji and Mabuchi, Eiichiro and Ogasawara, Kuniaki and Kubo, Naohiko and Shimizu, Yukihiko and Saito, Hitoshi and Saito, Keiichi and Yamanome, Tatumi and Yoshino, Kimihiro and Yoshino, Atsuo and Fujitsuka, Mitsuyuki and Takami, Masaaki and Ohtaka, Hirotoshi and Hirano, Teruyuki and Shiokawa, Yosiaki and Okada, Takaharu and Kohno, Michihiro and Haraoka, Jou and Kawamura, Noriyoshi and Kawamura, Tadao and Isoshima, Akira and Yasue, Masaharu and Takayoshi Kobayashi, Mitsuhiko Hokari and Kawai, Kensuke and Maehara, Taketoshi and Noguchi, Makoto and Hoshino, Haruhiko and Hiyama, Hirofumi and Yoshida, Kensaku and Yoshida, Kazunari and Utsugi, Osamu and Takeda, Yasuaki and Takeda, Yuu and Tamaki, Kouichi and Karasudani, Hirohide and Urabe, Takao and Kobayashi, Nozomu and Kobayashi, Shiro and Nakamura, Yusaku and Nakamura, Kazuhito and Nakamura, Michio and Nakamura, Yoshinari and Koguchi, Yorio and Ono, Junichi and Suda, Sumio and Hadeishi, Hiromu and Fukutake, Toshio and Wakui, Kenji and Tanno, Hirokazu and Ishige, Naoki and Ohasi, Takashi and ... and J-ASPECT Study Collaborators
World Neurosurgery, ISSN 1878-8750, 10/2019, Volume 130, pp. e26 - e46
The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging societies.... 
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | SURGERY | CARE | CLINICAL NEUROLOGY | IMPACT | CLINICAL-VARIABLES | HOSPITAL VOLUME | GENDER | COMA
Journal Article
Cytologia, ISSN 0011-4545, 07/2019, Volume 84, Issue 3, p. 255
A male sterile (ms) plant was identified in each of the two turnip (Brassica rapa) cultivars ‘Kida-aokabu’ and ‘Tennhoji-kabu,’ which are two of the main... 
Collapse | Brassica | Developmental stages | Microspores | Organs | Anthers | Male sterility | Pollen | Flowers | Sterility | Cultivars
Journal Article
Nature Genetics, ISSN 1061-4036, 04/2012, Volume 44, Issue 4, pp. 376 - 378
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 11/2013, Volume 305, Issue 10, pp. L737 - L746
Mitochondria are dynamic organelles that continuously change their shape through fission and fusion. Disruption of mitochondrial dynamics is involved in... 
Mitochondria dynamics | Chronic obstructive pulmonary disease | GLOBAL BURDEN | OXIDATIVE STRESS | PHYSIOLOGY | FUSION | LIPID-PEROXIDATION | CREATINE-KINASE B | mitochondria dynamics | OBSTRUCTIVE PULMONARY-DISEASE | RESPIRATORY SYSTEM | DYNAMICS | FISSION | DYSFUNCTION | chronic obstructive pulmonary disease | COPD | RNA, Small Interfering - genetics | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Epithelial Cells - drug effects | Humans | Cellular Senescence - drug effects | Mitochondrial Proteins - genetics | GTP Phosphohydrolases - antagonists & inhibitors | Immunoenzyme Techniques | Bronchi - drug effects | Mitochondrial Proteins - metabolism | Bronchi - metabolism | Membrane Proteins - metabolism | Bronchi - pathology | Membrane Proteins - genetics | Mitochondrial Proteins - antagonists & inhibitors | Cells, Cultured | Epithelial Cells - pathology | Mitochondria - metabolism | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - drug effects | Mitochondria - pathology | Microscopy, Electron | Blotting, Western | GTP Phosphohydrolases - metabolism | Membrane Proteins - antagonists & inhibitors | GTP Phosphohydrolases - genetics | Tobacco - adverse effects | Physiological aspects | Aging | Epithelial cells | Health aspects | Smoking
Journal Article
IEEJ Transactions on Industry Applications, ISSN 0913-6339, 2018, Volume 138, Issue 9, pp. 730 - 738
This paper describes the influence of rotor eccentricity on the vibration characteristics of a concentrated winding interior permanent magnet synchronous motor... 
Permanent magnet synchronous motor | Rotor eccentricity | Vibration acceleration | Radial electromagnetic force | Finite element method | Vibration | Eccentricity | Electromagnetic forces | Synchronous motors | Magnetism | Permanent magnets | Acceleration
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 07/2019, Volume 10, Issue 1, pp. 3145 - 14
Journal Article
Anticancer research, ISSN 0250-7005, 01/2019, Volume 39, Issue 1, pp. 191 - 200
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 4408 - 9
Sensorineural hearing loss is a common deficit and mainly occurs due to genetic factors. Recently, copy number variants (CNVs) in the STRC gene have also been... 
DEAFNESS | MUTATIONS | GENE | MULTIDISCIPLINARY SCIENCES | GJB2 protein | Phenotypes | Clonal deletion | Copy number | Genetic factors | Hybridization | Hearing protection | Hearing impairment | Hearing loss
Journal Article
Journal of Medical Genetics, ISSN 0022-2593, 12/2017, Volume 54, Issue 12, pp. 836 - 842
Journal Article
Respiratory Research, ISSN 1465-9921, 06/2017, Volume 18, Issue 1, pp. 114 - 14
Background: Pirfenidone (PFD) is an anti-fibrotic agent used to treat idiopathic pulmonary fibrosis (IPF), but its precise mechanism of action remains elusive.... 
Mitophagy | Pirfenidone | Autophagy | Myofibroblast | IPF | MANAGEMENT | MECHANISMS | SUPPRESSION | PATHOGENESIS | RESPIRATORY SYSTEM | PARK2-MEDIATED MITOPHAGY | DISEASE | ORBITAL FIBROBLASTS | IDIOPATHIC PULMONARY-FIBROSIS | HYPERTENSION | Reactive Oxygen Species - metabolism | Humans | Autophagy - drug effects | Myofibroblasts - metabolism | Bleomycin | Transfection | RNA Interference | Lung - metabolism | Pulmonary Fibrosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Autophagy-Related Proteins - genetics | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Myofibroblasts - pathology | Lung - pathology | Mice, Inbred C57BL | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Mitochondria - metabolism | Pulmonary Fibrosis - pathology | Antioxidants - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Myofibroblasts - drug effects | Mice, Knockout | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Mitochondrial Degradation - drug effects | Receptors, Platelet-Derived Growth Factor - metabolism | Lung - drug effects | Pulmonary Fibrosis - chemically induced | Oxidative Stress - drug effects | Pulmonary Fibrosis - drug therapy | Ubiquitin-Protein Ligases - genetics | Autophagy-Related Proteins - metabolism | Pyridones - pharmacology | Genetic aspects | Pulmonary fibrosis | Research | Drug therapy | Reactive oxygen species | Animal models | Platelet-derived growth factor | Pathogenesis | Transforming growth factor | Lysosomes | AKT protein | Activation | Kinases | Remodeling | Machinery | Accumulation | Machinery and equipment | Degradation | Proteins | Mitochondria | Modulation | Fibroblasts | Extracellular matrix | Attenuation | Inhibition | Stress response | Growth factors | Activation analysis | Oxygen | Immunoglobulins | Cytokines | Lung diseases | 1-Phosphatidylinositol 3-kinase | Augmentation | Fibrosis | Mice | Models | Differentiation | In vitro methods and tests | Phagocytosis
Journal Article